Body, mind, and the sensory gateways.

Mode of access: Internet.

Access to multimedia technology by people with sensory disabilities /

"March 13, 1998."

Fused-quartz fibers : a survey of properties, applications, and production methods /

Bibliography: p. 18-26.

Vegetable fibers and their uses.

Includes bibliography.

High strength, high modulus fibers by pyrolysis of polymeric fibers

Mode of access: Internet.

Wool statistics, including mohair and other animal fibers.

Cover-title.

A possible application of organic fibers in high temperature environment /

This report was prepared by Fabric Research Laboratories, Inc. under USAF Contract No. AF33 (616)--6234 under Project No. 7320, Task No. 73201.

The coloring of post-war synthetic fibers /

Mode of access: Internet.

Identification of fibers in textile materials.

Mode of access: Internet.

The dyeing of cotton and other vegetable fibers with dyestuffs of Leopold Cassella & Co.

Supplements. Nos. 4-14, 16-19.

The demand for textile fibers in the United States /

Includes bibliographical references (p. 101-105).

The changing uses of textile fibers in clothing and household articles /

Caption title.

Principles of high-polymer theory and practice; fibers, plastics, rubbers, coatings, adhesives,

Includes bibliographies.

An ancient neurotrophin code in zebrafish; the Runx/Cbfβ complex specifies the fate switch from TrkB+ to TrkC+ nociceptors and sensory receptor expression

Thesis (Ph.D.)--University of Washington, 2016-06

Cardiotrophin-like cytokine/cytokine-like factor 1 is an essential trophic factor for lumbar and facial motoneurons in vivo

The ciliary neurotrophic factor alpha-receptor(CNTFRalpha) is required for motoneuron survival during development, but the relevant ligand(s) has not been determined. One candidate is the heterodimer formed by cardiotrophin-like cytokine (CLC) and cytokine-like factor 1 (CLF). CLC/CLF binds to CNTFRalpha and enhances the survival of developing motoneurons in vitro; whether this novel trophic factor plays a role in neural development in vivo has not been tested. We examined motor and sensory neurons in embryonic chicks treated with CLC and in mice with a targeted deletion of the clf gene. Treatment with CLC increased the number of lumbar spinal cord motoneurons that survived the cell death period in chicks. However, this effect was regionally specific, because brachial and thoracic motoneurons were unaffected. Similarly, newborn clf -/- mice exhibited a significant reduction in lumbar motoneurons, with no change in the brachial or thoracic cord. Clf deletion also affected brainstem motor nuclei in a regionally specific manner; the number of motoneurons in the facial but not hypoglossal nucleus was significantly reduced. Sensory neurons of the dorsal root ganglia were not affected by either CLC treatment or clf gene deletion. Finally, mRNA for both clc and clf was found in skeletal muscle fibers of embryonic mice during the motoneuron cell death period. These findings support the view that CLC/CLF is a target-derived factor required for the survival of specific pools of motoneurons. The in vivo actions of CLC and CLF can account for many of the effects of CNTFRalpha on developing motoneurons.