On the Study of Network Coded AF Transmission Protocol for Wireless Multiple Access Channels

In this paper, the performance of the network coded amplify-forward cooperative protocol is studied. The use of network coding can suppress the bandwidth resource consumed by relay transmission, and hence increase the spectral efficiency of cooperative diversity. A distributed strategy of relay selection is applied to the cooperative scheme, which can reduce system overhead and also facilitate the development of the explicit expressions of information metrics, such as outage probability and ergodic capacity. Both analytical and numerical results demonstrate that the proposed protocol can achieve large ergodic capacity and full diversity gain simultaneously.

V-band 57-65 GHz receiver

A simple V-band radio IQ receiver architecture based around a six-port monolithic microwave integrated circuit (MMIC) is presented. The receiver assembly is designed to cover the 57-65 GHz broadband wireless communication system frequency allocation. The receiver that has an integral 10 dB microstrip antenna consumes 120 mW of dc power and occupies an area of 23 mm x 16 mm. The receiver can be used in heterodyne or in homodyne mode and has the capacity to demodulate quadrature amplitude modulation (QAM), binary phase shift keying (BPSK)/quadrature phase shift keying (QPSK)/offset quadrature phase shift keying (OQPSK). At 60 GHz the receiver can operate over 10 m range for transmitter effective isotropic radiated power (EIRP) of 20 dBm.

Planar High-Gain WLAN PCB Antenna

A simple design for a low-profile high-gain planar antenna has been presented in the letter. The antenna has the realized gain between 9 and 11 dBi and the return loss better than 10 dB over the 5.6-6.3-GHz frequency band, i.e. 11% bandwidth. A numerical study highlighting effects of key geometrical parameters on the gain and return loss of the antenna has been performed. It has been shown as well that the presented antenna occupies area 20% smaller than a conventional microstrip patch antenna array with a similar gain.

Capacity and resource allocation of cooperative MIMO in ad hoc networks

Cooperative MIMO (Multiple Input–Multiple Output) allows multiple nodes share their antennas to emulate antenna arrays and transmit or receive cooperatively. It has the ability to increase the capacity for future wireless communication systems and it is particularly suited for ad hoc networks. In this study, based on the transmission procedure of a typical cooperative MIMO system, we first analyze the capacity of single-hop cooperative MIMO systems, and then we derive the optimal resource allocation strategy to maximize the end-to-end capacity in multi-hop cooperative MIMO systems. The study shows three implications. First, only when the intra-cluster channel is better than the inter-cluster channel, cooperative MIMO results in a capacity increment. Second, for a given scenario there is an optimal number of cooperative nodes. For instance, in our study an optimal deployment of three cooperative nodes achieve a capacity increment of 2 bps/Hz when compared with direct transmission. Third, an optimal resource allocation strategy plays a significant role in maximizing end-to-end capacity in multi-hop cooperative MIMO systems. Numerical results show that when optimal resource allocation is applied we achieve more than 20% end-to-end capacity increment in average when compared with an equal resource allocation strategy.

The Carboxy-Terminal Modulator Protein (CTMP) Regulates Mitochondrial Dynamics

Background: Mitochondria are central to the metabolism of cells and participate in many regulatory and signaling events. They are looked upon as dynamic tubular networks. We showed recently that the Carboxy-Terminal Modulator Protein (CTMP) is a mitochondrial protein that may be released into the cytosol under apoptotic conditions.

Heat shock protein 70-mediated sensitization of cells to apoptosis by Carboxyl-Terminal Modulator Protein

Background: The serine/threonine protein kinase B (PKB/Akt) is involved in insulin signaling, cellular survival, and transformation. Carboxyl-terminal modulator protein (CTMP) has been identified as a novel PKB binding partner in a yeast two-hybrid screen, and appears to be a negative PKB regulator with tumor suppressor-like properties. In the present study we investigate novel mechanisms by which CTMP plays a role in apoptosis process.

DNA-dependent Protein Kinase-mediated Phosphorylation of Protein Kinase B Requires a Specific Recognition Sequence in the C-terminal Hydrophobic Motif

DNA-dependent protein kinase (DNA-PK) has been implicated in a variety of nuclear processes including DNA double strand break repair, V(D)J recombination, and transcription. A recent study showed that DNA-PK is responsible for Ser-473 phosphorylation in the hydrophobic motif of protein kinase B (PKB/Akt) in genotoxic-stressed cells, suggesting a novel role for DNA-PK in cell signaling. Here, we report that DNA-PK activity toward PKB peptides is impaired in DNA-PK knock-out mouse embryonic fibroblast cells when compared with wild type. In addition, human glioblastoma cells expressing a mutant form of DNA-PK (M059J) displayed a lower DNA-PK activity when compared with glioblastoma cells expressing wild-type DNA- PK (M059K) when PKB peptide substrates were tested. DNA- PK preferentially phosphorylated PKB on Ser-473 when compared with its known in vitro substrate, p53. A consensus hydrophobic amino acid surrounding the Ser-473 phospho-acceptor site in PKB containing amino acids Phe at position +1 and +4 and Tyr at position -1 are critical for DNA- PK activity. Thus, these data define the specificity of DNA- PK action as a Ser-473 kinase for PKB in DNA repair signaling.