Geshikhṭe fun yidisher ṭeaṭer-ḳunsṭ un drame : fun di elṭsṭe tsayṭn biz 1750

Yitsḥaḳ Shiper

Margaret OH̀ea, Brustbild

Signatur des Originals: S 36/F09444

Mus S 21/536 - Costumes aus der Oper "Der Baum der Diana" [L'arbore di Diana], für die EA Frankfurt 12.06.1788

1. Diana, 2. Endimion, 3. Amor, 4. Dorist, 5. Silvio, 6. Silvio als Priester, 7. Nymphe

Ms. lat. oct. 69 - Collegium Institutionum in quo praecipue ad Notas Locamerianas ea quae deficiunt supplentur

Die tatsächliche Foliierung weicht von den Angaben des Kataloges ab.

Secunda pars dictionarij in qua dictio quelibet per literas alphabeti iuxta co[n]gruentia[m] distinguitur vtqua[m] facillime omnia in ea contenta reperia[n]tur co[n]tinetq[ue] dictio[n]es his literis incipientes E, F, G, H, I, K, L, M, N, O / [editu[m] pe

Sign.: 2a-2z8, 2A-2Y8, 2Z10

D. Hieronymi epistolarum secundus tomus : complectens elegktika kai apologetika, nimirum ea, quae pertinent ad refellendas diuersas haereseis, & maledicoru[m] calumnias : vnà cum argumentis & scholijs / Des. Erasmi Roterodami.

Imp. tomados del colofón del t. 9

In festo S. Salome Viduae : omnia de communi sanctarum nec Virginum, praeter, ea, quae, sequuntur ..

Precede al tit.: "Die XXII. Octobris."

Copia del Oficio pasado al S[eñ]or Reg[en]te de la R[ea]l Aud[ienci]a y [al] Rector de la Universi[da]d Literaria, sobre la Protectoria del Colegio de los Santos Reyes dicho de Villena [Manuscrito]

Alcance y contenido: Copia ms. sin firmar de un oficio envíado por el Subdelegado en Valencia de la "Colecturía general de espolios y vacantes", ramo Inquisición, organismo creado en Madrid en 1753, unida a la Comisaría general de Cruzada, aunque con función independiente y jurisdicción especial. El Colector General, tenía subdelegados en todas las diócesis. Esta Colecturía se suprimió en 1851. El subdelegado le reclama tanto al Regente de la Real Audiencia de Valencia, como al Rector de la Universidad Literaria la protectoría del Colegio de los Santos Reyes de Villena por ser una institución perteneciente a la Inquisición.

Pius Papa V... Romanus Pontifex ad ea libente intendere consueuit per que quarun libet ciuitatum primum almae urbi propij videlicet domicilij... [Texto impreso]

Precede al tít. : "Ad perpetuam rei memoriam"

Commentaria expositio... Hieronymi Perez... ordinis sanctae Mariae de mercede redemptionis... super primam partem summae s. Thomae Aquinatis, quantum ad ea quae concernunt primum librum sententiarum [Texto impreso]

Pie de imp. tomado de colofón

Role of the major histocompatibility complex class II Ea gene in lupus susceptibility in mice

The gene(s) encoded within major histocompatibility complex (MHC) act as one of the major genetic elements contributing to the susceptibility of murine systemic lupus erythematosus (SLE). We have recently demonstrated that lupus susceptibility is more closely linked to the I-E− H-2b haplotype than to the I-E+ H-2d haplotype in lupus-prone BXSB and (NZB × BXSB)F1 hybrid mice. To investigate whether the reduced susceptibility to SLE in H-2d mice is related to the expression of the MHC class II Ea gene (absent in H-2b mice), we determined the possible role of the Ea gene as a lupus protective gene in mice. Our results showed that (i) the development of SLE was almost completely prevented in BXSB (H-2b) mice expressing two copies of the Ead transgene at the homozygous level as well as in BXSB H-2k (I-E+) congenic mice as for H-2d BXSB mice, and (ii) the expression of two functional Ea (transgenic and endogenous) genes in either H-2d/b (NZB × BXSB)F1 or H-2k/b (MRL × BXSB)F1 mice provided protection from SLE at levels comparable to those conferred by the H-2d/d or H-2k/k haplotype. In addition, the level of the Ea gene-mediated protection appeared to be dependent on the genetic susceptibility to SLE in individual lupus-prone mice. Our results indicate that the reduced susceptibility associated with the I-E+ H-2d and H-2k haplotypes (versus the I-E− H-2b haplotype) is largely, if not all, contributed by the apparent autoimmune suppressive effect of the Ea gene, independently of the expression of the I-A or other MHC-linked genes.