Mischnath ha-mmiddoth : aus einem Manuscripte der Münchener Bibliothek, bezeichnet Cod. Hebr. 36, als erste geometrische Schrift in hebräischer Sprache

hrsg. u. mit eigenen Bemerkungen versehen von M. Steinschneider ; ins Deutsche übers., erläutert und mit einem Vorwort versehen von Hermann Schapira

Overtura : per 2 violini, alto, violoncello e basso, 2 flauti, 2 clarini, 2 oboi, 2 fagotti, 2 corni, 2 trombe, trombone, basso e timbani ; op. 36

composta da Aloys Schmitt

Inc. hebr. 36 - Sefer ha-Mitsṿot gadol

Vorbesitzer: Abraham Merzbacher

{[N.N.], Rollenbild, Brustbild, in: Hanns Johst: Thomas Paine, 1935/36

Signatur des Originals: S 36/F11352

Na 1 Nachlass Max Horkheimer, 652 - Vorlesungen: "National Socialism", "Autoritarian doctrines and modern European institutions", "Eclipse of Reason" (p. IX 35a.1-2-IX 36.1-5b)

"National Socialism": 1. Ankündigung einer Vorlesungsreihe November/Dezember 1941 von: Herbert Marcuse, A.R.L. Gurland, Franz Neumann, Otto Kirchheimer, Frederick Pollock. a) als Typoskript verfielfältigt, 1 Blatt, b) Typoskript, 1 Blatt; 2. Antwortbrief auf Einladungen zur Vorlesungsreihe, von Neilson, William A.; Packelis, Alexander H.; Michael, Jerome; McClung Lee, Alfred; Youtz, R.P.; Ginsburg, Isidor; Ganey, G.; Nunhauer, Arthur. 8 Blätter; "Autoritarian doctrines and modern European institutions" (1924): 1. Vorlesungs-Ankündigung Typoskript, 2 Blatt; 2. Ankündigungen der Vorlesungen von Neumann, Franz L.: "Stratification and Dominance in Germany"; "Bureaucracy as a Social and Political Institution", Typoskript, 2 Blatt; 3. Evans, Austin P.: 1 Brief (Abschrift) an Frederick Pollock, New York, 26.2.1924; "Eclipse of Reason", Fünf Vorlesungen 1943/44:; 1. I. Lecture. a) Typoskript mit eigenhändigen Korrekturen, 38 Blatt b) Typoskript, 29 Blatt c) Typoskript mit eigenhändigen und handschriftlichen Korrekturen, 31 Blatt d) Teilstück, Typoskript mit eigenhändigen Korrekturen, 2 Blatt e) Entwürfe, Typoskript mit eigenhändigen Korrekturen, 6 Blatt; 2. II. Lecture. a) Typoskript mit eigenhändigen Korrekturen, 27 Blatt, b) Typoskript mit handschriftlichen Korrekturen, 37 Blatt; 3. III. Lecture. Typoskript mit eigenhändigen Korrekturen, 27 Blatt; 4. IV. Lecture. Typoskript mit eigenhändigen Korrekturen, 23 Blatt; 5. V. Lecture. a) Typoskript mit eigenhändigen Korrekturen, 25 Blatt, b) Teilstücke, Typoskript mit eigenhändigen und handschriftlichen Korrekturen, 3 Blatt;

Na 1 Nachlass Max Horkheimer, 653 - Vorlesungen: "Eclipse of Reason"; "Society and Reason" (p. IX 36.6-12)

6. Vorlesungen 1. - 5. mit dem Titel "Society and Reason". a) Typoskript mit handschriftlichen Randbemerkungen von Theodor W. Adorno, 139 Blatt b) Typoskript, 139 Blatt; 7. Teilstück, Typoskript mit eigenhändigen Korrekturen, 1 Blatt; 8. Eigenhändige Notizen, 4 Blatt; 9. Stichworte zu den Vorlesungen I-III. Typoskript mit eigenhändigen Ergänzungen, 2 Blatt a) Einladungskarte zur Vorlesungsreihe "Society and Reason". Als Typoskript vervielfältigt, 1 Blatt b) Gliederung. Typoskript, 1 Blatt; 10. "Über Probleme des wissenschaftlichen Stils". Reaktion auf ein Schriftstück (aus einem Brief an Friedrich Pollock). a) Typoskript, datiert: 28.11.1943, 3 Blatt b) Max Horkheimer: 1 Brief an Friedrich Pollock, ohne Ort, 28.11.1943, 5 Blatt; 11. Columbia University, New York: 1 Brief (Abschrift) an Max Horkheimer, New York, 5.3.1943, und Stichworte zu den Vorlesungen, 2 Blatt; 12. Zeitungsausschnitt, 1 Blatt.;

Na 1 Nachlass Max Horkheimer, 585 - Korrespondenzen mit Friedrich (Frederick) Pollock (p. VI 36, 1-109)

1 Brief von Frederick Pollock an Hedwig Cornelius, 11.05.1948;

Na 1 Nachlass Max Horkheimer, 586 - Korrespondenzen mit Friedrich (Frederick) Pollock (p. VI 36, 110-334)

1 Brief von Frederick Pollock an Walter Munding, 16.08.1950; 1 Brief von Frederick Pollock an Joseph Christ, 24.05.1950; 1 Brief von Albert Flegenheimer an Frederick Pollock, 18.05.1950; 2 Briefe von Frederick Pollock an E. Wehrle, 03.04.1950; 1 Brief von Margot von Mendelssohn an Max Horkheimer, 18.01.1950; 1 Brief von Nothern Life Insurance Co. (Seattle) an Max Horkheimer, 16.01.1950;

Na 1 Nachlass Max Horkheimer, 587 - Korrespondenzen mit Friedrich (Frederick) Pollock (p. VI 36, 335-401)

2 Briefe zwischen A. P. Bersohn und Frederick Pollock, 1952; 1 Brief von Albert Ehrenzweig an Frederick Pollock, 01.06.1952; 1 Brief von Frederick Pollock an Leo Löwenthal, 20.12.1951; 1 Telegramm von Franz Neumann an Frederick Pollock, 11.05.1951;

Ms. lat. qu. 97 Bd. 1 - Memorie originali risguardanti le belle arti, Nr. 1 - 36

Vorbesitzer: Michelangelo Gualandi;

Ms. lat. oct. 36 - Aerarium medicum

Vorbesitzer: Johannes Matthaeus Valentini

Ms. lat. qu. 36 - Epistolae

Vorbesitzer: M. G. T.;

Die Heiligung des göttlichen Namens : ein Kanzelvortrag über Ezechiel 36, 17-26

von Salomon Marcus Schiller,

Tumor heterogeneity dictates sensitivity to gefitinib (Iressa(TM)/ZD1839) in solid tumor models

The epidermal growth factor receptor (EGFR) and its ligands are overexpressed in many human tumors, including bladder and pancreas, correlating with a more aggressive tumor phenotype and poor patient prognosis. We initiated the present study to characterize the heterogeneity of gefitinib responsiveness in a panel of human bladder and pancreatic cancer cell lines in order to identify the biological characteristics of EGFR-dependent proliferation that could be used to prospectively identify drug-sensitive tumors. A second objective was to elucidate how to best exploit these results by utilizing gefitinib in combination therapy. To these ends, we examined the effects of the EGFR antagonist gefitinib on proliferation and apoptosis in a panel of 18 human bladder cancer cell lines and 9 human pancreatic cancer cell lines. Our data confirmed the existence of marked heterogeneity in Iressa responsiveness with less than half of the cell lines displaying significant growth inhibition by clinically relevant concentrations of the drug. Gefitinib responsiveness was found to be p27 kip1 dependent as DNA synthesis was restored following exposure to p27siRNA. Unfortunately, Iressa responsiveness was not closely linked to surface EGFR or TGF-α expression in the bladder cancer cells, however, cellular TGF-α expression correlated directly with Iressa sensitivity in the pancreatic cancer cell lines. These findings provide the potential for prospectively identifying patients with drug-sensitive tumors. ^ Further studies aimed at exploiting gefitinib-mediated cell cycle effects led us to investigate if gefitinib-mediated TRAIL sensitization correlated with increased p27kip1 accumulation. We observed that increased TRAIL sensitivity following gefitinib exposure was not dependent on p27 kip1 expression. Additional studies initiated to examine the role(s) of Akt and Erk signaling demonstrated that exposure to PI3K or MEK inhibitors significantly enhanced TRAIL-induced apoptosis at concentrations that block target phosphorylation. Furthermore, combinations of TRAIL and the PI3K or MEK inhibitors increased procaspase-8 processing above levels observed with TRAIL alone, indicating that the effects were exerted at the level of caspase-8 activation, considered the earliest step in the TRAIL pathway. ^

Adenovirus-36 infection and obesity related hormones in children

Obesity has a complex, multi-factorial etiology. Infectious agents have recently emerged as a possible contributor to the current obesity epidemic. Seven viruses have demonstrated an association with obesity in animals; however, Adenovirus-36 (Ad-36) is the only known virus associated with obesity in humans. The primary aim of this research was to determine the association between Ad-36 infection and the expression of obesity related hormones in children. Additionally, this study proposed to compare the mean three year change in the level of obesity related hormones between Ad-36 positive and negative children. This study utilized pilot data collected from 98 children at baseline and year three of the Project Heartbeat! cohort. Fasting serum samples were analyzed for the concentration of adiponectin, insulin and leptin. The crude analysis uncovered Ad-36 positive children had significantly lower mean concentrations of insulin (p=0.039) and leptin (p=0.038) at baseline compared to Ad-36 negative children. The results of the adjusted analysis indicated the leptin association with Ad-36 infection at baseline was statistically significant even after controlling for age, sex, ethnicity, and BMI percentile. The longitudinal evaluation revealed individuals with a history of Ad-36 infection experienced a larger mean decrease in adiponectin, a larger mean increase in leptin, and a smaller mean increase in insulin levels over a three year period compared to individuals without a history of infection. These results suggest Ad-36 infection may produce changes in hormone expression. The only statistically significant findings in the crude and adjusted longitudinal analysis occurred at baseline when the children were younger, suggesting physical changes that occur during sexual maturation may mask or enhance Ad-36 induced changes in hormone expression. Furthermore, the longitudinal analysis revealed the duration and course of Ad-36 infection may influence changes in the expression of obesity-related hormones. Taken together, the results of this pilot study are suggestive of an association between Ad-36 infection and the expression of obesity-related hormones.^