Five papers on the past proceedings and experience of the Society of Friends, in connexion with the education of youth : read at the meeting of the Friends Educational Society at Ackworth in the years 1838, 1839, 1840, 1841, 1842

Smith

The answer of Mr. Sullivan, to the letter and mis-statements of the Hon. Cadwallader D. Colden, in his "Brief exposition" of himself as the advocate of monopoly. The unconstitutionality or limitation of the monopoly demonstrated. The bad policy and injurious effects of it on the community exposed. The legality of an extension of the term of time of a patent when for the good of a state; and the just views and claims of patentees in steam navigation fully explained.

Pages 29-34 contain "Mr. Duer's opinion on the claim of Mr. Sullivan as a patentee, to navigate with steam boats, the waters of the state of New-York."

The Saginaw River port survey, 1957.

Mode of access: Internet.

Annual report of the Board of Education and the Superintendent of Public Instruction of New Jersey, with accompanying documents, for the school year ending ...

Title varies slightly.

The Stoddard library; a thousand hours of entertainment with the world's great writers,

Mode of access: Internet.

Guide to popular U.S. Government publications.

Mode of access: Internet.

"Sunny memories" : containing personal recollections of some celebrated characters /

Mrs. M. Lloyd supposed author.

Annual statements of the railroad and canal companies of the state of New Jersey.

Vols. for 1867-1915 include report of the state director of the United New Jersey Railroad and Canal Company issued through 1878 under its earlier names: 1867-71, Delaware and Raritan Canal and Camden and Amboy Railroad and Transportation Companies (which continues the reports of the State Directors); 1873-78, United Rail Road and Canal Company of New Jersey.

Program evaluation support for the motor vehicle diagnostic inspection demonstration projects /

Mode of access: Internet.

Farmers' produce markets in the United States.

Pt. 2 issued as U.S. Farm Credit Administration. Cooperative Research and Service Division. Miscellaneous report no. 107 (HD1491.U5A3); pt. 3 as U.S. Dept. of Agriculture. Marketing research report no. 17 (HD1751.A9183)

Dimensional control in precision manufacturing, as applied in production machining to effect higher production and lower unit costs.

Mode of access: Internet.

The Evolving Nature of Small-Scale Forestry in Australia

Two forms of small-scale forestry are developing in Australia, each with different impacts on rural communities. One is based on growing short-rotation Eucalyptus globulus (blue gum) for pulp and the other on production of higher-value products from longer-rotation native hard-woods. Several impediments exist to further development of small-scale forestry, including the lack of a small-scale forestry culture, concerns over harvest rights, lack of market development, the long wait for returns, and satisfaction with current land uses. Nevertheless, the rapid increase in farm woodlot establishment in the past five years has paralleled the strong increase in the private industrial plantation estate. As markets develop and hindrances are overcome, landholders not previously interested in small-scale forestry may consider ita worthwhile land use.

Exposing extinction risk analysis to pathogens: Is disease just another form of density dependence?

In the United States and several other countries., the development of population viability analyses (PVA) is a legal requirement of any species survival plan developed for threatened and endangered species. Despite the importance of pathogens in natural populations, little attention has been given to host-pathogen dynamics in PVA. To study the effect of infectious pathogens on extinction risk estimates generated from PVA, we review and synthesize the relevance of host-pathogen dynamics in analyses of extinction risk. We then develop a stochastic, density-dependent host-parasite model to investigate the effects of disease on the persistence of endangered populations. We show that this model converges on a Ricker model of density dependence under a suite of limiting assumptions, including. a high probability that epidemics will arrive and occur. Using this modeling framework, we then quantify: (1) dynamic differences between time series generated by disease and Ricker processes with the same parameters; (2) observed probabilities of quasi-extinction for populations exposed to disease or self-limitation; and (3) bias in probabilities of quasi-extinction estimated by density-independent PVAs when populations experience either form of density dependence. Our results suggest two generalities about the relationships among disease, PVA, and the management of endangered species. First, disease more strongly increases variability in host abundance and, thus, the probability of quasi-extinction, than does self-limitation. This result stems from the fact that the effects and the probability of occurrence of disease are both density dependent. Second, estimates of quasi-extinction are more often overly optimistic for populations experiencing disease than for those subject to self-limitation. Thus, although the results of density-independent PVAs may be relatively robust to some particular assumptions about density dependence, they are less robust when endangered populations are known to be susceptible to disease. If potential management actions involve manipulating pathogens, then it may be useful to. model disease explicitly.

Double-strand break repair gene polymorphisms and risk of breast or ovarian cancer

Deficiencies in DNA repair have been hypothesized to increase cancer risk and excess cancer incidence is a feature of inherited diseases caused by defects in DNA damage recognition and repair. We investigated, using a case-control design, whether the double-strand break repair gene polymorphisms RAD51 5' untranslated region -135 G > C, XRCC2 R188H G > A, and XRCC3 T241M C > T were associated with risk of breast or ovarian cancer in Australian women. Sample sets included 1,456 breast cancer cases and 793 age-matched controls ages under 60 years of age, 549 incident ovarian cancer cases, and 335 controls of similar age distribution. For the total sample and the subsample of Caucasian women, there were no significant differences in genotype distribution between breast cancer cases and controls or between ovarian cancer cases and combined control groups. The crude odds ratios (OR) and 95% confidence intervals (95% CI) associated with the RAD51 GC/CC genotype frequency was OR, 1.10; 95% CI, 0.80-1.41 for breast cancer and OR, 1.22; 95% CI, 0.92-1.62 for ovarian cancer. Similarly, there were no increased risks associated with the XRCC2 GA/AA genotype (OR, 0.98; 95% CI, 0.76-1.26 for breast cancer and OR, 0.93; 95% CI, 0.69-1.25 for ovarian cancer) or the XRCC3 CT/TT genotype (OR, 0.92; 95% Cl, 0.77-1.10 for breast cancer and OR, 0.87; 95% CI, 0.71-1.08 for ovarian cancer). Results were little changed after adjustment for age and other measured risk factors. Although there was little statistical power to detect modest increases in risk for the homozygote variant genotypes, particularly for the rare RAD51 and XRCC2 variants, the data suggest that none of these variants play a major role in the etiology of breast or ovarian cancer.