922 resultados para Bogey, Joseph (1...-1877)


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Thesis (doctoral)--

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Vols. 1, 3, 1893; v. 2, 5, 1891; v. 4, "Revised edition," 1892; v. 6, "A new edition," 1889.

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Thesis (doctoral)--Georg-August-Universitat, Gottingen.

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Appendix to v. 4: Elephant pipes and inscribed tablets in the museum of the Academy of Natural Sciences, Davenport, Iowa. By Charles E. Putnam, p. [253]-347

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v. 1. Reizen in Midden-Sumatra, 1877-1879. 2 v.--v. 2. Aardri jkskundige beschri jving van Midden-Sumatra.--v. 3:1:1, Volksbeschrijving van Midden-Sumatra, door A. L. van Hasselt.--v. 3:1:2, Ethnographische atlas van Midden-Sumatra, met verklarenden tekst, door A. L. van Hasselt.--v. 3:2, De talen en letterkunde van Midden-Sumatra, door A. L. van Hasselt.--v. 4:1, Bi jdragen tot de kennis der fauna van Midden-Sumatra, door Joh. F. Snelleman. 2 v.--v. 4:2, Bijdragen tot de kennis der flora van Midden-Sumatra, door A. L. van Hasselt en Dr. J. G. Boerlage.

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November 15, 1899, "Extract from the "Official register of the United States" of July 1, 1899, corrected to November 15, 1899 as to the offices in Washington, D. C., and all residential appointments."

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"The Enterprise Databases Group has the responsibility for planning and development of the PUFs catalog under Betty Jackson, Director and Joseph F. Daniloski, Deputy Director. Karen Beebe ... made major contributions ... Key contributors to the content and format included Laquetta McNeal ... [et al.]."

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"Pieśni" (unacc. melodies) : v. 5-6.

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v.1. Macaulay, on the life and writings of Addison. [Prefaces, etc., to earlier editions] Translations. Poems on several occasions. The campaign. Miscellaneous poems. Dramas: Rosamund; The drummer; Cato. Poemata.--v.2. Dialogues on medals. Travels. Essay on Virgil's Georgics. Discourse on ancient and modern learning. Of the Christian religion. Letters. Political writings.--v.3. The Freeholder. Swift's notes on the Free-holder. The Plebian, by Sir Richard Steele, with The Old whig, by Mr. Addison. The Lover.--v.4. The Tatler. The Guardian.--v.5-6. The Spectator.

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Mode of access: Internet.

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The granulocyte colony-stimulating factor (G-CSF) and Fit-3 receptor agonist progenipoietin-1 (ProGP-1) has potent effects on dendritic cell (DC) expansion and may be an alternative to G-CSF for the mobilization of stem cells for allogeneic stem cell transplantation (SCT). We studied the ability of stem cell grafts mobilized with this agent to induce graft-versus-host disease (GVHD) to minor and major histocompatibility antigens in the well-described B6 --> B6D2F1 SCT model. ProGP-1, G-CSIF, or control diluent was administered to donor B6 mice. ProGP-1 expanded all cell lineages in the spleen, and unseparated splenocytes from these animals produced large amounts of interleukin 10 (IL-10) and transforming growth factor beta (TGFbeta) whereas the expression of T-cell adhesion molecules was diminished. Transplantation survival was 0%, 50%, and 90% in recipients of control-, G-CSF-, and ProGP-1-treated allogeneic donor splenocytes, respectively (P < .0001). Donor pretreatment with ProGP-1 allowed a 4-fold escalation in T-cell dose over that possible with G-CSF. Donor CD4 T cells from allogeneic SCT recipients of ProGP-1 splenocytes demonstrated an anergic response to host antigen, and cytokine production (interferon gamma [IFNγ], IL-4, and IL-10) was also reduced while CD8 T-cell cytotoxicity to host antigens remained intact. Neither CD11c(hi) DCs nor CD11c(dim)/B220(hi) DCs from ProGP-1-treated animals conferred protection from GVHD when added to control spleen. Conversely, when equal numbers of purified T cells from control-, G-CSF-, or ProGP-1-treated allogeneic donors were added to allogeneic T-cell-depleted control spleen, survival at day 60 was 0%, 15%, and 90%, respectively (P < .0001). The improved survival in recipients of ProGP-1 T cells was associated with reductions in systemic tumor necrosis factor alpha generation and GVHD of the gastrointestinal tract. We conclude that donor pretreatment with ProGP-1 is superior to G-CSIF for the prevention of GVHD after allogeneic SCT, primarily due to incremental affects on T-cell phenotype and function