¿Desde dónde intervenimos cuando intervenimos? El caso del "proyecto Tomate Platense"

Este trabajo se propone una reflexión crítica sobre una experiencia de intervención (El Proyecto Tomate Platense) en la que los autores de este artículo hemos participado desde su comienzo. Dicha reflexión implica poner en evidencia y en discusión, el qué y el cómo de la intervención para el desarrollo rural. Para esto, trabajamos los conceptos de: modelos de desarrollo e intervención, tecnología, hegemonía y agricultura familiar; y planteamos a su vez como estrategia metodológica, al estudio de caso. Posteriormente, realizamos una descripción del proceso de la intervención, identificando etapas y poniendo en relieve aspectos que consideramos significativos. En ese sentido visualizamos un proceso de legitimación de una propuesta alternativa, que implica en un sentido un quiebre del espiral tecnológico dominante presente en el cinturón hortícola de La Plata. La formulación inicial de la propuesta surge a partir de un diagnóstico de gabinete, realizado en el ámbito de la Facultad de Ciencias Agrarias y Forestales de la UNLP, que tiene un posterior devenir participativo que implica reformulaciones parciales y periódicas de la propuesta inicial

Newly discovered prokaryotic plant pathogens : a bibliography /

Includes index.

Bulletin /

Description based on: No. 10; title from cover.

Research bulletin /

Title from cover.

Mount telemark site analysis.

Cover title.

Bulletin.

Some nos. issued in revised editions.

Privately owned campgrounds in Wisconsin /

Cover title.

Annual report.

Title varies slightly.

Assessing consistency of fish survey data : uncertainties in the estimation of mackerel icefish (Champsocephalus gunnari) abundance at South Georgia

Acknowledgments The authors wish to thank the crews, fishermen and scientists who conducted the various surveys from which data were obtained, and Mark Belchier and Simeon Hill for their contributions. This work was supported by the Government of South Georgia and South Sandwich Islands. Additional logistical support provided by The South Atlantic Environmental Research Institute with thanks to Paul Brickle. Thanks to Stephen Smith of Fisheries and Oceans Canada (DFO) for help in constructing bootstrap confidence limits. Paul Fernandes receives funding from the MASTS pooling initiative (The Marine Alliance for Science and Technology for Scotland), and their support is gratefully acknowledged. MASTS is funded by the Scottish Funding Council (grant reference HR09011) and contributing institutions. We also wish to thank two anonymous referees for their helpful suggestions on earlier versions of this manuscript.

Electric shock causes physiological stress responses in shore crabs, consistent with prediction of pain

Animal pain is defined by a series of expectations or criteria, one of which is that there should be a physiological stress response associated with noxious stimuli. While crustacean stress responses have been demonstrated they are typically preceded by escape behaviour and thus the physiological change might be attributed to the behaviour rather than a pain experience. We found higher levels of stress as measured by lactate in shore crabs exposed to brief electric shock than non-shocked controls. However, shocked crabs showed more vigorous behaviour than controls. We then matched crabs with the same level of behaviour and still found that shocked crabs had stronger stress response compared with controls. The finding of the stress response, coupled with previous findings of long-Term motivational change and avoidance learning, fulfils the criteria expected of a pain experience.

Graduates of higher education in the food and agricultural sciences : an analysis of supply/demand relationships /

Issued July 1980.

Histological and morphometric lesions in the pre-clinical, developmental phase of insulin-induced laminitis in Standardbred horses

Lamellar pathology in experimentally-induced equine laminitis associated with euglycaemic hyperinsulinaemia is substantial by the acute, clinical phase (∼48 h post-induction). However, lamellar pathology of the developmental, pre-clinical phase requires evaluation. The aim of this study was to analyse lamellar lesions both qualitatively and quantitatively, 6, 12 and 24 h after the commencement of hyperinsulinaemia. Histological and histomorphometrical analyses of lamellar pathology at each time-point included assessment of lamellar length and width, epidermal cell proliferation and death, basement membrane (BM) pathology and leucocyte infiltration. Archived lamellar tissue from control horses and those with acute, insulin-induced laminitis (48 h) was also assessed for cellular proliferative activity by counting the number of cells showing positive nuclear immuno labelling for TPX2. Decreased secondary epidermal lamellar (SEL) width and increased histomorphological evidence of SEL epidermal basal (and supra-basal) cell death occurred early in disease progression (6 h). Increased cellular proliferation in SELs, infiltration of the dermis with small numbers of leucocytes and BM damage occurred later (24 and 48 h). Some lesions, such as narrowing of the SELs, were progressive over this time period (6–48 h). Cellular pathology preceded leucocyte infiltration and BM pathology, indicating that the latter changes may be secondary or downstream events in hyperinsulinaemic laminitis.