Vuorineuvos Tapio Koski 17/4 1974

Kirje 17.4.1974

Fil.maisteri Irma Karvikko 7/12 1972

Kirje 7.12.1972

Kirjailija Lassi Nummi 5/12 1974

Kirje 5.12.1974

Matti Ahtiainen 28/8 1972

Kirje 28.8.1972

Eversti evp. Alpo K. Marttinen 8/5 1973

Kirje 8.5.1973

Professori Yrjö Blomstedt 12/2 1969

Kirje 12.2.1969

Maanviljelijä Kalle Keränen 15/6 1970

Kirje 15.6.1970

Rakas Heini 22/11 1974

Kirje 22.11.1974

Respiratory effects of an exposure to grain dust among grain workers in the Vaud region (Switzerland)

Introduction: Bioaerosols such as grain dust (GD) elicit direct immunological reactions within the human respiratory system. Workplace-dependent exposure to GD may induce asthma, chronic bronchitis, and hypersensitivity pneumonitis. Aims: To assess the clinical impact of occupational exposure to GD and to determine quantitative biological markers of bioaerosol exposure in grain workers. Methods: This longitudinal study has been conducted from summer 2012 to summer 2013, comprising 6 groups of 30 active workers with different GD exposure patterns (4 groups of grain workers, 2 control groups). Two evaluations at high- and low-exposing seasons take place, during which an occupational and a medical history are questionnaire-assessed, lung function is evaluated by spirometry, airway inflammation is measured by exhaled nitric oxide (eNO) and specific blood IgG and IgE are titrated. Results: The preliminary results are those of 2 of the 4 exposed groups, (harvesters and mill workers), compared to the control groups, at first assessment (n=100). Mean age is 38.4 [years]; 98% are male. Exposed groups differ from controls (p<0.05) in daily contact with animals (57% vs. 40%) and active smoking (39% vs. 11%). Grain workers have more respiratory (50%), nasal (57%), ocular (45%) and dermatologic (36%) occupational symptoms than controls (6.4%, 19%, 16%, 6.4% respectively, p<0.05). Lower mean peak-expiratory-flow (PEF) values (96.1 ± 18.9 vs. 108.2 ± 17.4 [% of predicted], p<0.05) and eNO values (13.9 ± 9.6 vs. 20.5 ± 14.7 [ppm], p<0.05) are observed in the exposed groups. Conclusion: Preliminary results show a higher prevalence of clinical symptoms and a lower mean PEF value in the groups exposed to GD.

Nimimerkki "Vanha Kissa" 30/8 1971

Kirje 30.8.1971

Piispa Olavi Kares 25/5 1972

Kirje 25.5.1972

Hajahuomioita päivän poliittisista näkymistä 5/4 1975

Kirje 5.4.1975

Tohtori Matti Aro 23/8 1971

Kirje 23.8.1971

Herra Henry Delias 25/10 1973

Kirje 25.10.1973

Pääjohtaja Pekka Kuusi 24/8 1973

Kirje 24.8.1973