959 resultados para Cr^4
Resumo:
NdYAG808nmNdYAGQ90Cr^4YAGQ119mW465mW1064nm16ns1818kHz34J61mWQ
Resumo:
LDNdYLFCr^4YAGQ1053nm05mm90Cr^4 YAG17W606ns15W95kHz1579mJ055mm95Cr^4 YAG17W686ns135W14kHz
Resumo:
(Cr^4+Nd^3+GGG)QCrCr^4+GGGNd^3+GGG(Cr^4+Nd^3+)GGG(Cr^4+Nd^3+)GGGNd^3+GGG33s250s(Cr^4+Nd^3+)GGGQ
Resumo:
(Cr^4+Nd^3+:GGG)CrNdJuddofeltt(t=246)McCumber^4F3/2^4I11/2Cr^3+300900nmCr^3+Nd^3+Cr^4+106m
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CrAl2O3Al2O3Cr^3Mg^2Cr^4Cr^41223nmCr^437nm
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X,(SESAM),KerrCr4+YAG.80 fs,120 MHz,100 W.
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YbYAGYb^3YbYbYAGXYb^3CrYbYAGCr^4
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Edge-sharing bioctahedral (ESBO) complexes [Ru-2(OMe)(O2CC6H4-p-X)3(1-MeIm)(4)](ClO4)2 (X = OMe (1a), Me (1b)) and [Ru-2(O2CC6H4-P-X)(4)(1-MeIm)(4)](ClO4)(2) (X = OMe (2a), Me (2b)) are prepared by reacting Ru2Cl(O(2)CR)(4) with 1-methylimidazole (1-MeIm) in methanol followed by treatment with NaClO4. Complex 2a and the PF6- salt (1a') of 1a have been structurally characterized. Crystal data for 1a.1.5MeCN. 0.5Et(2)O: triclinic, P (1) over bar, a = 13.125(2) Angstrom, b = 15.529(3) Angstrom, c 17.314(5) Angstrom, a; 67.03(2)degrees, beta 68.05(2)degrees, gamma = 81.38(1)degrees, V 3014(1) Angstrom(3), Z = 2. Crystal data for 2a: triclinic, P (1) over bar, a 8.950(1) Angstrom, b = 12.089(3) Angstrom, c = 13.735(3) Angstrom, alpha 81.09(2)degrees, beta = 72.27(1)degrees, gamma = 83.15(2)degrees, V = 1394(1) Angstrom(3), Z = 1. The complexes consist of a diruthenium(III) unit held by two monoatomic and two three-atom bridging ligands. The 1-MeIm ligands are at the terminal sites of the [Ru-2(mu-L)(eta(1):mu-O(2)CR)(eta(1):eta(1):mu-O(2)CR)(2)](2+) core having a Ru-Ru single bond (L = OMe or eta(1)-O(2)CR). The Ru-Ru distance and the Ru-O-Ru angle in the core of 1a' and 2a are 2.49 Angstrom and similar to 76 degrees. The complexes undergo one-electron oxidation and reduction processes in MeCN-0.1 M TBAP to form mixed-valence diruthenium species with Ru-Ru bonds of orders 1.5 and 0.5, respectively.
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Organic thin-film transistors (OTFTs) using high dielectric constant material tantalum pentoxide (Ta2O5) and benzocyclobutenone (BCBO) derivatives as double-layer insulator were fabricated. Three metals with different work function, including Al (4.3 eV), Cr (4.5 eV) and Au (5.1 eV), were employed as gate electrodes to study the correlation between work function of gate metals and hysteresis characteristics of OTFTs. The devices with low work function metal Al or Cr as gate electrode exhibited high hysteresis (about 2.5 V threshold voltage shift). However, low hysteresis (about 0.7 V threshold voltage shift) OTFTs were attained based on high work function metal Au as gate electrode.
Resumo:
88000 t80% 1. 18.2%6.9-32.1%3.7-9.6 t9.8%6.6-14.5%1.3-2.7 t 2. 7-40000 ng•m-315000 ng•m-3120 ng•m-324 ng•m-343-2100 ng•L-10.30-1.1 ng•L-192-2300 ng•L-12.6-7.9 ng•L-11.3-360 mg•kg-10.18-47.5 mg•kg-1pH 3. 1060 g•g-1 Cr347 g•g-1 Cr917 g•g-1 Cr-50 g•g-1 Cr40.0 g•g-1 Cr66.2 g•g-1 Cr56.9 g•g-1 Cr1.30 g•g-1 Cr2.5 g•g-1 Cr 4. 37.31.92.3 g•g-12.81.31.5 g•g-1358.521.333.1 ng•g-114.65.74.0 ng•g-1397.5%94.1%93.5% (r=0.65, p<0.01) 5. 5.05 g•g-12.35-10.6 g•g-15.63 g•g-12.54-9.55 g•g-13.53 g•g-11.87-5.65 g•g-14.01 g•g-12.29-9.23 g•g-132.81.31.5 g•g-10.47-5.69 g•g-10.65 g•g-10.26¬-1.38 g•g-12003-0.23 g•d-1•kg-10.1 g•d-1•kg-1 (USEPA, 1997)
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Defective efferocytosis may perpetuate inflammation in smokers with or without chronic obstructive pulmonary disease (COPD). Macrophages may phenotypically polarize to classically activated M1 (proinflammatory; regulation of antigen presentation) or alternatively activated M2 (poor antigen presentation; improved efferocytosis) markers. In bronchoalveolar lavage (BAL)–derived macrophages from control subjects and smoker/ex-smoker COPD subjects, we investigated M1 markers (antigen-presenting major histocompatibility complex [MHC] Classes I and II), complement receptors (CRs), the high-affinity Fc receptor involved with immunoglobulin binding for phagocytosis (Fc-gamma receptor, FcγR1), M2 markers (dendritic cell–specific intercellular adhesion molecule-grabbing nonintegrin [DC-SIGN] and arginase), and macrophage function (efferocytosis and proinflammatory cytokine production in response to LPS). The availability of glutathione (GSH) in BAL was assessed, because GSH is essential for both M1 function and efferocytosis. We used a murine model to investigate macrophage phenotype/function further in response to cigarette smoke. In lung tissue (disaggregated) and BAL, we investigated CRs, the available GSH, arginase, and efferocytosis. We further investigated the therapeutic effects of an oral administration of a GSH precursor, cysteine l-2-oxothiazolidine-4-carboxylic acid (procysteine). Significantly decreased efferocytosis, available GSH, and M1 antigen–presenting molecules were evident in both COPD groups, with increased DC-SIGN and production of proinflammatory cytokines. Increased CR-3 was evident in the current-smoker COPD group. In smoke-exposed mice, we found decreased efferocytosis (BAL and tissue) and available GSH, and increased arginase, CR-3, and CR-4. Treatment with procysteine significantly increased GSH, efferocytosis (BAL: control group, 26.2%; smoke-exposed group, 17.66%; procysteine + smoke-exposed group, 27.8%; tissue: control group, 35.9%; smoke-exposed group, 21.6%; procysteine + smoke-exposed group, 34.5%), and decreased CR-4 in lung tissue. Macrophages in COPD are of a mixed phenotype and function. The increased efferocytosis and availability of GSH in response to procysteine indicates that this treatment may be useful as adjunct therapy for improving macrophage function in COPD and in susceptible smokers.
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Fundao de Amparo Pesquisa do Estado de So Paulo (FAPESP)
