951 resultados para Mason, William, 1725-1797.
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Mode of access: Internet.
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Mode of access: Internet.
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Vol. II with Reminiscences by Norton Nicholls.
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Mode of access: Internet.
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Concerns Mason's poem which was written in vindication of Samuel Parker, Bishop of Oxford.
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Mode of access: Internet.
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Appendix: The sentiments of Charles Alphonse Du Fresnoy, on the works of the principal and best painters of the two last ages. The preface of Mr. Dryden to his translation, containing a parallel between poetry and painting. Epistle of Mr. Pope to Mr. Jervas. A chronological list of painters from the revival of the art to the beginning of the present century: p. [127]-213.
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Mode of access: Internet.
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"Prayers ... (with very few exceptions) are compiled from a work written by the late Rev. W. Hill."
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An answer to William Mason's Animadversions.
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Mode of access: Internet.
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Edited by John Mason Good (cf. NUC pre-1956).
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Pages [3],276-496 contain his: The Pilgrim's progress ... Part the second... New York, l822, with special title page.
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From early in the AIDS epidemic, psychosocial stressors have been proposed as contributors to the variation in disease course. To test this hypothesis, rhesus macaques were assigned to stable or unstable social conditions and were inoculated with the simian immunodeficiency virus. Animals in the unstable condition displayed more agonism and less affiliation, shorter survival, and lower basal concentrations of plasma cortisol compared with stable animals. Early after inoculation, but before the emergence of group differences in cortisol levels, animals receiving social threats had higher concentrations of simian immunodeficiency virus RNA in plasma, and those engaging in affiliation had lower concentrations. The results indicate that social factors can have a significant impact on the course of immunodeficiency disease. Socially induced changes in pituitary–adrenal hormones may be one mechanism mediating this relationship.
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Secondary amyloidosis is a common disease of water fowl and is characterized by the deposition of extracellular fibrils of amyloid A (AA) protein in the liver and certain other organs. Neither the normal role of serum amyloid A (SAA), a major acute phase response protein, nor the causes of secondary amyloidosis are well understood. To investigate a possible genetic contribution to disease susceptibility, we cloned and sequenced SAA cDNA derived from livers of domestic ducks. This revealed that the three C-terminal amino acids of SAA are removed during conversion to insoluble AA fibrils. Analysis of SAA cDNA sequences from several animals identified a distinct genetic dimorphism that may be relevant to susceptibility to secondary amyloid disease. The duck genome contained a single copy of the SAA gene that was expressed in liver and lung tissue of ducklings, even in the absence of induction of acute phase response. Genetic analysis of heterozygotes indicated that only one SAA allele is expressed in livers of adult birds. Immunofluorescence staining of livers from adult ducks displaying early symptoms of amyloidosis revealed what appear to be amyloid deposits within hepatocytes that are expressing unusually high amounts of SAA protein. This observation suggests that intracellular deposition of AA may represent an early event during development of secondary amyloidosis in older birds.