Elroy "Crazy Legs" Hirsch scores TD vs Minnesota, 1943

[appended caption: "Elroy "Crazy Legs" Hirsch scores on a 61-yard run, the very first play of the game in the 1943 route of Minnesota."]

Dicks' standard plays

(cont.) [v.8]. Wild oats; Serious family; Paul Pry; Charles II; Game of Love; Queen Mary's bower; Andy Blake; Naval engagements; Rochester; Artist's wife; Delicate ground; Two queens; Damon and Pythias; Rose of Arragon; Charles I; Mary Stuart; Love's frailties; Fanchon, the cricket; Lear of private life; Robert Macaire.

A select collection of old plays : in eight volumes ; with additional notes and corrections /

Apparently an edition made up from the 12 v. ed. of 1825-27, omitting v. 2-4 and 10.

Personal recollections of service in the Army of the Cumberland and Sherman's Army : from August 17, 1861 to July 20, 1865 /

Mode of access: Internet.

Chefs-d'œuvre des auteurs comiques ...

t. 1. Scarron : Jodelet. Don Japhet d'Armenie. Montfleury: La femme juge et partie. La fille capitaine. La Fontaine: Le florentin. Boursault: Le mercure galant. Baron: L'homme à bonnes fortunes. - t. 2. Dancourt: Le chevalier à la mode. Le marie retrouvé. Les trois cousines. Le galant jardinier. Les bourgeoises de qualité. Dufresny: L'esprit de contradiction. Le double veuvage. La coquette de village. Le mariage fait en rompu. - t. 3. Brueys et Palaprat: Le grondeur. L'avocat Patelin. Le Sage: Crispin rival de son maître. Turcaret. D'Allainval: L'école des bourgeois. La Chaussée: Le préjujé à la mode. L'école des mères. - t. 4. Destouches: Le philosophe marié. Le glorieux. Le dissipateur. La fausse Agnès. Fagan: La pupille. Les originaux. Boissy: Les dehors trompeurs. - t. 5. Marivaus: Le legs. Les fausses confidances. Le jeu de l'amour et du hasard. Piron: Le métromanie. Gresset: Le Méchant. Voltaire: Nanine. Rousseau, J. J.: Le devin du village. - t. 6. Desmahis: L'impertinent. La Noue: La coquette corrigée. Saurin: Les moeurs du temps. Favart: Les trois sultanes. La chercheuse d'esprit. Les amours de Bastien et Bastienne. Annette et Lubin. Ninette à la coeur. Les reveries renouvelées des grecs. Barthe: Les fausses infidélités. Poinsinet de Sivry: Le cercle. - t. 7. Sedaine: Le philosophe sans le savoir. Le gageure imprévue. Marmontel: L'ami de la maison. Collé: La partie de chasse de Henri IV. Monvel: L'amant bourru. Andrieux: Les étourdis. Le rêve du mari. Anaximandre. Chéron: Le tartuffe de moeurs. - t. 8. Collin d'Harleville: Les châteaux en Espagne. Le vieux célibataire. Fabre d'Eglantine: La philinte de Molière. L'intrigue épistolaire. Desforges: Le sourd. Lemercier: Plaute. Pinto.

Census Bureau programs to measure consumer purchase expectations, 1959-1973 /

Mode of access: Internet.

Black Koreans in Korean children’s literature : A study of Won You Soon’s book “Please find Chartlon Sunja Kim”

The purpose of this study is to depict and examine the perception of black Koreans in South Korean children’s literature. This study examines my research questions through four theoretical frameworks: “culture and identity”, “post-colonialism, nationalism and racism”, “blackness and black Koreans’ portrayal in Korean media” and “multiculturalism in Korea”. My study raises the question how multicultural literature can help or not promote a new perception of otherness in South Korea. The method used for this study is qualitative text analysis. The primary source of information is a close-reading of Won You Soon’s book “Please find Charlton Sunja Kim” and interviews with the author of this book. The findings show that there are still some stereotypes about black Koreans and blackness that prevail in South Korean society and can still be found in recent literary works.

Predator species identification from saliva at kill sites with limited remains

Thesis (Master's)--University of Washington, 2016-06

Histone deacetylase inhibitors specifically kill nonproliferating tumour cells

Conventional chemotherapeutic drugs target proliferating cells, relying on often small differences in drug sensitivity of tumour cells compared to normal tissue to deliver a therapeutic benefit. Consequently, they have significant limiting toxicities and greatly reduced efficacy against nonproliferating compared to rapidly proliferating tumour cells. This lack of selectivity and inability to kill nonproliferating cells that exist in tumours with a low mitotic index are major failings of these drugs. A relatively new class of anticancer drugs, the histone deacetylase inhibitors (HDI), are selectively cytotoxic, killing tumour and immortalized cells but normal tissue appears resistant. Treatment of tumour cells with these drugs causes both G1 phase cell cycle arrest correlated with increase p21 expression, and cell death, but even the G1 arrested cells died although the onset of death was delayed. We have extended these observations using cells that were stably arrested by either serum starvation or expression of the cyclin-dependent kinase inhibitor p16(ink4a). We report that histone deacetylase inhibitors have similar cytotoxicity towards both proliferating and arrested tumour and immortalized cells, although the onset of apoptosis is delayed by 24 h in the arrested cells. Both proliferating and arrested normal cells are unaffected by HDI treatment. Thus, the histone deacetylase inhibitors are a class of anticancer drugs that have the desirable features of being tumour-selective cytotoxic drugs that are equally effective in killing proliferating and nonproliferating tumour cells and immortalized cells. These drugs have enormous potential for the treatment of not only rapidly proliferating tumours, but tumours with a low mitotic index.

Inflammation suppressor genes: please switch out all the lights

An effective immune system requires rapid and appropriate activation of inflammatory mechanisms but equally rapid and effective resolution of the inflammatory state. A review of the canonical host response to gram-negative bacteria, the lipopolysaccharide-Toll-like receptor 4 signaling cascade, highlights the induction of repressors that act at each step of the activation process. These inflammation suppressor genes are characterized by their induction in response to pathogen, typically late in the macrophage activation program, and include an expanding class of dominant-negative proteins derived from alternate splicing of common signaling components. Despite the expanse of anti-inflammatory mechanisms available to an activated macrophage, the frailty of this system is apparent in the large numbers of genes implicated in chronic inflammatory diseases. This apparent lack of redundancy between inflammation suppressor genes is discussed with regard to evolutionary benefits in generating a heterogeneous population of immune cells and consequential robustness in defense against new and evolving pathogens.