988 resultados para Ode II, 12


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Este artículo busca indagar en los aspectos metaliterarios de la Oda II, 12 una clave de lectura de la propuesta poética horaciana. Observamos que el autor utiliza un artificio literario de raigambre helenística, ampliamente desarrollado en la literatura del período augusteo y romanizado, según D'Anna (1979), a partir de Virgilio en la Egloga VI: la recusatio-excusatio. Este recurso funciona en Horacio para distanciarse no sólo del genus grande sino también del género elegíaco y para proponer su propio genus tenue, cuyos rasgos característicos se concentran en la expresión dulcis cantus

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Este artículo busca indagar en los aspectos metaliterarios de la Oda II, 12 una clave de lectura de la propuesta poética horaciana. Observamos que el autor utiliza un artificio literario de raigambre helenística, ampliamente desarrollado en la literatura del período augusteo y romanizado, según D'Anna (1979), a partir de Virgilio en la Egloga VI: la recusatio-excusatio. Este recurso funciona en Horacio para distanciarse no sólo del genus grande sino también del género elegíaco y para proponer su propio genus tenue, cuyos rasgos característicos se concentran en la expresión dulcis cantus

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Este artículo busca indagar en los aspectos metaliterarios de la Oda II, 12 una clave de lectura de la propuesta poética horaciana. Observamos que el autor utiliza un artificio literario de raigambre helenística, ampliamente desarrollado en la literatura del período augusteo y romanizado, según D'Anna (1979), a partir de Virgilio en la Egloga VI: la recusatio-excusatio. Este recurso funciona en Horacio para distanciarse no sólo del genus grande sino también del género elegíaco y para proponer su propio genus tenue, cuyos rasgos característicos se concentran en la expresión dulcis cantus

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Trägerband: Ms. Carm. 2; Vorbesitzer: Karmeliterkloster Frankfurt am Main

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Fil: Pedernera, Soledad. Universidad Nacional de La Plata. Facultad de Humanidades y Ciencias de la Educación. Instituto de Investigaciones en Humanidades y Ciencias Sociales (UNLP-CONICET); Argentina.

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Contient : I詩扇記傳奇Shi shan ji chuan qi.Histoire d'un éventail avec une ode ; II夢裏緣傳奇Meng li yuan chuan qi.La fortune en songe ; III砥石齋韻品雜齣Zhi shi zhai yun pin za chu.Scènes diverses avec poésies du pavillon Zhi shi ; IV賞心幽品四種Shang xin you pin si zhong.Quatre scènes pour réjouir le fond du cœur ; V破牢愁Po lao chou .Le chagrin de l'appui brisé ; VI砥石齋散 (sic) 曲Zhi shi zhai san qu.Scènes diverses du pavillon Zhi shi

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We determined the effects of two classical angiotensin II (ANG II) antagonists, [Sar(1), Ala(8)]-ANG II and [Sar(1), Thr(8)]-ANG II, and losartan (a nonpeptide and selective antagonist for the AT 1 angiotensin receptors) on diuresis, natriuresis, kaliuresis and arterial blood pressure induced by ANG II administration into the median preoptic nucleus (MnPO) of male Holtzman rats weighing 250-300 g. Urine was collected in rats submitted to a water load (5% body weight) by gastric gavage, followed by a second water load (5% body weight) 1 h later. The volume of the drug solutions injected was 0.5 mu l over 10-15 s. Pre-treatment with [Sar(1), Ala(8)]-ANG II (12 rats) and [Sar(1), Thr(8)]-ANG II (9 rats), at the dose of 60 ng reduced (13.7 +/- 1.0 vs 11.0 +/- 1.0 and 10.7 +/- 1.2, respectively), whereas losartan (14 rats) at the dose of 160 ng totally blocked (13.7 +/- 1.0 vs 7.6 +/- 1.5) the urine excretion induced by injection of 12 ng of ANG II (14 rats). [Sar(1), Ala(8)]-ANG II impaired Na+ excretion (193 +/- 16 vs 120 +/- 19): whereas [Sar(1), Thr(8)]-ANG II and losartan blocked Na+ excretion (193 +/- 16 vs 77 +/- 15 and 100 +/- 12, respectively) induced by ANG II. Similar effects induced by ANG II on K+ excretion were observed with [Sar(1), Ala(8)]-ANG II, [Sar(1), Thr(8)]-ANG II, and losartan pretreatment (133 +/- 18 vs 108 +/- 11, 80 +/- 12, and 82 +/- 15, respectively). The same doses as above of [Sar(1), Ala(8)]-ANG II (8 rats), [Sar(1), Thr(8)]-ANG II (8 rats). and losartan (9 rats) blocked the increase in the arterial blood pressure induced by 12 ng of ANG II (12 rats) (32 +/- 4 ru 4 +/- 2, 3.5 +/- 1, and 2 +/- 1: respectively. The results indicate that the AT1 receptor subtype participates in the increases of diuresis, natriuresis. kaliuresis and arterial blood pressure induced by the administration of ANG II into the MnPO.

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We tested the effects of estradiol, progesterone and testosterone on water and salt intake induced by angiotensin II (ANG II) injected into the third ventricle of female Holtzman rats weighing 250-300 g. The water and salt ingestion observed after 120 min in the control experiments (injection of 0.5 mu l of 0.15 M NaCl into the third ventricle) was 1.6 +/- 0.3 ml (N = 10) and 0.3 +/- 0.1 ml (N = 8) in intact rats, respectively, and 1.4 +/- 0.3 ml (N = 10) and 0.2 +/- 0.1 (N = 8) in ovariectomized rats, respectively. ANG II injected in intact rats (4, 6, 12, 25, and 50 ng, icv, in 0.5 mu l saline) induced an increase in water intake (4.3 +/- 0.6, 5.4 +/- 0.7. 7.8 +/- 0.8, 10.4 +/- 1.2, 11.2 +/- 1.4 ml/120 min, respectively) (N = 43). The same doses of icv ANG II in intact rats increased the 3% NaCl intake (0.9 +/- 0.2; 1.4 +/- 0.3, 2.3 +/- 0.4, 2.2 +/- 0.3. and 2.5 +/- 0.4 ml/120 min, respectively) (N = 42). When administered to ovariectomized rats ANG II induced comparable amounts of water intake (4.0 +/- 0.5, 4.8 +/- 0.6, 6.9 +/- 0.7. 9.6 +/- 0.8, and 10.9 +/- 1.2 ml/120 min, respectively) (N = 43) but there was a significant decrease of 3% NaCl solution ingestion (0.3 +/- 0.1, 0.4 +/- 0.1, 0.8 +/- 0.2, 0.7 +/- 0.2, and 0.6 +/- 0.2 ml/120 min, respectively) (N = 44). Estrogen (50 mu g), progesterone (25 ng), and testosterone (300 mu g) were injected daily into ovariectomized rats for 21 days. Treatment with estrogen decreased the water intake and abolished the saline ingestion induced by icy injection of ANG II (12 ng (2.8 +/- 1.2 and 0.3 +/- 0.1 ml/120 min, respectively) (N = 8). Treatment with progesterone also reduced the water intake (3.3 +/- 0.6 ml/120 min) (N = 8) and abolished the ANG II-induced saline ingestion (0.4 +/- 0.1 ml/120 min) (N = 8), but these effects were not observed with testosterone (6.4 +/- 0.8 and 2.2 +/- 0.3 ml/120 min, respectively) (N = 8). These results indicate that ANG II induces a greater increase in sodium intake in intact female rats than in ovariectomized rats and that estrogen and progesterone impair water and sodium intake in ovariectomized rats.