The factorial analysis of human ability.

Errata slip inserted.

The preceptor : being a simple system for enabling young men to acquire a knowledge of the doctrines of the gospel and the ability to preach them /

Mode of access: Internet.

Impact of non-tariff barriers on the ability of small business to export to Japan [microform] : hearing before the Select Committee on Small Business, United States Senate, Ninety-sixth Congress, second session, on impact of non-tariff barriers on the ability of small business to export to Japan, June 25, 1980.

Reuse of record except for individual research requires license from Congressional Information Service, Inc.

Final report to the Illinois General Assembly : addressing the educational and related needs of children and youths who are parents, expectant parents, or survivors of domestic or sexual violence to ensure their ability to stay in school, stay safe, and successfully complete their education.

"June 2010."

Illinois public school district ability and effort factors, 1985-1986 /

"October, 1987."

Illinois school district ability and effort factors : 1981-1982.

On cover: Education is everyone's future.

Tips and techniques : ability grouping and performance evaluation in physical education.

Bibliography: p. 11.

The mediatorial reign of the Son of God; or the absolute ability and willingness of Jesus Christ to save all mankind, demonstrated from the scriptures,

Mode of access: Internet.

The evaluation of a causal model of job performance : the relation of job experience and general mental ability to job performance /

"December 1986."

The wonders of history : comprising remarkable battles, sieges, feats of arms, and instances of courage, ability, and magnanimity, occurring in the annals of the world, from the earliest ages to the present time.

[Binder's title: - Frost's pictorial wonders of history.]

The International coffee agreement : its impact on coffee prices, its ability to deal with unforeseen supply and demand conditions, alleged discrimination against U.S. ships in the carriage of coffee, and the soluble coffee controversy /

At head of title: 93d Congress, 1st session. Committee print.

Educational guidance; an experimental study in the analysis and prediction of ability of high school pupils, by Truman Lee Kelley.

Published also as thesis (Ph. D.)--Columbia University, 1914.

Types of reading ability as exhibited through tests and laboratory experiments

Mode of access: Internet.

Minimal mutation of the cytoplasmic tail inhibits the ability of E-cadherin to activate Rac but not phosphatidylinositol 3-kinase - Direct evidence of a role for cadherin-activated Rac signaling in adhesion and contact formation

Classic cadherins are adhesion-activated cell signaling receptors. In particular, homophilic cadherin ligation can directly activate Rho family GTPases and phosphatidylinositol 3-kinase (PI3-kinase), signaling molecules with the capacity to support the morphogenetic effects of these adhesion molecules during development and disease. However, the molecular basis for cadherin signaling has not been elucidated, nor is its precise contribution to cadherin function yet understood. One attractive hypothesis is that cadherin-activated signaling participates in stabilizing adhesive contacts ( Yap, A. S., and Kovacs, E. M. ( 2003) J. Cell Biol. 160, 11-16). We now report that minimal mutation of the cadherin cytoplasmic tail to uncouple binding of p120-ctn ablated the ability of E-cadherin to activate Rac. This was accompanied by profound defects in the capacity of cells to establish stable adhesive contacts, defects that were rescued by sustained Rac signaling. These data provide direct evidence for a role of cadherin-activated Rac signaling in contact formation and adhesive stabilization. In contrast, cadherin-activated PI3-kinase signaling was not affected by loss of p120-ctn binding. The molecular requirements for E-cadherin to activate Rac signaling thus appear distinct from those that stimulate PI3-kinase, and we postulate that p120-ctn may play a central role in the E-cadherin-Rac signaling pathway.