940 resultados para 3-D objects
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First lub. in 1841 under title: Histoire de la littérature française au moyen âge comparée aux littératures étrangères ... Introduction: histoire de la formation de la langue française.
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Vol. <20-> published: Stockholm : Hörberg.
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Popularly ascribed at the time of its appearance to a certain Benard, presumed by P. Lacroix to be J.F. bernard.- cf.Barbier, Dict. des ouvr. anon. 3.éd., 1872-79.
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Texto en frances y provenzal.
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Includes index.
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Tom. II is "3. éd."
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Publisher of v. 5: M. Ducloux.
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Vol. 2 in portfolio.
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"Cette nouvelle édition des Oeuvres de Laplace ... réunit, pour la première fois, aux divers ouvrages compris dans les éditions précédentes, la collection des Mémoires de l'illustre astronome, rangée par ordre chronologique."--Advertissement.
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Cover title.
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Rando brillant, op. 70, D.895-- Sonata, D major, op. 137, No. 1, D.384-- Sonata, A minor, op. 137, No. 2, D.385-- Sonata, G minor, op. 137, No. 3, D.408.
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LC copy of v. 3 has Bulletin de la librarie Plon, no. 11, Feb. 1905, and 2 other publisher's advertisements, laid in; v. 4 has 2 publisher's advertisements and 1 rail folder, laid in; v. 5 has Bulletin de la librarie Plon, no. 11, Feb. 1905 and a [2] p. publisher's advertisement, laid in; v. 6 has a [4] p. publisher's advertisement, laid in.
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Vol. 9 had title : Lucii Ferraris Promptae bibliothecae supplememtum continens recentiora acta S. Sedis et recentiores resolutiones ac decreta ss. romanarum congrationum edidit Ianarius Bucceroni.
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t.1, 2, 5 éd., 1911 t.3, 3 éd., 1910.
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Pyrithiamine-induced thiamine deficiency (TD) is a well-established model of Wernicke's encephalopathy in which a glutamate-mediated excitotoxic mechanism may play an important role in determining selective vulnerability. In order to examine this possibility, cultured astrocytes were exposed to TD and effects on glutamate transport and metabolic function were studied. TD led to decreases in cellular levels of thiamine and thiamine diphosphate (TDP) after 24 h of treatment and decreased activities of the TDP-dependent enzymes alpha-ketoglutarate dehydrogenase and transketolase after 4 and 7 days, respectively. TD treatment for 10 days led to a reversible decrease in the uptake of [H-3]-D-aspartate, a nonmetabolizable analogue of glutamate. Kinetic analysis revealed that the uptake inhibition was caused by a 47% decrease in the V-max for uptake of [H-3]-D-aspartate, with no change in the K-m value. Immunoblotting showed that this decrease in uptake was due to an 81% downregulation of the astrocyte-specific GLAST glutamate transporter. Loss of uptake activity and GLAST protein were blocked by treatment with the protein kinase C inhibitor H7, while exposure to DCG IV, a group II metabotropic glutamate receptor (mGluR) agonist, resulted in improvement of [H-3]-D-aspartate uptake and a partial reversal of transporter downregulation. These results are consistent with our recent in vivo findings of a loss of astrocytic glutamate transporters in TD and provide evidence that TD conditions may increase phosphorylation. of GLAST, contributing to its downregulation. In addition, manipulation of group II mGluR activity may provide an important strategy in the treatment of this disorder. (C) 2003 Wiley-Liss, Inc.