Therese Molling with Joel, Ilse, Adolf, Grete, and Leopold Meyerhof on rooftop of store and residence Meyerhof am Platze; Hildesheim, Germany.

From left to right, standing: Ilse, Adolf, Grete and Lepold Meyerhof; from left to right, sitting: Joel Meyerhof and Therese Molling

Gathering in the home of Dr. Alfred Stern and his wife Lotte nee Wallerstein; Paula Wallerstein nee Molling; Rheydt, Germany. Portraits Groups

Dr. Alfred Stern probably standing far left; Paula Wallerstein nee Molling seated in center

Tombstone of the Benno Molling family on the Jewish cemetery; An der Strangriede, Hannover, Germany. Cemeteries Tombs

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Fritz Gottschalk and Therese Gottschalk nee Molling on a ski trip; Berchtesgaden, Germany.

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Portrait of Liesel (Elizabeth) Gottschalk with her grandfather Adolf Molling; Germany.

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Portrait of Henny Molling nee Meyerhof and her daughter Therese Molling in living room; Germany.

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Studio portrait of Henny Molling nee Meyerhof and her daughter Therese Molling; Hannover, Germany.

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Walter, Ursula and Freddy, three of Fritz Gottschalk's chidren on the beach; Norderney, Germany.

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The Gottschalk family on the beach; Norderney, Germany.

(l-r) Elizabeth, Hans Ludwing (Hal), the maid, Ursula, Walter, Freddy, Therese (in Strandkorg) and Kurt standing

Characterization of stable isotope fractionation during methane production in the sediment of a eutrophic lake,lake dagow,germany

We measured delta C-13 of CO2, CH4, and acetate-methyl in profundal sediment of eutrophic Lake Dagow by incubation experiments in the presence and absence of methanogenic inhibitors chloroform, bromoethane sulfonate (BES), and methyl fluoride, which have different specificities. Methyl fluoride predominantly inhibits acetoclastic methanogenesis and affects hydrogenotrophic methanogenesis relatively little. Optimization of methyl fluoride concentrations resulted in complete inhibition of acetoclastic methanogenesis. Methane was then exclusively produced by hydrogenotrophic methanogenesis and thus allowed determination of the fractionation factors specific for this methanogenic pathway. Acetate, which was then no longer consumed, accumulated and allowed determination of the isotopic signatures of the fermentatively produced acetate. BES and chloroform also inhibited CH4 production and resulted in accumulation of acetate. The fractionation factor for hydrogenotrophic methanogenesis exhibited variability, e. g., it changed with sediment depth. The delta C-13 of the methyl group of the accumulated acetate was similar to the delta C-13 of sedimentary organic carbon, while that of the carboxyl group was by about 12 parts per thousand higher. However, the delta C-13 of the acetate was by about 5 parts per thousand lower in samples with uninhibited compared with inhibited acetoclastic methanogenesis, indicating unusual isotopic fractionation. The isotope data were used for calculation of the relative contribution of hydrogenotrophic vs. acetoclastic methanogenesis to total CH4 production. Contribution of hydrogenotrophic methanogenesis increased with sediment depth from about 35% to 60%, indicating that organic matter was only partially oxidized in deeper sediment layers.

Do fertility transitions influence infant mortality declines? Evidence from early modern Germany

The timing and sequencing of fertility transitions and early-life mortality declines in historical Western societies indicate that reductions in sibship (number of siblings) may have contributed to improvements in infant health. Surprisingly, however, this demographic relationship has received little attention in empirical research. We outline the difficulties associated with establishing the effect of sibship on infant mortality and discuss the inherent bias associated with conventional empirical approaches. We offer a solution that permits an empirical test of this relationship while accounting for reverse causality and potential omitted variable bias. Our approach is illustrated by evaluating the causal impact of family size on infant mortality using genealogical data from 13 German parishes spanning the sixteenth, seventeenth, eighteenth, and nineteenth centuries. Overall, our findings do not support the hypothesis that declining fertility led to increased infant survival probabilities in historical populations.

22/5/20, stade Jean Bouin, équipe [de football] du 166e [régiment d']infanterie : [photographie de presse] / [Agence Rol]

Référence bibliographique : Rol, 59354