Targeting androgen receptor activation function-1 with EPI to overcome resistance mechanisms in castration-resistant prostate cancer


Autoria(s): Yang, Yu Chi; Banuelos, C Adriana; Mawji, Nasrin R; Wang, Jun; Kato, Minoru; Haile, Simon; McEwan, Iain J; Plymate, Stephen; Sadar, Marianne D
Contribuinte(s)

University of Aberdeen, Medicine, Medical Sciences & Nutrition, Medical Sciences

Data(s)

05/08/2016

05/08/2016

02/05/2016

Resumo

Financial support: This research was supported by grants to MDS from the NCI (2R01CA105304), the Canadian Institutes of Health Research (MOP79308) and the US Army Medical Research and Materiel Command Prostate Cancer Research Program (E81XWH-11-1-0551). Research by IJM’s group was supported by the Chief Scientist’s Office of the Scottish Government (ETM-258 and -382). We are grateful to Country Meadows Senior Men’s Golf Charity Classic for financial support of this research.

Peer reviewed

Postprint

Identificador

Yang , Y C , Banuelos , C A , Mawji , N R , Wang , J , Kato , M , Haile , S , McEwan , I J , Plymate , S & Sadar , M D 2016 , ' Targeting androgen receptor activation function-1 with EPI to overcome resistance mechanisms in castration-resistant prostate cancer ' Clinical Cancer Research . , 10.1158/1078-0432.CCR-15-2901

1078-0432

PURE: 65918540

PURE UUID: 3712c05c-8129-47e7-95ea-db55a4a528b8

http://hdl.handle.net/2164/7041

http://dx.doi.org/10.1158/1078-0432.CCR-15-2901

Idioma(s)

eng

Relação

Clinical Cancer Research

Palavras-Chave #androgen receptor #N-terminal domain #EPI #castration-resistant prostate cancer #resistance mechanisms #RC0254 Neoplasms. Tumors. Oncology (including Cancer) #Chief Scientist Office (CSO) #ETM-258 #ETM-382 #RC0254
Tipo

Journal article