An Investigation of instabilities characteristic of liquid propellant rocket motors :

"February 1952."

Pressurization systems for liquid rockets.

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Liquid rocket actuators and operators

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Liquid rocket valve assemblies /

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Liquid rocket engine combustion stabilization devices /

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Liquid rocket engine injectors.

"Prepared under the direction of Howard W. Douglass ... written by G. S. Gill and W. H. Nurick ... and ... edited by Russell B. Keller, Jr."

Liquid rocket pressure regulators, relief valves, check valves, burst disks, and explosive valves.

Cover title.

Liquid rocket disconnects, couplings, fittings, fixed joints, and seals /

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Solid propellant rockets, an introductory handbook; second stage.

Includes the original work (p. 1-162) published separately in 1955.

Solid propellant processing factors in rocket motor design.

At head of title: NASA space vehicle design criteria (chemical propulsion).

Solid propellant combustion; analytical survey

"Based on Soviet open literature available at the Aerospace Technology Division and the Library of Congress. The survey covers the period from January 1962 through May 1966."

The origin and development of hyperammonemia following exposure to hydrazine in rabbits

Hydrazine $\rm (N\sb2H\sb4),$ an important liquid propellant and derivative chemical for pharmaceuticals and pesticides, produces coma and convulsions sometimes resulting in death. Hyperammonia was found in rabbits exposed to 18 mg/Kg of hydrazine. Results of Part One of this study of rabbits emphasize the importance of acute ammonia toxicity during the first three hours following exposure to hydrazine. At no time during this post exposure period did a significant reduction of hydrazine to ammonia occur. Therefore, the elevated blood ammonia was apparently secondary to the effects of hydrazine on metabolic pathways. Further, the results support the theory of competitive inhibition of ammonia by hydrazine and emphasize the need to monitor plasma ammonia following toxic exposure to hydrazine.^ In Part Two, urea, ammonia, CO$\sb2,$ pH, glucose, sodium, potassium, chloride and creatinine were measured for up to 4 hours following injection of 18 mg/Kg of hydrazine in each of two groups of five rabbits. One group received normal saline and the other group received 5% dextrose and water/normal saline. Hyperammonemia, minimal metabolic acidosis and hyperglycemia without increased urea were found in the rabbits receiving normal saline intravenous infusion and hydrazine injection. Hence, hypoglycemia does not appear to play a role in the development of hyperammonemia. A significant difference in the elevated ammonia levels between the two groups receiving dextrose and water/normal saline and normal saline at 1 hour occurred. There was no significant difference in the elevated ammonia levels seen between the two groups receiving dextrose and water/normal saline and normal saline at 2.5 and 4 hours. Thus at 1 hour the group receiving dextrose was able to utilize excess glucose to detoxify ammonia, while at 2.5 and 4 hours there was no significant difference in the two groups' ability to detoxify ammonia.^ Findings support the theory that hydrazine inhibits the formation of urea resulting in hyperammonemia. Results suggest that hydrazine at 18 mg/Kg, a known hypoglycemic agent, causes serious hyperammonemia without increasing urea production during hyperglycemia. These experiments support a unified theory for the toxic mechanism of action of hydrazine, i.e., the intermediary metabolic effects of hydrazine are brought about by the formation of hydrazones which encumber ATP synthesis and vitamin B$\sb6$ enzymatic reactions. ^

Solid rocket thrust vector control.

Cover title.

Solid rocket motor nozzles /

"...written by Russell A. Ellis."

Captive-fired testing of solid rocket motors

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