Exaltation : for violin, violoncello, harp (or piano) and organ. [music] /

Cover title.

Tu es Petrus; an offertorium for soprano solo, chorus, orchestra and organ, composed for and sung at the dedication of the St. Peter's Church Boston.

Text in Latin and English.

Tu es Petrus; an offertorium for soprano solo, chorus, orchestra and organ.

Pl. no. 5221-20.

Jesus! thy boundless love to me; motet for chorus, soli or semi-chorus, orchestra, and organ.

Mode of access: Internet.

Christmas tidings : a cantata for Christmas : for soli, chorus, and organ /

Duration: 30:00.

The legend of St. Christopher; a dramatic oratorio, for solo voices, chorus, orchestra, and organ.

Pl. no. 8269.

Mass for 4 voices and organ with a special graduale and offertory for the feast of the Immaculate Conception ... Op. 62.

Mode of access: Internet.

Te Deum; for 3 choirs, orchestra and organ, op. 22.

Mode of access: Internet.

Psalm 150 : for chorus, orchestra and organ /

Mode of access: Internet.

Expression profiling of type 2 diabetes susceptibility genes in the pancreatic islets, adipose tissue and liver of obese mice

Type 2 diabetes (T2D) is characterized by impaired beta cell function and insulin resistance. T2D susceptibility genes identified by Genome-wide association studies (GWAS) are likely to have roles in both impaired insulin secretion from the beta cell as well as insulin resistance. The aim of this study was to use gene expression profiling to assess the effect of the diabetic milieu on the expression of genes involved in both insulin secretion and insulin resistance. We measured the expression of 43 T2D susceptibility genes in the islets, adipose and liver of leptin-deficient Ob/Ob mice compared with Ob/+ littermates. The same panel of genes were also profiled in cultured rodent adipocytes, hepatocytes and beta cells in response to high glucose conditions, to distinguish expression effects due to elevated glycemia from those on the causal pathway to diabetes or induced by other factors in the diabetic microenviroment. We found widespread deregulation of these genes in tissues from Ob/Ob mice, with differential regulation of 23 genes in adipose, 18 genes in liver and one gene (Tcf7l2) in islets of diabetic animals (Ob/Ob) compared to control (Ob/+) animals. However, these expression changes were in most cases not noted in glucose-treated adipocyte, hepatocyte or beta cell lines, indicating that they may not be an effect of hyperglycemia alone. This study indicates that expression changes are apparent with diabetes in both the insulin producing beta cells, but also in peripheral tissues involved in insulin resistance. This suggests that incidence or progression of diabetic phenotypes in a mouse model of diabetes is driven by both secretory and peripheral defects. © J. A. Barth Verlag in Georg Thieme Verlag KG Stuttgart New York.

Drug Transporter Gene Expression in Human Colorectal Tissue and Cell Lines : Modulation with Antiretrovirals for Microbicide Optimization

This work was supported by the European Union's Seventh Programme for research, technological development and demonstration under grant agreement No 305316 as part of the MOTIF (Microbicides Formulation Through Innovative Formulation for Vaginal and Rectal Delivery) project.

Drug Transporter Gene Expression in Human Colorectal Tissue and Cell Lines : Modulation with Antiretrovirals for Microbicide Optimization

This work was supported by the European Union's Seventh Programme for research, technological development and demonstration under grant agreement No 305316 as part of the MOTIF (Microbicides Formulation Through Innovative Formulation for Vaginal and Rectal Delivery) project.