Workplace nonfatal injuries and illnesses, Illinois, 2001 /

"This project was supported by cooperative agreement number W9081170P from the U.S. Department of Labor, Bureau of Labor Statistics."

An inquiry into the process of nature in repairing injuries of the intestines: illustrating the treatment of penetrating wounds and strangulated hernia.

Mode of access: Internet.

Preventing injuries.

Mode of access: Internet.

Patterns of education and employment : rehabilitants from severe spinal cord injuries, fiscal year 1972-73 /

"June 1, 1974."

Postal service management of work-related injuries /

"September 28, 1989."

Female employees nonfatal days lost, injuries at coal and metal/nonmetal mining operations, 1996-2000 /

Shipping list no.: 2002-0451-M.

Workers' compensation and the distribution of occupational injuries /

"April 1991."

The law relating to compensation for injuries to workmen : being an exposition of the Workmen's Compensation Act, 1906, and of the case law relevant thereto ... with the whole of the regulations and forms made under the provisions of the act ... /

Includes index.

Railway injuries: with special reference to those of the back and nervous system, in their medico-legal and clinical aspects. ...

"Reprinted from Wood's medical and surgical monographs."

Louis Kuhne's Facial diagnosis ...

Later ed. has title: The science of facial expression.

How to save lives and reduce injuries : a citizen activist guide to effectively fight drunk driving /

Mode of access: Internet.

Physical treatment of injuries of the brain and allied nervous disorders.

Mode of access: Internet.

Cardiotrophin-like cytokine/cytokine-like factor 1 is an essential trophic factor for lumbar and facial motoneurons in vivo

The ciliary neurotrophic factor alpha-receptor(CNTFRalpha) is required for motoneuron survival during development, but the relevant ligand(s) has not been determined. One candidate is the heterodimer formed by cardiotrophin-like cytokine (CLC) and cytokine-like factor 1 (CLF). CLC/CLF binds to CNTFRalpha and enhances the survival of developing motoneurons in vitro; whether this novel trophic factor plays a role in neural development in vivo has not been tested. We examined motor and sensory neurons in embryonic chicks treated with CLC and in mice with a targeted deletion of the clf gene. Treatment with CLC increased the number of lumbar spinal cord motoneurons that survived the cell death period in chicks. However, this effect was regionally specific, because brachial and thoracic motoneurons were unaffected. Similarly, newborn clf -/- mice exhibited a significant reduction in lumbar motoneurons, with no change in the brachial or thoracic cord. Clf deletion also affected brainstem motor nuclei in a regionally specific manner; the number of motoneurons in the facial but not hypoglossal nucleus was significantly reduced. Sensory neurons of the dorsal root ganglia were not affected by either CLC treatment or clf gene deletion. Finally, mRNA for both clc and clf was found in skeletal muscle fibers of embryonic mice during the motoneuron cell death period. These findings support the view that CLC/CLF is a target-derived factor required for the survival of specific pools of motoneurons. The in vivo actions of CLC and CLF can account for many of the effects of CNTFRalpha on developing motoneurons.