998 resultados para P2Y receptor

Peroxisome proliferator-activated receptor-α and NADPH oxidases interact to play an important regulatory role in pressure-overload cardiac hypertrophy.

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Does interaction between Peroxisome proliferator-activated receptor-α and NADPH oxidases play an important role in regulating pressure-overload cardiac hypertrophy?

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Cross-talk between NADPH oxidases and peroxisome proliferator-activated receptor-α differentially regulates left ventricular hypertrophy via specific activation of key signalling pathways.

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Alteration of neuropeptide Y1, Y2 and Y5 receptor expression and their functional consequences in diabetes. (Phys Soc Leeds “Cardiac & Respiratory Physiology Themed Meeting” 17-19 March 2008.)

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A possible role for transient receptor potential (melastatin) 8 (TRPM8) channels in human cutaneous blood flow (Irish J Med Sci. 177)

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Enhanced plasma stability, receptor binding, cyclic AMP production and insulin-releasing activity of two novel penultimate N-terminally substituted analogues of glucagon-like peptide-1.

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Comparative studies with exendin(9-39)amide and the novel (Pro3)GIP receptor antagonist indicates that glucose-dependent insulinotropic polypeptide is the major hormone of the enteroinsular axis.

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DPP IV degradation, receptor binding, insulin secreting and antihyperglycaemic characteristics of palmitate-dervitised GLP-1 analogues.

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(Lys9)GLP-1 lacks insulinotropic activity but acts as a potent novel GLP-1 receptor antagonist in vivo.

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Novel long-acting enzyme-resistant GIP receptor agonists with improved antihyperglycaemic and insulinotropic activity.

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Effects of long term activation of glucose-dependent insulinotropic polypeptide receptor on glucose tolerance and insulin secretion in normal mice.

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Long-acting glucose-dependent insulinotropic polypeptide receptor agonist restores beta-cell responsiveness in ageing mice.

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Early administration of the GIP receptor antagonist (Pro3)GIP counters development of hyperglycaemia, glucose intolerance and insulin resistance in genetic obesity.

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Cellular and metabolic evaluation of (hydroxyproline3)-GIP as new stable GIP receptor antagonist with antidiabetic potential

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Insulin-releasing and glucose homeostatic effects of a low molecular weight GLP-1 receptor agonist.

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