184 resultados para BURKINA FASO


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A partir del análisis de los filmes La vendedora de rosas (Colombia, 1998), Pizza, birra, faso (Argentina, 1997), Ratas, ratones y rateros (Ecuador, 2000) y Amores Perros (México, 2000) esta investigación realiza un análisis de conflictos culturales contemporáneos formulados desde América Latina. Estas películas dan cuenta de un Cine de la Marginalidad que irrumpe en los años noventa y se caracteriza por: a) el uso de modelos narrativos de género reelabotados a partir de fotografia documental b) el tratamiento de temáticas cotidianas, la crisis de los valores y la marginalidad social e) la crisis de la modernidad y la cultura nacional d) el descentramiento del sujeto. Este cine plantea una paradójica visibilización de las culturas marginales intraducibles a la lógica integradora del Estado y la nación. Despojada de todo narrativa de redención y progreso, la representación del marginal que ofrece pone en escena la misma intraducibilidad y opacidad del subalterno. Al hacerlo, muestra el límite de la racionalidad capitalista -caracterizada por la acumulación de bienes y valores- y la cultura letrada -caracterizada por la acumulación de saberes y acervos simbólicos-.

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Programa de doctorado: Ingeniería de telecomunicación avanzada.

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UV filters belong to a group of compounds that are used by humans and are present in municipal waste-waters, effluents from sewage treatment plants and surface waters. Current information regarding UV filters and their effects on fish is limited. In this study, the occurrence of three commonly used UV filters - 2-phenylbenzimidazole-5-sulfonic acid (PBSA), 2-hydroxy-4-methoxybenzophenone (benzophenone-3, BP-3) and 5-benzoyl-4-hydroxy-2-methoxy-benzenesulfonic acid (benzophenone-4, BP-4) - in South Bohemia (Czech Republic) surface waters is presented. PBSA concentrations (up to 13μgL(-1)) were significantly greater than BP-3 or BP-4 concentrations (up to 620 and 390ngL(-1), respectively). On the basis of these results, PBSA was selected for use in a toxicity test utilizing the common model organism rainbow trout (Oncorhynchus mykiss). Fish were exposed to three concentrations of PBSA (1, 10 and 1000µgL(-1)) for 21 and 42 days. The PBSA concentrations in the fish plasma, liver and kidneys were elevated after 21 and 42 days of exposure. PBSA increased activity of certain P450 cytochromes. Exposure to PBSA also changed various biochemical parameters and enzyme activities in the fish plasma. However, no pathological changes were obvious in the liver or gonads.

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Atenolol is a highly prescribed anti-hypertensive pharmaceutical and a member of the group of β-blockers. It has been detected at concentrations ranging from ng L(-1) to low μg L(-1) in waste and surface waters. The present study aimed to assess the sub-lethal effects of atenolol on rainbow trout (Oncorhynchus mykiss) and to determine its tissue-specific bioconcentration. Juvenile rainbow trout were exposed for 21 and 42 days to three concentration levels of atenolol (1 μg L(-1) - environmentally relevant concentration, 10 μg L(-1), and 1000 μg L(-1)). The fish exposed to 1 μg L(-1) atenolol exhibited a higher lactate content in the blood plasma and a reduced haemoglobin content compared with the control. The results show that exposure to atenolol at concentrations greater than or equal to 10 μg L(-1) significantly reduces both the haematocrit value and the glucose concentration in the blood plasma. The activities of the studied antioxidant enzymes (catalase and superoxide dismutase) were not significantly affected by atenolol exposure, and only the highest tested concentration of atenolol significantly reduced the activity of glutathione reductase. The activities of selected CYP450 enzymes were not affected by atenolol exposure. The histological changes indicate that atenolol has an effect on the vascular system, as evidenced by the observed liver congestion and changes in the pericardium and myocardium. Atenolol was found to have a very low bioconcentration factor (the highest value found was 0.27). The bioconcentration levels followed the order liver>kidney>muscle. The concentration of atenolol in the blood plasma was below the limit of quantification (2.0 ng g(-1)). The bioconcentration factors and the activities of selected CYP450 enzymes suggest that atenolol is not metabolised in the liver and may be excreted unchanged.