Kitāb Tuḥfat al-Murtaḍawīyah [microform] /

Microfilm.

Sulāfat al-nadīm fī al-muntakhabāt /

No more published?

The fat years and the lean.

Mode of access: Internet.

Gourmet cookery for a low-fat diet,

Mode of access: Internet.

The fat and the thin = (le ventre de Paris) /

Mode of access: Internet.

A practical treatise on the fabrication of volatile and fat varnishes, lacquers, siccatives, and sealing-waxes.

Tr. of:Die fabrikation der Lacke, Firnisse und des Siegellackes.

Boneless beef : raw, cooked, and served : results of analyses for moisture, protein, fat, and ash /

Includes bibliographical references (p. 32-33).

A chemical and physical study of the large and small fat globules in cows' milk /

"January 11, 1909."

Carotin; the principal natural yellow pigment of milk fat; chemical and physiological relations of pigments of milk fat to the carotin and xanthophylls of green plants,

Biography.

Iʻānat al-ṭālibīn lil-ʻallāmah al-fāḍil al-ṣāliḥ al-kāmil al-sayyid Abī Bakr al-mashhūr bi-al-Sayyid al-Bakrī ibn al-ʻārif billāh Muḥammad Shaṭā al-Dimyāṭī nazīl Makkah al-musharrafah zādahā Allāh sharafan wa-rifʻah ʻalá ḥall alfāẓ Fatḥ al-muʻīn lil-ʻallāmah Zayn al-Dīn al-Malībārī [microform]. Wuḍiʻa bi-al-hāmish Fatḥ al-muʻīn al-madhkūr. Maʻa taqrīrāt sharīfah wa-ziyādāt munīfah / lil-Bakrī.

Microfilm.

Nutrient partitioning during treatment of tuberculosis: gain in body fat mass but not in protein mass

Background: Tuberculosis is an important cause of wasting. The functional consequences of wasting and recovery may depend on the distribution of lost and gained nutrient stores between protein and fat masses. Objective: The goal was to study nutrient partitioning, ie, the proportion of weight change attributable to changes in fat mass (FM) versus protein mass (PM), during anti mycobacterial treatment. Design: Body-composition measures were made of 21 men and 9 women with pulmonary tuberculosis at baseline and after 1 and 6 mo of treatment. All subjects underwent dual-energy X-ray absorptiometry and deuterium bromide dilution tests, and a four-compartment model of FM, total body water (TBW), bone minerals (BM), and PM was derived. The ratio of PM to FM at any time was expressed as the energy content (p-ratio). Changes in the p-ratio were related to disease severity as measured by radiologic criteria. Results: Patients gained 10% in body weight (P < 0.001) from baseline to month 6. This was mainly due to a 44% gain in FM (P < 0.001); PM, BM, and TBW did not change significantly. Results were similar in men and women. The p-ratio decreased from baseline to month 1 and then fell further by month 6. Radiologic disease severity was not correlated with changes in the p-ratio. Conclusions: Microbiological cure of tuberculosis does not restore PM within 6 mo, despite a strong anabolic response. Change in the p-ratio is a suitable parameter for use in studying the effect of disease on body composition because it allows transformation of such effects into a normal distribution across a wide range of baseline proportion between fat and protein mass.

Human growth hormone presented by K14hGH-transgenic skin grafts induces a strong immune response but no graft rejection

Although immune responses leading to rejection of transplantable tumours have been well studied, requirements for epithelial tumour rejection are unclear. Here, we use human growth hormone (hGH) expressed in epithelial cells (skin keratinocytes) as a model neo-self antigen to investigate the consequences of antigen presentation from epithelial cells. Mice transgenic for hGH driven from the keratin 14 promoter express hGH in skin keratinocytes. This hGH-transgenic skin is not rejected by syngeneic non-transgenic recipients, although an antibody response to hGH develops in grafted animals. Systemic immunization of graft recipients with hGH peptides, or local administration of stimulatory anti-CD40 antibody, induces temporary macroscopic graft inflammation, and an obvious dermal infiltrate of inflammatory cells, but not graft rejection. These results suggest that a neo-self antigen expressed in somatic cells in skin can induce an immune response that can be enhanced further by induction of specific immunity systemically or non-specific immunity locally. However, immune responses do not always lead to rejection, despite induction of local inflammatory changes. Therefore, in vitro immune responses and in vivo delayed type hypersensitivity are not surrogate markers for immune responses effective against epithelial cells expressing neoantigens.