Don Joseph Nasi, Herzog von Naxos, seine Familie und zwei jüdische Diplomaten seiner Zeit : eine Biographie nach neuen Quellen dargest.

von M. A. Levy

Marcus Aurelius Antolius als Zeitgenosse und Freund des Rabbi Jehuda ha-Nasi

Mehr nicht erschienen

Unbekannte Huldigungsgedichte für einen Nasi

Ismar Elbogen

Genealogisches und Chronologisches bezüglich der Patriarchen aus dem Hillel'schen Hause bis auf Rabbi Jehuda ha-Nasi, Redacteur der Mischna

Saul Isaak Kaempf

Genealogisches und Chronologisches bezüglich der Patriarchen aus dem Hillel'schen Hause bis auf Rabbi Jehuda ha-Nasi, Redacteur der Mischna

Saul Isaak Kaempf

Einige Worte Don Joseph Nasi, Herzog von Naxos betreffend

Matthias Bersohn

R. Juda ha-Nasi's Antheil an unserer Mischnah

Josef Zebi Hirsch Dünner

R. Juda ha-Nasi's Antheil an unserer Mischnah

Josef Zebi Hirsch Dünner

Zur Geschichte des Don Josef Nasi

Moses Schorr

Zur Geschichte des Don Joseph Nasi

Moses Schorr

Abraham Bar Samuel Ha-Levi Ibn Chisdai Ha-Nasi : sein Leben und seine Schriften, insbesondere das Sēfer ben ham-melek we-han-nāzîr, Vergleichung des hebr., arab. u. griech. Textes

München, Univ., Diss., 1889

Die innere Einrichtung des grossen Synedrions zu Jerusalem und ihre Fortsetzung im späteren palästinensischen Lehrhause bis zur Zeit des R. Jehuda ha-Nasi : ein Beitrag zum Verständnisse und zur Würdigung der ältesten talmudischen Quellen

Leipzig, Univ., Diss., [1894]

Dissertatio satyrica physico-medico-moralis de pica nasi, sive tabaci sternutatorii moderno abusu, & noxa /

Mode of access: Internet.

Hyposulfatemia, growth retardation, reduced fertility, and seizures in mice lacking a functional NaSi-1 gene

inorganic sulfate is required for numerous functions in mammalian physiology, and its circulating levels are proposed to be maintained by the Na+-SO42- cotransporter, (NaSi-1). To determine the role of NaSi-1 in sulfate homeostasis and the physiological consequences in its absence, we have generated a mouse lacking a functional NaSi-1 gene, Nas1. Serum sulfate concentration was reduced by >75% in Nas1(-/-) mice when compared with Nas1(+/+) mice. Nas1(-/-) mice exhibit increased urinary sulfate excretion, reduced renal and intestinal Na+-SO42- cotransport, and a general growth retardation. Nas1(-/-) mouse body weight was reduced by >20% when compared with Nas1(+/+) and Nas1(+/-) littermates at 2 weeks of age and remained so throughout adulthood. Nas1(-/-) females had a lowered fertility, with a 60% reduction in litter size. Spontaneous clonic seizures were observed in Nas1(-/-) mice from 8 months of age. These data demonstrate NaSi-1 is essential for maintaining sulfate homeostasis, and its expression is necessary for a wide range of physiological functions.

NaSi-1 and Sat-1: structure, function and transcriptional regulation of two genes encoding renal proximal tubular sulfate transporters

Sulfate (SO42-) is an important anion regulating many metabolic and cellular processes. Maintenance Of SO42- homeostasis occurs in the renal proximal tubule via membrane transport proteins. Two SO42- transporters that have been characterized and implicated in regulating serum SO42- levels are: NaSi- 1, a Na+-SO4 (2-) cotransporter located at the brush border membrane and Sat-1, a SO4 (2-) -anion exchanger located on the basolateral membranes of proximal tubular cells. Unlike Sat-1, for which very few studies have looked at regulation of its expression, NaSi- 1 has been shown to be regulated by various hormones and dietary conditions in vivo. To study this further, NaSj- I (SLC13A1) and Sat- I (SLC26A1) gene structures were determined and recent studies have characterized their respective gene promoters. This review presents the current understanding of the transcriptional regulation of NaSj- I and Sat- 1, and describes possible pathogenetic implications which arise as a consequence of altered SO(4)(2-)homeostasis. (c) 2005 Elsevier Ltd. All rights reserved.