1.° Liber inscriptus : Cautelae Bagaroti, juris Professoris. — 2.° Anonymi tractatus de causarum exercitiis. — 3.° Summula de libellis : authore Bernardo Dorna, Provinciali. — 4.° Cavillationes domini Uberti de Bobio. — 5.° Distinctiones a domino Ugone compositae, et a domino Alberico consummatae. — 6.° Summa de arbitris a domino B. promulgata, missa ad A. F. Romanae Ecclesiae Cancellarium. — 7.° Opusculum, cujus titulus est : Arbor actionum. — 8.° Anonymi tractatus de actionibus. — 9.° Formularium procurationum, secundum usum curiae Romanae

Colbertinus

1.° P. Ovidii Nasonis heroïdes : epistolae octo priores integrae et nonae initium desiderantur. — 2.° Ejusdem amorum libri tres. — 3.° Ejusdem de arte amandi libri tres. — 4.° Ejusdem de remedio amoris libri duo. — 5.° Ejusdem carmen in ibin. — 6.° Ejusdem elegia de nuce. — 7.° Ejusdem carmen de medicamine faciei. — 8.° Ejusdem somnium. — 9.° Bernardi Silvestri megacosmus et microcosmus.

Mentellianus

Secure and Prepared Newsletter, August 2013, Vol. 9, no. 8

A bi-weekly newsletter for those involved in the fields of homeland security and/or emergency management

EPI Update, August 9, 2013

Weekly newsletter for Center For Acute Disease Epidemiology of Iowa Department of Public Health.

Iowa Crop and Weather Report, September 9, 2013 Vol 13, no.27

Report produced by the The Department of Agriculture and Land Stewardship, Climatology Bureau. Iowa Secretary of Agriculture Bill Northey today commented on the Iowa Crops and Weather report released by the USDA National Agricultural Statistical Service. The report is released weekly from April through October.

Interleukin-17 is a critical mediator of vaccine-induced reduction of Helicobacter infection in the mouse model.

BACKGROUND & AIMS: Despite the proven ability of immunization to reduce Helicobacter infection in mouse models, the precise mechanism of protection has remained elusive. This study explores the possibility that interleukin (IL)-17 plays a role in the reduction of Helicobacter infection following vaccination of wild-type animals or in spontaneous reduction of bacterial infection in IL-10-deficient mice. METHODS: In mice, reducing Helicobacter infection, the levels and source of IL-17 were determined and the role of IL-17 in reduction of Helicobacter infection was probed by neutralizing antibodies. RESULTS: Gastric IL-17 levels were strongly increased in mice mucosally immunized with urease plus cholera toxin and challenged with Helicobacter felis as compared with controls (654 +/- 455 and 34 +/- 84 relative units for IL-17 messenger RNA expression [P < .01] and 6.9 +/- 8.4 and 0.02 +/- 0.04 pg for IL-17 protein concentration [P < .01], respectively). Flow cytometry analysis showed that a peak of CD4(+)IL-17(+) T cells infiltrating the gastric mucosa occurred in immunized mice in contrast to control mice (4.7% +/- 0.3% and 1.4% +/- 0.3% [P < .01], respectively). Gastric mucosa-infiltrating CD4(+)IL-17(+) T cells were also observed in IL-10-deficient mice that spontaneously reduced H felis infection (4.3% +/- 2.3% and 2% +/- 0.6% [P < .01], for infected and noninfected IL-10-deficient mice, respectively). In wild-type immunized mice, intraperitoneal injection of anti-IL-17 antibodies significantly inhibited inflammation and the reduction of Helicobacter infection in comparison with control antibodies (1 of 12 mice vs 9 of 12 mice reduced Helicobacter infection [P < .01], respectively). CONCLUSIONS: IL-17 plays a critical role in the immunization-induced reduction of Helicobacter infection from the gastric mucosa.

Footnotes, September 2013, Vol. 38, no.7-9

Monthly newsletter of State Library

Secure and Prepared Newsletter, October 2013, Vol. 9, no. 10

A bi-weekly newsletter for those involved in the fields of homeland security and/or emergency management

Secure and Prepared Newsletter, September 2013, Vol. 9, no. 9

A bi-weekly newsletter for those involved in the fields of homeland security and/or emergency management

PathBeat. Number 9 (Winter 2008) University of Iowa Department of Pathology.

University of Iowa Department of Pathology newsletter.