A theory of asynchronous circuits I /

"This work has been supported in part by the Office of Naval Research under Contract NR 044 001"

A method for factoring the action of asynchronous circuits /

"Presented at the AIEE fall general meeting, Chicago, Illinois, October l1, 1960. This work was supported in part by the Office of Naval Research under contract Nonr-1834 (27)."

Design of totally self-checking asynchronous sequential machines.

Thesis (M.Sc.)--University of Illinois at Urbana-Champaign.

A theory of asynchronous circuits I /

"This work has been supported in part by the Office of Naval Research under Contract NR 044 001"

Theory of asynchronous circuits /

"December 6, 1955"

Stimulation /

Mode of access: Internet.

Electronic Combat Stimulation Branch : Atlantic Ranges and Facilities, Naval Air Warfare Center Aircraft Division, Patuxent River, Maryland.

Mode of access: Internet.

Cumulative auditory effects resulting from multiple exposure to intense acoustic stimulation.

Pt. 1. Deafening effects of noise on the cat -- Pt. 2. Histological effects of intense sound on the inner ear.

Reaction time to retinal stimulation, with special reference to the time lost in conduction through nerve centers,

Mode of access: Internet.

The effects of induced muscle tension and auditory stimulation on tachistoscopic perception.

Cover title.

United States Housing Act of 1936 : Hearings before the Committee on Education and Labor, United States Senate, Seventy-fourth Congress, second session, on S. 4424, a bill to provide financial assistance to the states and political subdivisions thereof for the elimination of unsafe and insanitary housing conditions, for the development of decent, safe, and sanitary dwellings for families of low income, and for the reduction of unemployment and the stimulation of business activity, to create a United States Housing Authority, and for other purposes. April 20, 21, 22, 23, 24, 25, and 29, 1936.

David I. Walsh, chairman.

Closed-Loop Deep Brain Stimulation: Bidirectional Neuroprosthetics for Tremor and BCI

Thesis (Ph.D.)--University of Washington, 2016-06

Effects of purinergic stimulation, CFTR and osmotic stress on amiloride-sensitive Na+ transport in epithelia and Xenopus oocytes

Both stimulation of purinergic receptors by ATP and activation of the cystic fibrosis transmembrane conductance regulator (CFTR) inhibit amiloride-sensitive Na+ transport and activate Cl-secretion. These changes in ion transport may well affect cell volume. We therefore examined whether cell shrinkage or cell swelling do affect amiloride-sensitive Na+ transport in epithelial tissues or Xenopus oocytes and whether osmotic stress interferes with regulation of Na+ transport by ATP or CFTR. Stimulation of purinergic receptors by ATP/UTP or activation of CFTR by IBMX and forskolin inhibited amiloride-sensitive transport in mouse trachea and colon, respectively, by a mechanism that was Cl- dependent. When exposed to a hypertonic but not hypotonic bath solution, amiloride-sensitive Na+ transport was inhibited in mouse trachea and colon, independent of the extracellular Cl- concentration. Both inhibition of Na+ transport by hypertonic bath solution and ATP were additive. When coexpressed in Xenopus oocytes, activation of CFTR by IBMX and forskolin inhibited the epithelial Na+ channel (ENaC) in a Cl(-)dependent fashion. However, both hypertonic and hypotonic bath solutions showed only minor effects on amiloride-sensitive conductance, independent of the bath Cl- concentration. Moreover, CFTR-induced inhibition of ENaC could be detected in chocytes even after exposure to hypertonic or bypotonic bath solutions. We conclude that amiloride-sensitive Na+ absorption in mouse airways and colon is inhibited by cell shrinkage by a mechanism that does not interfere with purinergic and CFTR-mediated inhibition of ENaC.

Does interhemispheric competition mediate motion-induced blindness? A transcranial magnetic stimulation study

Motion-induced blindness (MIB) is a phenomenon, perhaps related to perceptual rivalry, where stationary targets disappear and reappear in a cyclic mode when viewed against a background (mask) of coherent, apparent 3-D motion. Since MIB has recently been shown to share similar temporal properties with binocular rivalry, we probed the appearance-disappearance cycle of MIB using unilateral, single-pulse transcranial magnetic stimulation (TMS)-a manipulation that has previously been shown to influence binocular rivalry. Effects were seen for both hemispheres when the timing of TMS was determined prospectively on the basis of a given subject's appearance-disappearance cycle, so that it occurred on average around 300 ms before the time of perceptual switch. Magnetic stimulation of either hemisphere shortened the time to switch from appearance to disappearance and vice versa. However, TMS of left posterior parietal cortex more selectively shortened the disappearance time of the targets if delivered in phase with the disappearance cycle, but lengthened it if TMS was delivered in the appearance phase after the perceptual switch. Opposite effects were seen in the right hemisphere, although less marked than the left-hemisphere effects. As well as sharing temporal characteristics with binocular rivalry, MIB therefore seems to share a similar underlying mechanism of interhemispheric modulation. Interhemispheric switching may thus provide a common temporal framework for uniting the diverse, multilevel phenomena of perceptual rivalry.