Alzheimer's disease : a report to the legislature in response to resolution 513 /

Caption title.

Alzheimer's disease : a guide to services for families /

"December 1985."

An evaluation of the training program: "The Alzheimer's disease afflicted: understanding the disease and the resident" /

On cover: New horizons in long term care.

Illinois Alzheimer's disease assistance plan.

Description based on: 1994; title from cover.

The Alzheimer disease afflicted : understanding the disease and the resident instructors guide and teaching materials /

On cover: New horizons in long term care.

Women's health beat /

Contains 32 Spanish language pamphlets which use a Q and A (Question/Answer) format to provide information on various women's health topics.

La enfermedad de los místicos : patología-psíquica /

Includes bibliographical references.

Q & A : Alzheimer's disease.

Mode of access: Internet.

Moderating Variables Impacting Adherence to the Reducing Disability in Alzheimer's Disease (RDAD) Intervention

Thesis (Ph.D.)--University of Washington, 2016-06

Sequence variation in the proximity of IDE may impact age at onset of both Parkinson disease and Alzheimer disease

We recently reported that a linkage disequilibrium (LD) block on chromosome 10q encompassing the gene encoding insulin-degrading enzyme (IDE) harbors sequence variants that associate with Alzheimer disease (AD). Evidence also indicated effects upon a number of quantitative indices of AD severity, including age-at-onset (AAO). Since linkage of this immediate region to AAO has been shown in both AD and Parkinson disease (PD), we have explored the possibility that polymorphism within this LD block might also influence PD. Utilizing single nucleotide polymorphisms that delineate common haplotypes from this region, we observed significant evidence of association with AAO in an Australian PD case-control sample. Analyses were complemented with AAO data from two independent Swedish AD case samples, for which previously reported findings were replicated. Results were consistent between AD and PD, suggesting the presence of equivalent detrimental and protective alleles. These data highlight a genomic region in the proximity of IDE that may contribute to AD and PD in a similar manner.