958 resultados para pitture rimini restauro trecento nicolò michelino
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Top Row: Arthur Leete, Walter Fishleigh, Emmet Annis, Edward French, Van Lieu Minor
3rd Row: Irving Goodwin, Harry Coe, Frank Nicol, st. mngr. Harold Holmes, John S. Curtis, George Read, John Hodges
2nd Row: coach Keene Fitzpatrick, Floyd Rowe, Guy Leslie Waite, Horace Ramey, captain Arthur Rebstock, John Garrels, David Dunlap, Ath. Dir. Charles Baird
Front Row: Gayle Dull, Martin Daane, Ormond E. Hunt, Claude Pinch
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Catalogue from Lord Ashburnham's library.
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v. III. Scrittori comici: G.M. Cecchi, G.B. Porta, Pandolfo Collenuccio, Bernardo Divizio, Lorenzino de' Medici, Alessandro Piccolomini, G.B. Gelli, Agostino Ricchi, Girolamo Gigli, La commedia dell' arte alla corte di Baviera nel secolo XVI, I tipi comici.--v. IV. Poligrafi: G.B. Giraldi Cintio, Mescolanze d'amore, Senofonte Efesio, Giuseppe Betussi, Federigo Luigini, Lodovico Dolce, Mons. Paolo Giovio, G.F. Pico della Mirandola, Antonfrancesco Doni, Tommaso Moro e T. Campanella, Giuseppe Averani, Card. Guido Bentivoglio, Lucio Annea Seneca, Erasmo, Tullia d'Aragona, Tommaso Garzoni, M. Cervantes Saavedra, Antonio Picozzi, Il trecento in Francia.
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Library copy includes pen notations of prices realized.
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t. 4. Della pittura. Della prospettiva. Della statua. Arte edificatoria. Sulla cupola della chiesa di San Francesco di Rimini, lettera. De' ludi matematici -- t. 5. Trattato del governo della famiglia. Epistola consolatoria. Amiria. Efebie ovvero disputazion amatorie. Lettere amatorie. Poesie.
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Bibliography: pp. 309-312.
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Bibliography: p. [201]-205.
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I. Dai primordi dell'arte cristiana al tempo di Giustiniano.--II. Dall'arte barbarica alla romanica.--III. L'arte romanica.--IV. La scultura del trecento e le sue origini.--V. La pittura del trecento e le sue origini.--VI. La scultura del quattrocento.--VII. La pittura del quattrocento, parte I-IV.--VIII. L'architettura del quattrocento, parte I- .--IX. La pittura del cinquecento. parte I-IV.
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Mode of access: Internet.
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Mode of access: Internet.
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Bookplate of W. S. Appleton.
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Natural killer T (NKT) cells play an important role in controlling cancers, infectious diseases and autoimmune diseases. Although the rhesus macaque is a useful primate model for many human diseases such as infectious and autoimmune diseases, little is known about their NKT cells. We analyzed Valpha24TCR+ T cells from rhesus macaque peripheral blood mononuclear cells stimulated with aalpha-galactosylceramide (a-GalCer) and interleukin-2. We found that rhesus macaques possess Va24TCR+ T cells, suggesting that recognition of alpha-GalCer is highly conserved between rhesus macaques and humans. The amino acid sequences of the V-J junction for the Valpha24TCR of rhesus macaque and human NKT cells are highly conserved (93% similarity), and the CD1d alpha1-alpha2 domains of both species are highly homologous (95.6%). These findings indicate that the rhesus macaque is a useful primate model for understanding the contribution of NKT cells to the control of human diseases.
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Background Many clinical trials of DC-based immunotherapy involve administration of monocyte-derived DCs (Mo-DC) on multiple occasions. We aimed to determine the optimal cell processing procedures and timing (leukapheresis, RBC depletion and cryopreservation) for generation of Mo-DC for clinical purposes. Methods Leukapheresis was undertaken using a COBE Spectra. Two instrument settings were compared - the standard semi-automated software (Version 4.7) (n = 10) and the fully automated software (Version 6.0) (n = 40). Density gradient centrifugation using Ficoll, Percoll, a combination of these methods or neither for RBC depletion were compared. Outcomes (including cell yield and purity) were compared for cryopreserved unmanipulated monocytes and cryopreserved Mo-DC. Results Software Version 6.0 provided significantly better enrichment for monocytes (P
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New vessel formation, a highly-regulated, active process commencing in the embryo and evident notably during the pubertal growth spurt, is essential for normal prostate development. Reactivation of this process in response to physiological stimuli, particularly hypoxia in mature tissues, occurs with new vessels forming principally from stromal components. Although angiogenesis is complex, putatively involving a multitude of angiogenic factors and inhibitors, there is powerful evidence of the importance of the VEGF system in the development of both the normal prostate and prostate cancer. Recent advances include an understanding of how castration acts through the VEGF system to inhibit angiogenesis. Stromal-endothelial and epithelial-endothelial interactions are just beginning to be investigated. A better understanding of how physiological angiogenesis is controlled should help to provide further insights into the mechanism of disregulated angiogenesis in tumours. Ultimately, new antiangiogenic agents are likely to find a role in the management of patients with prostate cancer.