¿Jugamos con el Brain Training?

Esta experiencia, llevada a cabo con los alumnos de tercero de ESO del IES Forum 2004, de Sant Adriá de Besós (Barcelona), pretendía analizar si el uso de la consola con el juego Brain Training facilita el desarrollo del cálculo mental. Se constata que se trata de una herramienta útil que propicia el interés y el grado de participación de los alumnos.

The thinking child resource book : brain-based learning for the early years foundation stage.

Ofrece a los profesores, estudiantes y padres, prácticas y sugerencias para la aplicación del aprendizaje, basado en cómo está configurado el cerebro del niño en sus primeros años de vida. Se enumeran ideas sobre cómo usar técnicas de aprendizaje que incluyen las formas de promover la autoestima y la inteligencia emocional, las ideas para la enseñanza a través del juego, música y movimiento, actividades para la hora de la asamblea, asesoramiento sobre el manejo de la conducta positiva y fomento de las relaciones con padres y cuidadores. Los estudios de casos, tomados de una variedad de entornos, dan una idea de cómo las técnicas basadas en el cerebro pueden ser utilizados para enriquecer la experiencia de aprendizaje de los niños pequeños.

Kinesiología educativa : gimnasia para el cerebro-brain gym.

La 'Kinesiología Educativa' es un sistema de aprendizaje y enseñanza integral que consiste en una serie de ejercicios sensorio-motores sencillos que estimulan áreas cerebrales específicas y fomentan las conexiones neurológicas. Aquí, se presenta una aplicación práctica de la 'Kinesiología Educativa' a los alumnos de un centro escolar y los resultados obtenidos. La aplicación de este sistema en alumnos de educación infantil y primaria del centro ha permitido que alcancen mejoras en áreas como matemáticas y ciencias, además ha aumentado la capacidad de memoria de los alumnos y su autoestima.

The powerful pull of brain images. 'El enorme poder de atracción de las imágenes cerebrales'.

Resumen basado en el de la publicación

Using the brain to understand the developing mind : commentary on the article 'Where developmental psychology and neuroscience meet : a threatening or a felicitous encounter' by María-José Rodrigo. 'Usar el cerebro para comprender el desarrollo de la mente : un comentario al artículo 'Donde la psicología evolutiva y la neurociencia se encuentran : ¿un encuentro amenazador u oportuno?' de María-José Rodrigo'.

Resumen basado en el de la publicación

Social perception gaze patterns, symptom severity and resting brain function measured using Arterial Spin Labelling

Autistic spectrum disorder (ASD) is characterised by qualitative alterations in reciprocal social interactions. Some recent studies show alterations in gaze patterns during social perception and rest-functional abnormalities in the ‘social brain network’. This study investigated: i) social perception gaze patterns in children with ASD and controls, ii) the relationship between autism clinical severity and social perception gaze patterns, iii) the relationship between resting cerebral blood flow (rCBF) and social perception gaze patterns. Methods: Nine children with ASD and 9 children with typical development were studied. Eye-tracking was used to detect gaze patterns during presentation of stimuli depicting social scenes. Autism clinical severity was established using the Autism Diagnostic Interview Revised (ADI-R). Arterial spin labelling MRI was used to quantify rCBF. Results: The ASD group looked less at social regions and more at non-social regions than controls. No significant correlation was found between ASD clinical severity and social perception gaze patterns. In the ASD group, gaze behaviour was related to rCBF in the temporal lobe regions at trend level. Positive correlations were found between temporal rCBF and gaze to the face region, while negative correlations were found between temporal rCBF and gaze to non-social regions. Conclusions: These preliminary results suggest that social perception gaze patterns are altered in children with ASD, and could be related to temporal rCBF.

Latency and slope values of evoked responses in children in brain stem audiometry

This paper is a review of a study to compare latencies of early evoked responses in young children with those of adults.

Effects of Delta9-tetrahydrocannabivarin on [35S]GTPgammaS binding in mouse brain cerebellum and piriform cortex membranes

BACKGROUND AND PURPOSE: We have recently shown that the phytocannabinoid Delta9-tetrahydrocannabivarin (Delta9-THCV) and the CB1 receptor antagonist AM251 increase inhibitory neurotransmission in mouse cerebellum and also exhibit anticonvulsant activity in a rat piriform cortical (PC) model of epilepsy. Possible mechanisms underlying cannabinoid actions in the CNS include CB1 receptor antagonism (by displacing endocannabinergic tone) or inverse agonism at constitutively active CB1 receptors. Here, we investigate the mode of cannabinoid action in [35S]GTPgammaS binding assays. EXPERIMENTAL APPROACH: Effects of Delta9-THCV and AM251 were tested either alone or against WIN55,212-2-induced increases in [35S]GTPgammaS binding in mouse cerebellar and PC membranes. Effects on non-CB receptor expressing CHO-D2 cell membranes were also investigated. KEY RESULTS :Delta9-THCV and AM251 both acted as potent antagonists of WIN55,212-2-induced increases in [35S]GTPgammaS binding in cerebellar and PC membranes (Delta9-THCV: pA2=7.62 and 7.44 respectively; AM251: pA2=9.93 and 9.88 respectively). At micromolar concentrations, Delta9-THCV or AM251 alone caused significant decreases in [35S]GTPgammaS binding; Delta9-THCV caused larger decreases than AM251. When applied alone in CHO-D2 membranes, Delta9-THCV and AM251 also caused concentration-related decreases in G protein activity. CONCLUSIONS AND IMPLICATIONS: Delta9-THCV and AM251 act as CB1 receptors antagonists in the cerebellum and PC, with AM251 being more potent than Delta9-THCV in both brain regions. Individually, Delta9-THCV or AM251 exhibited similar potency at CB1 receptors in the cerebellum and the PC. At micromolar concentrations, Delta9-THCV and AM251 caused a non-CB receptor-mediated depression of basal [35S]GTPgammaS binding.