991 resultados para K CHANNELS


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This paper presents our investigation on iterativedecoding performances of some sparse-graph codes on block-fading Rayleigh channels. The considered code ensembles are standard LDPC codes and Root-LDPC codes, first proposed in and shown to be able to attain the full transmission diversity. We study the iterative threshold performance of those codes as a function of fading gains of the transmission channel and propose a numerical approximation of the iterative threshold versus fading gains, both both LDPC and Root-LDPC codes.Also, we show analytically that, in the case of 2 fading blocks,the iterative threshold root of Root-LDPC codes is proportional to (1 2)1, where 1 and 2 are corresponding fading gains.From this result, the full diversity property of Root-LDPC codes immediately follows.

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We examine a multiple-access communication system in which multiuser detection is performed without knowledge of the number of active interferers. Using a statistical-physics approach, we compute the single-user channel capacity and spectral efficiency in the large-system limit.

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Cont: Irn, potencia nuclear?; El reto nuclear de Irn; Dos respuestas al reto nuclear; Con la vista puesta en Irn; La provocacin nuclear norcoreana; Irn y la oferta de Obama

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Changes in expression and function of voltage-gated sodium channels (VGSC) in dorsal root ganglion (DRG) neurons may play a major role in the genesis of peripheral hyperexcitability that occurs in neuropathic pain. We present here the first description of changes induced by spared nerve injury (SNI) to Na(v)1 mRNA levels and tetrodotoxin-sensitive and -resistant (TTX-S/TTX-R) Na(+) currents in injured and adjacent non-injured small DRG neurons. VGSC transcripts were down-regulated in injured neurons except for Na(v)1.3, which increased, while they were either unchanged or increased in non-injured neurons. TTX-R current densities were reduced in injured neurons and the voltage dependence of steady-state inactivation for TTX-R was positively shifted in injured and non-injured neurons. TTX-S current densities were not affected by SNI, while the rate of recovery from inactivation was accelerated in injured neurons. Our results describe altered neuronal electrogenesis following SNI that is likely induced by a complex regulation of VGSCs.

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Titre uniforme : [Fantaisies. Piano. KV 475. Do mineur]

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[Traditions. Asie. Inde. Prsidence de Madras [i.e. Chennai]. Etat du Kerala. Rgion du Malabar]

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[Traditions. Asie. Inde. Province de Madras [i.e. Chennai]. tat de Karnataka. Rgion du Kanara]