924 resultados para Copper mines and mining
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1869-70 has half-title: Third biennial report of the state mineralogist.
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At head of title: 89th Congress, 1st session, Committee print.
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Includes bibliography.
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At head of title: Canada, Department of mines, Geological survey branch ...
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At head of title: 88th Cong., 2d sess. Committee print.
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-1898 include also the reports of the State Inspectors of Mines; 1899-1907, Report of the Illinois Free Employment Offices; 1917- , reports of the Miners' Examining Board and the Mine Rescue and First Aid Division (formerly Mine Rescue Station Commission).
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Mode of access: Internet.
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Mode of access: Internet.
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Title varies slightly
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Mode of access: Internet.
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Mode of access: Internet.
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"July 12, 1988" -- pt.2.
USGS geochemical studies outline mineral potential and environmental hazards in southeastern Oregon.
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Mode of access: Internet.
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Experimental laboratory methods have been developed that enable phase-equilibria studies to be carried out on slags in the system Ca-Cu-Fe-O in equilibrium with metallic copper. These techniques involve equilibration at temperature, rapid quenching, and chemical analysis of the phases using electron-probe X-ray microanalysis (EPNIA). Equilibration experiments have been carried out in the temperature range of 1150 degreesC to 1250 degreesC (1423 to 1523 K) and in the composition range of 4 to 80 wt pct "Cu2O," 0 to 25 wt pct CaO, and 20 to 75 wt pct "Fe2O3" in equilibrium with metallic copper. Liquidus and solidus data are reported for the primary-phase fields of spinel (magnetite) and dicalcium ferrite. The resulting data have been used to construct liquidus isotherms of the CaO-"Cu2O"-"Fe2O3" system at metallic copper saturation.
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Canine copper toxicosis is an important inherited disease in Bedlington terriers, because of its high prevalence rate and similarity to human copper storage disease. It can lead to chronic liver disease and occasional haemolytic anaemia due to impaired copper excretion. The responsible gene for copper toxicosis in Bedlington terriers has been recently identified and was found not to be related to human Wilson's disease gene ATP7B. Although our understanding of copper metabolism in mammals has improved through genetic molecular technology, the diversity of gene mutation related to copper metabolism in animals will help identify the responsible genes for non-Wilsonian copper toxicoses in human. This review paper discusses our knowledge of normal copper metabolism and the pathogenesis, molecular genetics and current research into copper toxicosis in Bedlington terriers, other animals and humans. (C) 2004 Elsevier GmbH. All rights reserved.