Investigation of diseases of swine, and infectious and contagious diseases incident to other classes of domesticated animals.

Microfilmed for preservation

Rules and regulations of the Board of live stock commissioners of the state of Illinois, with extracts from the law in relation in live stock sanitation, cruelty to animals, meat inspection and other matters relating to live stock interests, 1908 ...

Mode of access: Internet.

Contagious diseases; what they are and how to deal with them

"First edition."

A treatise on cattle : shewing the most approved methods of breeding, rearing, and fitting for use, horses, asses, mules, horned cattle, sheep, goats, and swine ; with directions for the proper treatment of them in their several disorders ; to which is added, a dissertation on their contagious diseases /

Includes bibliographical references and index.

A treatise on the means of purifying infected air, of preventing contagion, and arresting its progress /

Includes bibliographical references.

Contagious diseases of domesticated animals. Continuation of investigation by Department of Agriculture.

Investigation of swine plague -- Investigation of southern cattle fever -- Contagious lung plague of cattle -- Miscellaneous -- Correspondence.

Contagious diseases of domesticated animals.

Continuation of Special reports no. 12 and 22. No. 12 entitled: Investigation of diseases of swine [etc.]

Report on Contagious Diseases of Animals

Title Varies Slightly. Annual, 1887-1904; Biennial, 1904/06-1908/10. for Later Reports See Maine. Live Stock Sanitary Commissioner

Report of the Live Stock Sanitary Commissioner of the State of Maine on Contagious Diseases of Animals

Succeeds Main. Cattle Commissioners. Report on Contagious Diseases of Animals. the Cattle Commissioners Were Abolished In 1911. No Report Issued for 1911-12

Gender-specific differences of interaction between obesity and air pollution on stroke and cardiovascular diseases in Chinese adults from a high pollution range area: A large population based cross sectional study

Background Little information exists regarding the interaction effects of obesity with long-term air pollution exposure on cardiovascular diseases (CVDs) and stroke in areas of high pollution. The aim of the present study is to examine whether obesity modifies CVD-related associations among people living in an industrial province of northeast China. Methods We studied 24,845 Chinese adults, aged 18 to 74 years old, from three Northeastern Chinese cities in 2009 utilizing a cross-sectional study design. Body weight and height were measured by trained observers. Overweight and obesity were defined as a body mass index (BMI) between 25–29.9 and ≥ 30 kg/m2, respectively. Prevalence rate and related risk factors of cardiovascular and cerebrovascular diseases were investigated by a questionnaire. Three-year (2006–2008) average concentrations of particulate matter (PM10), sulfur dioxide (SO2), nitrogen dioxides (NO2), and ozone (O3) were measured by fixed monitoring stations. All the participants lived within 1 km of air monitoring sites. Two-level logistic regression (personal level and district-specific pollutant level) was used to examine these effects, controlling for covariates. Results We observed significant interactions between exposure and obesity on CVDs and stroke. The associations between annual pollutant concentrations and CVDs and stroke were strongest in obese subjects (OR 1.15–1.47 for stroke, 1.33–1.59 for CVDs), less strong in overweight subjects (OR 1.22–1.35 for stroke, 1.07–1.13 for CVDs), and weakest in normal weight subjects (OR ranged from 0.98–1.01 for stroke, 0.93–1.15 for CVDs). When stratified by gender, these interactions were significant only in women. Conclusions Study findings indicate that being overweight and obese may enhance the effects of air pollution on the prevalence of CVDs and stroke in Northeastern metropolitan China. Further studies will be needed to investigate the temporality of BMI relative to exposure and onset of disease.

Genome-wide association studies and musculoskeletal diseases

Bone and joint diseases are major causes of morbidity and mortality worldwide, and their prevalence is increasing as the average population age increases. Most common musculoskeletal diseases show significant heritability, and few have treatments that prevent disease or can induce true treatment-free, disease-free remission. Furthermore, despite valiant efforts of hypothesis-driven research, our understanding of the etiopathogenesis of these conditions is, with few exceptions, at best moderate. Therefore, there has been a long-standing interest in genetics research in musculoskeletal disease as a hypothesis-free method for investigating disease etiopathogenesis. Important contributions have been made through the identification of monogenic causes of disease, but the holy grail of human genetics research has been the identification of the genes responsible for common diseases. The development of genome-wide association (GWA) studies has revolutionized this field, and led to an explosion in the number of genes identified that are definitely involved in musculoskeletal disease pathogenesis. However, this approach will not identify all common disease genes, and although the current progress is exciting and proves the potential of this research discipline, other approaches will be required to identify many of the types of genetic variation likely to be involved.