Corrosion Fatigue Behaviour of Laser-welded Shape Memory NiTi Wires in a Simulated Body Fluid

Corrosion fatigue is a fracture process as a consequence of synergistic interactions between the material structure, corrosive environment and cyclic loads/strains. It is difficult to be detected and can cause unexpected failure of engineering components in use. This study reveals a comparison of corrosion fatigue behaviour of laser-welded and bare NiTi wires using bending rotation fatigue (BRF) test coupled with a specifically-designed corrosion cell. The testing medium was Hanks’ solution (simulated body fluid) at 37.5 oC. Electrochemical impedance spectroscopic (EIS) measurement was carried out to monitor the change of corrosion resistance of sample during the BRF test at different periods of time. Experiments indicate that the laser-welded NiTi wire would be more susceptible to the corrosion fatigue attack than the bare NiTi wire. This study can serve as a benchmark for the product designers and engineers to understand the corrosion fatigue behaviour of the NiTi laser weld joint and determine the fatigue life safety factor for NiTi medical devices/implants involving laser welding in the fabrication process.

Automated retinal image quality assessment on the UK Biobank dataset for epidemiological studies

Morphological changes in the retinal vascular network are associated with future risk of many systemic and vascular diseases. However, uncertainty over the presence and nature of some of these associations exists. Analysis of data from large population based studies will help to resolve these uncertainties. The QUARTZ (QUantitative Analysis of Retinal vessel Topology and siZe) retinal image analysis system allows automated processing of large numbers of retinal images. However, an image quality assessment module is needed to achieve full automation. In this paper, we propose such an algorithm, which uses the segmented vessel map to determine the suitability of retinal images for use in the creation of vessel morphometric data suitable for epidemiological studies. This includes an effective 3-dimensional feature set and support vector machine classification. A random subset of 800 retinal images from UK Biobank (a large prospective study of 500,000 middle aged adults; where 68,151 underwent retinal imaging) was used to examine the performance of the image quality algorithm. The algorithm achieved a sensitivity of 95.33% and a specificity of 91.13% for the detection of inadequate images. The strong performance of this image quality algorithm will make rapid automated analysis of vascular morphometry feasible on the entire UK Biobank dataset (and other large retinal datasets), with minimal operator involvement, and at low cost.

Caractéristiques familiales associées à l'initiation précoce à la consommation de psychotropes

Cette recherche porte sur les caractéristiques familiales associées à l'initiation précoce à la consommation de psychotropes chez les enfants âgés de 10, 11 et 12 ans. Cette étude s'appuie sur le constat suivant : bien que la plupart des individus s'initient à la consommation de psychotropes au cours de l'adolescence ou au début de l'âge adulte, un certain nombre d'entre eux en consommeront dès l'enfance (Oxford, Harachi, Catalano et Abbott, 2000). Aussi, on note que depuis le début des années 1990, les jeunes manifesteraient de moins en moins leur désapprobation à l'égard de la consommation de psychotropes, augmentant ainsi le niveau d'inquiétude face à cette situation (Johnston, O'Malley et Bachman, 1999). Il est reconnu que les enfants qui s'initient à la consommation de psychotropes, tels que le tabac, l'alcool et la marijuana, avant l'âge de 12 ans augmentent singulièrement leurs risques de développer des problèmes de consommation (abus, dépendance) à l'adolescence et à l'âge adulte (Kuperman, Chan, Kramer, Bierut, Bucholz, Fox, Hesselbrock et al. , 2005; Lambert, 2005), et de présenter différents problèmes d'adaptation sur les plans personnel, familial et social (Kuperman et al. , 2005). II semble donc important de s'intéresser à cette problématique, d'autant plus que la période de développement prépubertaire est reconnue fondamentale, notamment sur le plan du développement social, émotionnel, physique, comportemental, cognitif et affectif (Thomassin, 2004). Afin d'intervenir en amont de ces difficultés et d'offrir des programmes de prévention adaptés aux enfants s'étant initiés précocement aux psychotropes, il s'avère essentiel de mieux connaître les caractéristiques familiales associées à ce phénomène (Gouvernement du Québec, 2001). Cela se justifie par le fait qu'il appartient au système familial d'exercer une influence positive afin que l'enfant adopte des comportements adaptés (Clark, Cornelius, Kirisci, et Tarter, 2005; Sung, Erkanli, Angold, et Costello, 2003; Thomassin, 2004). On retrouve effectivement différentes caractéristiques familiales qui seraient associées à l'initiation précoce des enfants à la consommation de psychotropes, notamment, la structure familiale (Saint-Jacques, Turcotte, Drapeau, Cloutier et Doré, 2004), les caractéristiques personnelles des parents dont la présence d'un problème avec la justice, de santé mentale ou de consommation de psychotropes, les pratiques éducatives lacunaires des parents à l'égard des enfants, ainsi qu'un faible engagement parental (Vitaro, Carbonneau, Gosselin, Tremblay et Zoccolillo, 2000). Les objectifs de l'étude sont donc : (1) d'identifier les caractéristiques familiales associées à l'initiation précoce à la consommation de psychotropes chez ces enfants et (2) d'identifier, parmi ces caractéristiques familiales, celles qui sont les plus fortement associées à une initiation précoce aux psychotropes."--Résumé abrégé par UMI.

Are Premorbid Abnormal Personality Traits Associated with Behavioural and Psychological Symptoms in Dementia?

Objective: The study aims to investigate associations between behavioural and psychological symptoms of dementia (BPSD) and abnormal premorbid personality traits. Methods: Data were obtained from 217 patients with a diagnosis of probable Alzheimer’s disease. Behavioural and psychological symptoms of late-onset dementia were assessed with the Neuropsychiatric Inventory. Premorbid personality traits were assessed using the Standardised Assessment of Personality. Abnormal premorbid personality traits were categorised with Diagnostic and Statistical Manual of Mental Disorders fourth edition and International Statistical Classification of Diseases and Related Health Problems—10 diagnostic criteria for personality disorders. Results: Abnormal premorbid personality traits were associated with increased behavioural and psychological symptoms in dementia. Cluster A (solitary/paranoid) premorbid personality traits were associated with anxiety, depression and hallucinations. Cluster C (anxious/dependent) traits were associated with a syndrome of depression. Conclusions: The presence of Clusters A (solitary/paranoid) and C (anxious/dependent) abnormal premorbid personality traits seems to affect the expression of certain behavioural and psychological symptoms in dementia, depression in particular. Copyright # 2016 John Wiley & Sons, Ltd

CarolFest 2008, December 2, 2008

Concert Program for CarolFest 2008, December 2, 2008

Language impairment in a case of a complex chromosomal rearrangement with a breakpoint downstream of FOXP2

BACKGROUND: We report on a young female, who presents with a severe speech and language disorder and a balanced de novo complex chromosomal rearrangement, likely to have resulted from a chromosome 7 pericentromeric inversion, followed by a chromosome 7 and 11 translocation. RESULTS: Using molecular cytogenetics, we mapped the four breakpoints to 7p21.1-15.3 (chromosome position: 20,954,043-21,001,537, hg19), 7q31 (chromosome position: 114,528,369-114,556,605, hg19), 7q21.3 (chromosome position: 93,884,065-93,933,453, hg19) and 11p12 (chromosome position: 38,601,145-38,621,572, hg19). These regions contain only non-coding transcripts (ENSG00000232790 on 7p21.1 and TCONS_00013886, TCONS_00013887, TCONS_00014353, TCONS_00013888 on 7q21) indicating that no coding sequences are directly disrupted. The breakpoint on 7q31 mapped 200 kb downstream of FOXP2, a well-known language gene. No splice site or non-synonymous coding variants were found in the FOXP2 coding sequence. We were unable to detect any changes in the expression level of FOXP2 in fibroblast cells derived from the proband, although this may be the result of the low expression level of FOXP2 in these cells. CONCLUSIONS: We conclude that the phenotype observed in this patient either arises from a subtle change in FOXP2 regulation due to the disruption of a downstream element controlling its expression, or from the direct disruption of non-coding RNAs.

Platelets induce Pai-1 mRNA expression in monocytes through TGF-BETA

Introduction: Plasminogen activator inhibitor type-1 (PAI-1) is a physiological modulator of fibrinolysis. High plasma PAI-1 is associated with the 4G/5G promoter polymorphism and with increased cardiovascular risk. Here we explored the role of platelets in regulating expression of the PAI-1 gene in monocytes. Methods: Blood from PAI-1 4G/5G genotyped volunteers (n=6) was incubated with the platelet GPVI-specific agonist, cross-linked collagen related peptide (CRP-XL), in the presence or absence of Mab 9E1 that blocks the binding of P-selectin to PSGL1. Monocytes were isolated by +ve selection on CD14 beads and monocyte PAI-1 mRNA expression was measured by real-time PCR. Results: Activation of platelets with CRP-XL resulted in platelets binding to >70% of monocytes and was accompanied by >5000-fold induction of PAI-1 mRNA, peaking at 4hrs. PAI-1 expression was independent of the 4G/5G genotype. Blocking the binding of platelets to monocytes enhanced PAI-1 induction (p<0.05 at 4 hrs). Incubation of isolated monocytes with the releasate from CRP-XL stimulated platelets also led to PAI-1 mRNA expression. The platelet secretome contains >100 different proteins. To identify the soluble factor(s) responsible for induction of PAI-1, neutralizing antibodies to likely candidates were added to monocytes incubated with the platelet releasate. Anti- TGF-beta inhibited platelet releasate-mediated PAI-1 mRNA induction by >80%. Monocyte PAI-1 was also induced by stimulation of PSGL-1 with a P-selectin-Fc chimera, in the absence of platelets, which was also blocked by the TGF-beta antibody. Conclusions: These results suggest that platelets induce PAI-1 mRNA in monocytes predominantly via TGF-beta, released from both platelets, and monocytes via activation by PSGL-1 signalling.This stimulation is independent of 4G/5G genotype

Consensus of Official Position of IOF/ISCD FRAX Initiatives in Asia-Pacific Region.

The fracture risk assessment tool (FRAX(®)) has been developed for the identification of individuals with high risk of fracture in whom treatment to prevent fractures would be appropriate. FRAX models are not yet available for all countries or ethnicities, but surrogate models can be used within regions with similar fracture risk. The International Society for Clinical Densitometry (ISCD) and International Osteoporosis Foundation (IOF) are nonprofit multidisciplinary international professional organizations. Their visions are to advance the awareness, education, prevention, and treatment of osteoporosis. In November 2010, the IOF/ISCD FRAX initiative was held in Bucharest, bringing together international experts to review and create evidence-based official positions guiding clinicians for the practical use of FRAX. A consensus meeting of the Asia-Pacific (AP) Panel of the ISCD recently reviewed the most current Official Positions of the Joint Official Positions of ISCD and IOF on FRAX in view of the different population characteristics and health standards in the AP regions. The reviewed position statements included not only the key spectrum of positions but also unique concerns in AP regions.

Subgroup-specific structural variation across 1,000 medulloblastoma genomes.

Medulloblastoma, the most common malignant paediatric brain tumour, is currently treated with nonspecific cytotoxic therapies including surgery, whole-brain radiation, and aggressive chemotherapy. As medulloblastoma exhibits marked intertumoural heterogeneity, with at least four distinct molecular variants, previous attempts to identify targets for therapy have been underpowered because of small samples sizes. Here we report somatic copy number aberrations (SCNAs) in 1,087 unique medulloblastomas. SCNAs are common in medulloblastoma, and are predominantly subgroup-enriched. The most common region of focal copy number gain is a tandem duplication of SNCAIP, a gene associated with Parkinson's disease, which is exquisitely restricted to Group 4α. Recurrent translocations of PVT1, including PVT1-MYC and PVT1-NDRG1, that arise through chromothripsis are restricted to Group 3. Numerous targetable SCNAs, including recurrent events targeting TGF-β signalling in Group 3, and NF-κB signalling in Group 4, suggest future avenues for rational, targeted therapy.

Gender stereotypes of personality : universal and accurate ?

Numerous studies have documented subtle but consistent sex differences in self-reports and observer-ratings of five-factor personality traits, and such effects were found to show well- defined developmental trajectories and remarkable similarity across nations. In contrast, very little is known about perceived gender differences in five-factor traits in spite of their potential implications for gender biases at the interpersonal and societal level. In particular, it is not clear how perceived gender differences in five-factor personality vary across age groups and national contexts and to what extent they accurately reflect assessed sex differences in personality. To address these questions, we analyzed responses from 3,323 individuals across 26 nations (mean age = 22.3 years, 31% male) who were asked to rate the five-factor personality traits of typical men or women in three age groups (adolescent, adult, and older adult) in their respective nations. Raters perceived women as slightly higher in openness, agreeableness, and conscientiousness as well as some aspects of extraversion and neuroticism. Perceived gender differences were fairly consistent across nations and target age groups and mapped closely onto assessed sex differences in self- and observer-rated personality. Associations between the average size of perceived gender differences and national variations in sociodemographic characteristics, value systems, or gender equality did not reach statistical significance. Findings contribute to our understanding of the underlying mechanisms of gender stereotypes of personality and suggest that perceptions of actual sex differences may play a more important role than culturally based gender roles and socialization processes.

PRDM1 is a tumor suppressor gene in natural killer cell malignancies.

Natural killer cell lymphoma (NKCL) constitutes a rare and aggressive form of non-Hodgkin lymphoma, and there is little insight into its pathogenesis. Here we show that PRDM1 is a tumor suppressor gene in NKCLs that is inactivated by a combination of monoallelic deletion and promoter CpG island hypermethylation. We observed monoallelic deletion of PRDM1 loci in 8 of 18 (44%) NKCL cases. The other allele showed significant promoter methylation in 12 of 17 (71%) cases. In support of its role as a tumor suppressor gene, the reconstitution of PRDM1 in PRDM1-null NK cell lines led to G2/M cell cycle arrest, increased apoptosis, and a strong negative selection pressure with progressive elimination of PRDM1-expressing cells, which was enhanced when IL-2 concentration is limiting. We observed a progressive increase in PRDM1 expression-in particular, PRDM1α-in normal NK cells in response to IL-2 and in normal NK cells activated with an engineered NK cell target, K562-Cl9-mb21, suggesting its role in NK cell homeostasis. In support of this role, knockdown of PRDM1 by shRNA in normal NK cells resulted in the positive selection of these cells. We identified MYC and 4-1BBL as targets of PRDM1 in NK cells. Disruption of homeostatic control by PRDM1 may be an important pathogenetic mechanism for NKCL.

The TREAT-NMD DMD Global Database: Analysis of More than 7,000 Duchenne Muscular Dystrophy Mutations.

Analyzing the type and frequency of patient-specific mutations that give rise to Duchenne muscular dystrophy (DMD) is an invaluable tool for diagnostics, basic scientific research, trial planning, and improved clinical care. Locus-specific databases allow for the collection, organization, storage, and analysis of genetic variants of disease. Here, we describe the development and analysis of the TREAT-NMD DMD Global database (http://umd.be/TREAT_DMD/). We analyzed genetic data for 7,149 DMD mutations held within the database. A total of 5,682 large mutations were observed (80% of total mutations), of which 4,894 (86%) were deletions (1 exon or larger) and 784 (14%) were duplications (1 exon or larger). There were 1,445 small mutations (smaller than 1 exon, 20% of all mutations), of which 358 (25%) were small deletions and 132 (9%) small insertions and 199 (14%) affected the splice sites. Point mutations totalled 756 (52% of small mutations) with 726 (50%) nonsense mutations and 30 (2%) missense mutations. Finally, 22 (0.3%) mid-intronic mutations were observed. In addition, mutations were identified within the database that would potentially benefit from novel genetic therapies for DMD including stop codon read-through therapies (10% of total mutations) and exon skipping therapy (80% of deletions and 55% of total mutations).

Matériaux pour une histoire du Blésois et du Vendomois, rassemblés par « monsieur BEGON, docteur de la maison et société de Sorbonne, sous-doyen dans l'eglise de S. Sauveur de Blois ». Français 5679

Contient : Notes sur l'abbaye de Fontaines les Blanches. Au feuillet 15 r° dessin à l'encre des armoiries de ladite abbaye ; Notes sur l'abbaye de La Guiche ; Notes sur l'abbaye de Lepvroux ; Note sur l'abbaye du Landais ; Notes sur Moulin, sous Lepvroux ; Note sur Buzançois ; Notes sur l'abbaye de Marmoutiers ; Notes sur l'abbaye de Pontlevoy. Au feuillet 127, « cachet de Pontlevoy ». Au feuillet 130 r°, dessin à l'encre des armes de Pontlevoy. Le feuillet 131 est occupé par une lettre autographe signée : « GIRARDEAU », datée de Pontlevoy, 14 mars 1678, et adressée à l'abbé Bégon. Les feuillets 132-133 sont occupés par une lettre autographe signée : « Fr. MARTIN FILANE, m. b. », datée de Pontlevoy, 7 mai 1678, et adressée à M. l'abbé Bégon. Au fol. 138-139, lettre incomplète datée de Monteaux, 1er juillet 1678. Au fol. 144-145, lettre autographe signée : « P. GIRARDEAU » et datée de Blois, 29 avril 1678, à l'abbé Bégon ; Notes sur l'abbaye de S. Calais ; Notes sur l'abbaye de S. Laumer. Au feuillet 203, dessin à l'encre intitulé : « Main de justice, non sine causa ». Aux feuillets 208, 208 bis et 209, dessins à l'encre représentant les armoiries de l'abbaye de S. Lomer et des « tombeaux de l'eglise de S. Laumer de Blois », savoir : « armes de M. l'abbé de Morvilliers » ; armes de « Guy Pot, chambellan de Charles VIII » ; « armes de Mr de La Vieuville, evesque de Rennes et abbé de S. Lomer ; armes de « monseigneur le cardinal de Sourdy, archevesque de Bourdeaux, abbé de S. Lomer » ; « armes de Mr Fouquet de La Varenne, abbé de S. Lomer, qui fit retablir et recouvrir l'eglise de S. Laumer, l'an 1610 » ; Lettre autographe, signée : « B. CAMUSAT » et datée de Paris, 8 janvier 1681. A l'abbé Bégon ; Armes de la ville de Vendôme, dessinées à l'encre ; Note sur S. Dyé, envoyée à l'abbé Bégon, avec, à la fin, quelques lignes autographes, signées : « POIRIER, presbtre, curé de St Dyé », et datées de S. Dyé, le 7 juillet 1679 ; Lettre autographe signée : « GAIGNAISON, curé de Thenay », et datée de Thenay le 25 août 1678. A l'abbé Bégon ; Note sur la paroisse de Centenay, écrite par le « prieur curé de Centenay », qui signe : « HONYER » ; Note sur la « paroisse de Fleuré », par le curé de Fleuré ; Note sur S. Claude de Diray, écrite par le curé dudit lieu qui signe : « VACHER, curé de S. Claude de Diray », 3 juillet 1682. A l'abbé Bégon ; Lettre autographe signée : « GRYMAULDI,... à monsieur, monsieur Begon, conseiller et secretaire du roy, maison et couronne de France, à Paris... A Blois, ce IX apvril 1653 » ; Lettre autographe datée : « Au Mans, le 28 aoust 1680, et signée : « ETTE. chan. ind. de St Pierre ». A l'abbé Begon ; Note sur la paroisse de Monts, envoyée à l'abbé Bégon par le curé de cette paroisse, dans une lettre dont la signature est mutilée ; Note sur la paroisse de Chambon par le curé de Chambon, qui a signé : « BLE » ; Lettre autographe de l'abbé Bégon à son père. Elle est datée de Paris, 28 juin 1662. La signature, si toutefois ladite lettre était signée, a été enlevée par une mutilation du feuillet 398

今古奇觀Jin gu qi guan.Merveilles de l'antiquité et des temps modernes.

Contient : I, livre 1三孝廉讓產立高名San xiao lian rang chan li gao ming.Les trois licenciés charitables qui acquièrent de la célébrité ; II, livre 2兩縣令競義婚孤女Liang xian ling jing yi hun gu nü.L'orpheline ; III, livre 3滕大尹鬼斷家私Teng da yin gui duan jia si.Le portrait de famille ; IV, livre 4裴晉公義還原配Pei jin gong yi huan yuan phei.Comment le mandarin Dan bi perdit et retrouva sa fiancée ; V, livre 5杜十娘怒沈百寶箱Du shi niang nu chen bai bao xiang.Du Shi niang, de colère, jette dans l'eau la cassette de bijoux ; VI, livre 6李謫仙醉草嚇蠻書Li zhe xian zui cao he man shu.Le poète Li Tai bai ; VII, livre 7賣油郎獨占花魁Mai you lang du zhan hua khoei.Le vendeur d'huile qui seul possède la reine de beauté ; VIII, livre 8灌園叟（alias 吏）晚逢仙女Guan yuan sou (alias li) wan feng xian nü.Les pivoines ; IX, livre 9轉運漢巧遇洞庭紅Zhuan yun han qiao yu dong ting hong.Le couli des transports, adroitement, reçoit la beauté du Dong ting ; X, livre 10看財奴刁買寃家主Kan cai nu diao mai yuan jia zhu.Richesse mal acquise (Comment le ciel donne et reprend les richesses) ; XI, livre 11吳保安棄家贖友Wu bao an qi jia shu you.Véritable amitié ; XII, livre 12羊角哀舍命全交Yang jue ai she ming quan jiao.Yang Jue ai fait le sacrifice do sa vie par dévouement pour un ami ; XIII, livre 13沈小霞相會出師表Shen xiao xia xiang hui chu shi biao.Shen Xiao xia rencontre et présente le modèle des maîtres ; XIV, livre 14宋金郎團圓破氊笠Song jin lang tuan yuan po zhan li.Les tendres époux ; XV, livre 15盧太學詩酒傲公侯Lu tai xue shi jiu ao gong hou.Lu tai xue, poète et ivre, brave les princes ; XVI, livre 16李汧公窮邸遇俠客Li qian gong qiong di yu xie ke.Li Qian gong, dans sa résidence misérable, traite un hôte magnanime ; XVII, livre 17蘇小妹三難新郎Su xiao mei san nan xin lang.La jeune Su trois fois maltraite un nouveau marié ; XVIII, livre 18劉元普雙生貴子Liu yuan pu shuang sheng gui zi.Liu Yuan pu obtient deux beaux enfants ; XIX, livre 19俞伯牙摔琴謝知音Yu bai ya choai qin xie zhi yin.Le luth brisé ; XX, livre 20莊子休鼓盆成大道Zhuang zi xiu gu phen cheng da dao.La matrone du pays de Song ; XXI, livre 21老門生三世報恩Lao men sheng san shi bao en.Le vieil élève montre sa reconnaissance à la troisième génération ; XXII, livre 22鈍秀才一朝交泰Dun xiu cai yi zhao jiao tai.Le bachelier obtus tout d'un coup exerce son influence formatrice ; XXIII, livre 23蔣興哥重會珍珠衫Jiang xing ge chong hui zheng zhu shan.Le négociant ruiné (La tunique de perles) ; XXIV, livre 24陳御史巧勘金釵鈿Chen yu shi qiao kan jin tchhai tian.Une cause célèbre ; XXV, livre 25徐老僕義憤成家Xu lao bu yi fen cheng jia.Un serviteur méritant ; XXVI, livre 26蔡小姐忍辱報讐Cai xiao jie ren ru bao chou .L'héroïsme de la piété filiale ; XXVII, livre 27錢秀才錯占鳳凰儔.Qian xiu cai tsho zhan feng huang chou .Mariage forcé ; XXVIII, livre 28喬太守亂㸃鴛鴦譜.Qiao tai shou luan dian yuan yang pu.Le préfet Qiao pointe à tort le registre des unions ; XXIX, livre 29懷私怨很僕告主Huai si yuan hen bu gao zhu.Le crime puni ; XXX, livre 30念親恩孝女藏兒Nian qin en xiao nü cang er.La calomnie démasquée ; XXXI, livre 31呂大郎還金完骨肉Lü da lang huan jin wan gu ru.Les trois frères ; XXXII, livre 32金玉奴棒打薄情郎.Jin yu nu bang da bai qing lang.Femme et mari ingrats ; XXXIII, livre 33唐解元玩世出奇.Tang jie yuan wan shi chu qi.Le mariage du licencié Tang (Tang le jie yuan) ; XXXIV, livre 34女秀才移花接木.Nü xiu cai yi hua jie mu.La bachelière du pays de Zhu ; XXXV, livre 35王喬鸞百年長恨.Wang qiao luan bai nian chang hen.Le ressentiment perpétuel de Wang Qiao luan ; XXXVI, livre 36十三郎五歲朝天.Shi san lang wu sui chao tian.Shi san lang pendant cinq ans se tourne vers le ciel ; XXXVII, livre 37崔俊臣巧合芙蓉屏.Cui jun chen qiao he fu yong ping.Paravent révélateur ; XXXVIII, livre 38趙縣君喬送黃柑子.Zhao xian jun qiao song huang gan zi.Chantage ; XXXIX, livre 39誇妙術丹容提金.Khoa miao shu dan yong ti jin.Les alchimistes ; XL, livre 40逞多財（alias 錢多） 白丁橫帶Cheng duo cai (qian duo) bai ding heng dai.Cupidité et dérèglement

今古奇觀Jin gu qi guan.Merveilles de l'antiquité et des temps modernes.

Contient : I, livre 1三孝廉讓產立高名San xiao lian rang chan li gao ming.Les trois licenciés charitables qui acquièrent de la célébrité ; II, livre 2兩縣令競義婚孤女Liang xian ling jing yi hun gu nü.L'orpheline ; III, livre 3滕大尹鬼斷家私Teng da yin gui duan jia si.Le portrait de famille ; IV, livre 4裴晉公義還原配Pei jin gong yi huan yuan phei.Comment le mandarin Dan bi perdit et retrouva sa fiancée ; V, livre 5杜十娘怒沈百寶箱Du shi niang nu chen bai bao xiang.Du Shi niang, de colère, jette dans l'eau la cassette de bijoux ; VI, livre 6李謫仙醉草嚇蠻書Li zhe xian zui cao he man shu.Le poète Li Tai bai ; VII, livre 7賣油郎獨占花魁Mai you lang du zhan hua khoei.Le vendeur d'huile qui seul possède la reine de beauté ; VIII, livre 8灌園叟（alias 吏）晚逢仙女Guan yuan sou (alias li) wan feng xian nü.Les pivoines ; IX, livre 9轉運漢巧遇洞庭紅Zhuan yun han qiao yu dong ting hong.Le couli des transports, adroitement, reçoit la beauté du Dong ting ; X, livre 10看財奴刁買寃家主Kan cai nu diao mai yuan jia zhu.Richesse mal acquise (Comment le ciel donne et reprend les richesses) ; XI, livre 11吳保安棄家贖友Wu bao an qi jia shu you.Véritable amitié ; XII, livre 12羊角哀舍命全交Yang jue ai she ming quan jiao.Yang Jue ai fait le sacrifice do sa vie par dévouement pour un ami ; XIII, livre 13沈小霞相會出師表Shen xiao xia xiang hui chu shi biao.Shen Xiao xia rencontre et présente le modèle des maîtres ; XIV, livre 14宋金郎團圓破氊笠Song jin lang tuan yuan po zhan li.Les tendres époux ; XV, livre 15盧太學詩酒傲公侯Lu tai xue shi jiu ao gong hou.Lu tai xue, poète et ivre, brave les princes ; XVI, livre 16李汧公窮邸遇俠客Li qian gong qiong di yu xie ke.Li Qian gong, dans sa résidence misérable, traite un hôte magnanime ; XVII, livre 17蘇小妹三難新郎Su xiao mei san nan xin lang.La jeune Su trois fois maltraite un nouveau marié ; XVIII, livre 18劉元普雙生貴子Liu yuan pu shuang sheng gui zi.Liu Yuan pu obtient deux beaux enfants ; XIX, livre 19俞伯牙摔琴謝知音Yu bai ya choai qin xie zhi yin.Le luth brisé ; XX, livre 20莊子休鼓盆成大道Zhuang zi xiu gu phen cheng da dao.La matrone du pays de Song ; XXI, livre 21老門生三世報恩Lao men sheng san shi bao en.Le vieil élève montre sa reconnaissance à la troisième génération ; XXII, livre 22鈍秀才一朝交泰Dun xiu cai yi zhao jiao tai.Le bachelier obtus tout d'un coup exerce son influence formatrice ; XXIII, livre 23蔣興哥重會珍珠衫Jiang xing ge chong hui zheng zhu shan.Le négociant ruiné (La tunique de perles) ; XXIV, livre 24陳御史巧勘金釵鈿Chen yu shi qiao kan jin tchhai tian.Une cause célèbre ; XXV, livre 25徐老僕義憤成家Xu lao bu yi fen cheng jia.Un serviteur méritant ; XXVI, livre 26蔡小姐忍辱報讐Cai xiao jie ren ru bao chou .L'héroïsme de la piété filiale ; XXVII, livre 27錢秀才錯占鳳凰儔.Qian xiu cai tsho zhan feng huang chou .Mariage forcé ; XXVIII, livre 28喬太守亂㸃鴛鴦譜.Qiao tai shou luan dian yuan yang pu.Le préfet Qiao pointe à tort le registre des unions ; XXIX, livre 29懷私怨很僕告主Huai si yuan hen bu gao zhu.Le crime puni ; XXX, livre 30念親恩孝女藏兒Nian qin en xiao nü cang er.La calomnie démasquée ; XXXI, livre 31呂大郎還金完骨肉Lü da lang huan jin wan gu ru.Les trois frères ; XXXII, livre 32金玉奴棒打薄情郎.Jin yu nu bang da bai qing lang.Femme et mari ingrats ; XXXIII, livre 33唐解元玩世出奇.Tang jie yuan wan shi chu qi.Le mariage du licencié Tang (Tang le jie yuan) ; XXXIV, livre 34女秀才移花接木.Nü xiu cai yi hua jie mu.La bachelière du pays de Zhu ; XXXV, livre 35王喬鸞百年長恨.Wang qiao luan bai nian chang hen.Le ressentiment perpétuel de Wang Qiao luan ; XXXVI, livre 36十三郎五歲朝天.Shi san lang wu sui chao tian.Shi san lang pendant cinq ans se tourne vers le ciel ; XXXVII, livre 37崔俊臣巧合芙蓉屏.Cui jun chen qiao he fu yong ping.Paravent révélateur ; XXXVIII, livre 38趙縣君喬送黃柑子.Zhao xian jun qiao song huang gan zi.Chantage ; XXXIX, livre 39誇妙術丹容提金.Khoa miao shu dan yong ti jin.Les alchimistes ; XL, livre 40逞多財（alias 錢多） 白丁橫帶Cheng duo cai (qian duo) bai ding heng dai.Cupidité et dérèglement