Les mémoires de messire Jean, seigneur de Haynin et de Louvegnies... 1465-1477.... Tome 1 / [Édité par R. Chalon]

Collection : Publications de la Société des bibliophiles belges séant à Mons ; 11

Les mémoires de messire Jean, seigneur de Haynin et de Louvegnies... 1465-1477.... Tome 2 / [Édité par R. Chalon]

Collection : Publications de la Société des bibliophiles belges séant à Mons ; 11

Le siège de Paris par les Normands, en 885 et 886 / poème d'Abbon ; avec la trad. en regard, accompagné de notes explicatives et historiques, par N. R. Taranne,...

[Bella Parisiacae urbis (français-latin). 1834]

Attentional networks efficiency in preterm children.

Recent studies have reported specific executive and attentional deficits in preterm children. However, the majority of this research has used multidetermined tasks to assess these abilities, and the interpretation of the results lacks an explicit theoretical backdrop to better understand the origin of the difficulties observed. In the present study, we used the Child Attention Network Task (Child ANT; Rueda et al. 2004) to assess the efficiency of the alerting, orienting and executive control networks. We compared the performance of 25 preterm children (gestational age < or = 32 weeks) to 25 full-term children, all between 5(1/2) and 6(1/2) years of age. Results showed that, as compared to full-term children, preterm children were slower on all conditions of the Child ANT and had a specific deficit in executive control abilities. We also observed a significantly higher correlation between the orienting and executive control networks in the preterm group, suggesting less differentiation of these two networks in this population.

BAFF-R, a newly identified TNF receptor that specifically interacts with BAFF.

B cell homeostasis has been shown to critically depend on BAFF, the B cell activation factor from the tumor necrosis factor (TNF) family. Although BAFF is already known to bind two receptors, BCMA and TACI, we have identified a third receptor for BAFF that we have termed BAFF-R. BAFF-R binding appears to be highly specific for BAFF, suggesting a unique role for this ligand-receptor interaction. Consistent with this, the BAFF-R locus is disrupted in A/WySnJ mice, which display a B cell phenotype qualitatively similar to that of the BAFF-deficient mice. Thus, BAFF-R appears to be the principal receptor for BAFF-mediated mature B cell survival.