Operator, organizational, direct support, and general support maintenance manual for air conditioner, vertical compact : type I, vertical, size C, 18,000 BTU/hr, class I, 208 volt, 3 phase, 50/60 hertz, Keco model F18T-2 : NSN 4120-00-168-1781.

"21 December 1979."

Organizational and direct support maintenance repair parts and special tools list (including depot maintenance repair parts and special tools) for viewers, driver's night vision, AN/VVS-2(V)1 (NSN 5855-00-629-5278), AN/VVS-2(V)2 (NSN 5855-01-057-1880), AN/VVS-2(V)3 (NSN 5855-01-105-7793), AN/VVS-2(V)1A (NSN 5855-01-096-0871), and AN/VVS-2(V)2A (NSN 5855-01-096-0872).

Includes index.

Organizational, direct support, and general support maintenance repair parts and special tools lists (including depot maintenance repair parts and special tools) for radio terminal sets AN/TRC-145(V)1, AN/TRC-145(V)2, AN/TRC-145(V)3, AN/TRC-145A(V)1, AN/TRC-145A(V)2, and AN/TRC-145A(V)3, (NSN 5820-00-791-3365).

Includes index.

The thermodynamic properties of helium from 3 to 300⁰ K between 0.5 and 100 atmospheres

Bibliography: p. 13-14.

Dushan lei gao : 6 juan, you 3 juan, shi gao 4 juan /

Double leaves, oriental style, in case.

Uber Salzbildung, Umlagerung und Aufspaltung des 1-Phenyl-4-oximido-5-triazolons /

Thesis (doctoral)--K. Bayer. Ludwig-Maximilians- Universitat zu Munchen, 1908.

America annuals of education. v. 1-4, Jan. 18260-Dec. 1829; new ser., v. 1, no. 1-5, Jan.-July 1830; 3d ser., v.1-9, Aug. 1830-Dec. 1839.

From Jan. 1826-July 1830 title reads: American journal of education. In August 1830 the title was changed to American Annuals of Education and instruction. The numbers for August-Dec. 1830, however, were issued both under this later title (as 3d ser., b.1, no 1-4) and under the title American journal and annals of education and instruction, new ser., v.1 of the 3d ser. They are preceded by 24 pages ("Editor's address" and "Sketches of Hofwyl") reprinted from the numbers for Aug.-Dec. 1830) In 1838 the title was shortened to American annuals of education. Editors: 1826-29, Williams Russell.--1830-37, W.C. Woodbridge.--1837--38 W.A. Alcott.

Fragm. lat. II 34 - Biblia glossata (Dt 4,43 - 5,9; 12,15 - 13,16)

Trägerband: 'Spirae 3a Julii 1655'; Vorbesitzer: Stadtarchiv Frankfurt am Main

Fragm. lat. I 62 - Bibelkommentar (Apc 5,13; 6,2; 12,1 - 4)

Trägerband: Ms. lat. oct. 26; Ms. lat. oct. 30; Vorbesitzer: Johann Hartmann Beyer

Fragm. lat. I 69 - Psalterium (Ps 22,4 - 6; 23,5 - 9; 29,13 - 30,4; 30,10 - 12; 31,5 - 8; 32,1 - 7; 37,5 - 11; 37,13 - 18)

Vorbesitzer: Gustav Freytag

ROR alpha regulates the expression of genes involved in lipid homeostasis in skeletal muscle cells - Caveolin-3 and CPT-1 are direct targets of ROR

The staggerer mice carry a deletion in the RORalpha gene and have a prolonged humoral response, overproduce inflammatory cytokines, and are immunodeficient. Furthermore, the staggerer mice display lowered plasma apoA-I/-II, decreased plasma high density lipoprotein cholesterol and triglycerides, and develop hypo-alpha-lipoproteinemia and atherosclerosis. However, relatively little is known about RORalpha in the context of target tissues, target genes, and lipid homeostasis. For example, RORalpha is abundantly expressed in skeletal muscle, a major mass peripheral tissue that accounts for similar to40% of total body weight and 50% of energy expenditure. This lean tissue is a primary site of glucose disposal and fatty acid oxidation. Consequently, muscle has a significant role in insulin sensitivity, obesity, and the blood-lipid profile. In particular, the role of RORalpha in skeletal muscle metabolism has not been investigated, and the contribution of skeletal muscle to the ROR-/- phenotype has not been resolved. We utilize ectopic dominant negative RORalpha expression in skeletal muscle cells to understand the regulatory role of RORs in this major mass peripheral tissue. Exogenous dominant negative RORalpha expression in skeletal muscle cells represses the endogenous levels of RORalpha and -gamma mRNAs and ROR-dependent gene expression. Moreover, we observed attenuated expression of many genes involved in lipid homeostasis. Furthermore, we show that the muscle carnitine palmitoyltransferase-1 and caveolin-3 promoters are directly regulated by ROR and coactivated by p300 and PGC-1. This study implicates RORs in the control of lipid homeostasis in skeletal muscle. In conclusion, we speculate that ROR agonists would increase fatty acid catabolism in muscle and suggest selective activators of ROR may have therapeutic utility in the treatment of obesity and atherosclerosis.

N-(3,5-dichloro-2-pyridyl) formamide

The molecule of the title compound, C6H4Cl2N2O, is essentially planar.

Contribution of the collagen I alpha 1 and vitamin D receptor genes to the risk of hip fracture in elderly women

Context and Objective: Hip fracture is partially genetically determined. The present study was designed to examine the contributions of vitamin D receptor (VDR) and collagen I alpha 1 (COLIA1) genotypes to the liability to hip fracture in postmenopausal women. Design: The study was designed as a prospective population-based cohort investigation. Subjects: Six hundred seventy-seven postmenopausal women of Caucasian background, aged 70 +/- 7 yr (mean +/- SD), have been followed for up to 14 yr. Sixty-nine women had sustained a hip fracture during the period. Main Outcome: Atraumatic hip fractures were prospectively identified through radiologists' reports. Bone mineral density (BMD) at the hip and lumbar spine was measured by dual-energy x-ray absorptiometry. Genotypes: The TaqI and SpI COLIA1 polymorphisms of the VDR and COLIA1 genes were determined. Using the Single Nucleotide Polymorphism database, VDR TT, Tt, and tt genotypes were coded as TT, TC, and CC, whereas COLIA1 SS, Ss, and ss were coded as GG, GT, and TT. Results: Women with VDR CC genotype (16% prevalence) and COLIA1 TT genotype (5% prevalence) had an increased risk of hip fracture [odds ratio (OR) associated with CC, 2.6; 95% confidence interval (CI), 1.2-5.3; OR associated with TT, 3.8; 95% CI, 1.3-10.8] after adjustment for femoral neck BMD (OR, 3.4 per SD; 95% CI, 2.3-5.0) and age (OR, 1.4 per 5 yr; 95% CI, 1.1-1.7). Approximately 20 and 12% of the liability to hip fracture was attributable to the presence of the CC genotype and TT genotype, respectively. Conclusion: The VDR CC genotype and COLIA1 TT genotype were associated with increased hip fracture risk in Caucasian women, and this association was independent of BMD and age.