988 resultados para sexual differentiation
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Tesis (Maestría en Psicología Laboral) U.A.N.L.
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Tesis (Maestría en Derecho Penal) U.A.N.L.
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Tesis (Maestría en Área Específica Clínica Psicoanalítica) UANL, 2010.
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Tesis (Maestría en Ciencias de Enfermería) UANL, 2011.
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Tesis (Maestría en Ciencias de Enfermería) UANL, 2014.
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Tesis (Maestría en Ciencias de Enfermería) UANL, 2014.
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Tesis (Maestría en Ciencias de Enfermería) UANL, 2014.
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Tesis (Maestría en Ciencias de Enfermería) UANL, 2014.
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Tesis (Maestría en Psicología con orientación en Clínica Psicoanalítica) UANL, 2014.
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Tesis (Maestría en Psicología con orientación en Terapia Breve) UANL, 2014.
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UANL
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BACKGROUND: The role of ss-catenin signaling in mesodermal lineage formation and differentiation has been elusive. METHODOLOGY: To define the role of ss-catenin signaling in these processes, we used a Dermo1(Twist2)(Cre/+) line to target a floxed beta-catenin allele, throughout the embryonic mesenchyme. Strikingly, the Dermo1(Cre/+); beta-catenin(f/-) conditional Knock Out embryos largely phenocopy Pitx1(-/-)/Pitx2(-/-) double knockout embryos, suggesting that ss-catenin signaling in the mesenchyme depends mostly on the PITX family of transcription factors. We have dissected this relationship further in the developing lungs and find that mesenchymal deletion of beta-catenin differentially affects two major mesenchymal lineages. The amplification but not differentiation of Fgf10-expressing parabronchial smooth muscle progenitor cells is drastically reduced. In the angioblast-endothelial lineage, however, only differentiation into mature endothelial cells is impaired. CONCLUSION: Taken together these findings reveal a hierarchy of gene activity involving ss-catenin and PITX, as important regulators of mesenchymal cell proliferation and differentiation.
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Tesis (Doctorado en Filosofía con Especialidad en Trabajo Social y Políticas Comparadas de Bienestar Social) UANL
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Although some are excited about the possibility of using current scientific research into the biological causes of sexual orientation to ground rights claims, I argue that basing rights claims on this research is unwise because this research, specifically the hormonal, genetic, and structural research, is organized around the inversion assumption, a conceptual scheme within which some aspect of the biology of gay men and lesbians is thought to be inverted along sex lines.While there are many reasons to worry about the use of the inversion assumption, I focus on problems that arise from a further set of claims that must be assumed in order to make the use of the inversion assumption coherent. This further set of assumptions includes the claims (1) that heterosexuality is the standard state and that (2) this standard state is sexually-dimorphic and (3) deterministic. I argue that this set of assumptions is problematic because it results in ideological consequences that are both sexist and heterosexist.
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Tesis (Doctor en Ciencias de Enfermería) UANL, 2012.