School of Nursing Class of 2010

Top Row: Jocelyn Aden, Rachel Ades, Katrina Allen, Kayla Ashcraft, Kristie Baker, Amy Beaudoin, Heidi Beck, Beth Bentrum, Amber Blake, Lee Anna Braden, Dan Burd, Meaghan Burke, Mallory Calus, Irene Casillas, Veronica Cherney, Samantha Cholewa, Molly Conlen

Row 2: Wendy Corriveau, Meaghan Cotter, Kara DeGlopper, Colleen DeVoe, Hadley Dobbs, Kimberly Drury-Wallace, Hyesun Eitel, Sarah Elner, Douglas E. Elsey, Alyssa Fallot, Folake Famoye, Kristen Farr, Christine Fleck, Jennifer Fleming, Soncerae Gardner, Sarah Gilley, Joelle Gilmet

Row 3: Sara Goss, Amy Guffey, Taylor Griglak, Bridget Belvitch, Jaclyn Janks, Andrea Engles, Cassandra Smith, Lyndsy Brenner, Mallorie Patterson, Kristen Oltersdorf, Laura Kokx, Ross Zoet, Mary Osbach, Courtney Norman, Monica Habeck, Erica Hadley

Row 4: Amanda Hanert, Dayna Hasty, Nicole Heller, Ashley Howard, Robert Humburg, Andrew Humes, Grace Hwang, Amira Jackson, Kathryn Jipping, Shelly Johnson

Row 5: Lindsey Kappler, Jacqueline Klaiman, Sarah Knoedler, Jessica Kopicki, Kathryn Kovanda, Sarah Kovats, Emily Krogel, Kellie Kunkel, Kristin Lakatos, Chelsea Lazaroff, Bo Hwa Lee, Kelly Leja

Row 6: Kelli Littlejohn, Emilee Losey, Patricia Luna, Wilma MacKenzie, Matt Malkowski, Rachel Mallas, Emily McCallister, Diane McDonald, Dorian Michelson, Mary Miller, Nicole Miller, Kristen Muehlhauser

Row 7: Renee Muller, Katherine Mulvaney, Eugene Ngala, Christine Novotny, Colleen O'Connor, Cassey Parrish, Kimberly Peters, Kathleen Potempa, Bonnie Hagerty, Heather Poucher, Charles Reisdorf, Eric Retzbach, Sarah Rhem, Shannon Rice, Amy Roberts, Christie Schonsheck

Row 8: Franciska Schuett, Rhonda Schultz, Kristina Seidl, Teresa Semaan, Shelley Sibbold, Stacy Slater, Mary Snell, Mallory Stanton, Dennis Stevens, Miranda Stoddard, Tatiana Tafla, Priscilla Tang, Bethany Thelen, Jessica Thibert, Rebecca Thurk, Lauren Tormoehlen, Chinasa Uwandu

Row 9: Margaret van Buitenen, Stacey Victor, Jennifer Waag, Kirstyn Wade, Ariel Warren, Elizabeth White, Natalie Wierenga, Jessica Wihowski, Wendy Witkowski, Aliza Wolfe, DaShaunn Woolard, Ting Wan Yip, Alexander Young, Kellie Zenz, Kristen Ziulkowski, Jessica Zmierski

An outreach demonstration : a component of a manpower program in Houston, Texas, 1967 /

Originally published: Houston : Neighborhood Centers Assoc., 1967.

Cyclopædia of the practice of medicine.

Each volume contains "Biographical sketches of the authors."

The bookman.

Title from caption.

Beitrage zur Theorie des Eisenhochofenprozesses und Untersuchungen uber die gegenseitigen Beziehungen der verschiedenen Kohlenstoffmodifikationen.

Thesis (doctoral)--Universitat Marburg.

Ueber die einwirkung von amidoacetal auf anisaldehyd, salicylal...

Thesis (doctoral)--

Ueber die Einwirkung von Kohlenoxysulfid, Phosgen und Chlorkohlensaureester auf Phenylhydrazin /

Thesis (doctoral)--K. Bayer. Julius-Maximilians- Universitat Wurzburg, 1890.

Creep of engineering materials

Includes bibliography.

Didaskalia : Blätter für Geist, Gemüth und Publicität.

Editors: J.L. Heller, later, J.A. Hammerau and others.

Botanical explorations in southern Texas, during the season of 1894.

Photocopy.

Geology and landslide geomorphology of the Burpee Hills, Skagit County, Washington, USA

Landforms within the Skagit Valley record a complex history of land evolution from Late Pleistocene to the present. Late Pleistocene glacial deposits and subsequent incision by the Skagit River formed the Burpee Hills terrace. The Burpee Hills comprises an approximately 205-m-thick sequence of sediments, including glacio-lacustrine silts and clays, overlain by sandy advance outwash and capped by coarse till, creating a sediment-mantled landscape where mass wasting occurs in the form of debris flows and deep-seated landslides (Heller, 1980; Skagit County, 2014). Landslide probability and location are necessary metrics for informing citizens and policy makers of the frequency of natural hazards. Remote geomorphometric analysis of the site area using airborne LiDAR combined with field investigation provide the information to determine relative ages of landslide deposits, to classify geologic units involved, and to interpret the recent hillslope evolution. Thirty-two percent of the 28-km2 Burpee Hills landform has been mapped as landslide deposits. Eighty-five percent of the south-facing slope is mapped as landslide deposits. The mapped landslides occur predominantly within the advance outwash deposits (Qgav), this glacial unit has a slope angle ranging from 27 to 36 degrees. Quantifying surface roughness as a function of standard deviation of slope provides a relative age of landslide deposits, laying the groundwork for frequency analysis of landslides on the slopes of the Burpee Hills. The south-facing slopes are predominately affected by deep-seated landslides as a result of Skagit River erosion patterns within the floodplain. The slopes eroded at the toe by the Skagit River have the highest roughness coefficients, suggesting that areas with more frequent disturbance at the toe are more prone to sliding or remobilization. Future work including radiocarbon dating and hydrologic-cycle investigations will provide a more accurate timeline of the Burpee Hills hillslope evolution, and better information for emergency management and planners in the future.

Randomised controlled trial of health assessments for older Australian veterans and war widows

Objective: To assess the effect of home-based health assessments for older Australians on health-related quality of life, hospital and nursing home admissions, and death. Design: Randomised controlled trial of the effect of health assessments over 3 years. Participants and setting: 1569 community-living veterans and war widows receiving full benefits from the Department of Veterans' Affairs and aged 70 years or over were randomly selected in 1997 from 10 regions of New South Wales and Queensland and randomly allocated to receive either usual care (n = 627) or health assessments (n = 942). Intervention: Annual or 6-monthly home-based health assessments by health professionals, with telephone follow-up, and written report to a nominated general practitioner. Main outcome measures: Differences in health-related quality of life, admission to hospital and nursing home, and death over 3 years of follow-up. Results: 3-year follow-up interviews were conducted for 1031 participants. Intervention-group participants who remained in the study reported higher quality of life than control-group participants (difference in Physical Component Summary score, 0.90; 95% CI, 0.05-1.76; difference in Mental Component Summary score, 1.36; 95% CI, 0.40-2.32). There was no significant difference in the probability of hospital admission or death between intervention and control groups over the study period. Significantly more participants in the intervention group were admitted to nursing homes compared with the control group (30 v 7; P < 0.01). Conclusions: Health assessments for older people may have small positive effects on quality of life for those who remain resident in the community, but do not prevent deaths. Assessments may increase the probability of nursing-home placement.

The effects of high glucose and atorvastatin on endothelial cell matrix production

Background Statins are known to enhance atherosclerotic plaque stability through influences on extracellular matrix homeostasis. Net matrix production reflects the relative balance of matrix production and degradation through enzymes such as matrix metalloproteinases (MMPs) and their inhibitors, tissue inhibitor of MMP (TIMPs). The effects of statins on endothelial cell production of these parameters following co-exposure with a proatherogenic stimulus such as high glucose are not known. Methods Human endothelial cells were exposed for 72 h to 5 mM> (control) or 25 mM (high) glucose +/- atorvastatin (1 mumol/l). Extracellular matrix homeostasis was assessed by measuring matrix metalloproteinase (MMP)-2 secretion, tissue inhibitor of MMP (TIMP)-1 and -2 secretion and net collagen IV production. Results were expressed as percentage +/- SEM of control values. Results Exposure to high glucose increased cellular collagen IV expression to 190.1 +/- 11.7% (P < 0.0001) of control levels. No change in MMP-2 secretion (111.6 +/- 5.2%; P > 0.05) was observed but both TIMP-1 and TIMP-2 expression were increased to 136.3 +/- 6.4% and 144.0 +/- 27.5%, respectively (both P < 0.05). The presence of atorvastatin in high glucose conditions reduced collagen IV expression to 136.1 +/- 20.6%. This was paralleled by increased secretion of MMP-2 to 145.8 +/- 7.8% (P < 0.01), increased TIMP-2 expression to 208.0 +/- 21.3% (P < 0.005 compared with high glucose) but no change in TIMP-1 expression (155.1 +/- 14.6%) compared with high glucose alone. The presence of atorvastatin in control conditions did not affect levels of collagen IV expression (114.5 +/- 13.2%). Conclusions Endothelial cell exposure to high glucose was associated with a MMP/TIMP profile that increased extracellular matrix production which was attenuated by concurrent exposure to atorvastatin. Consequently, a mechanism by which the atherosclerotic plaque regression that is observed in patients taking these drugs has been demonstrated.

Australian and New Zealand practice in intensive care (ANZPIC II): Predictors of physician confidence to diagnose pneumonia and determine illness severity in ventilated patients

The manner in which elements of clinical history, physical examination and investigations influence subjectively assessed illness severity and outcome prediction is poorly understood. This study investigates the relationship between clinician and objectively assessed illness severity and the factors influencing clinician's diagnostic confidence and illness severity rating for ventilated patients with suspected pneumonia in the intensive care unit (ICU). A prospective study of fourteen ICUs included all ventilated admissions with a clinical diagnosis of pneumonia. Data collection included pneumonia type - community-acquired (CAP), hospital-acquired (HAP) and ventilator-associated (VAP), clinician determined illness severity (CDIS), diagnostic methods, clinical diagnostic confidence (CDC), microbiological isolates and antibiotic use. For 476 episodes of pneumonia (48% CAP, 24% HAP, 28% VAP), CDC was greatest for CAP (64% CAP, 50% HAP and 49% VAP, P < 0.01) or when pneumonia was considered life-threatening (84% high CDC, 13% medium CDC and 3% low CDC, P < 0.001). Life-threatening pneumonia was predicted by worsening gas exchange (OR 4.8, CI 95% 2.3-10.2, P < 0.001), clinical signs of consolidation (OR 2.0, CI 95% 1.2-3.2, P < 0.01) and the Sepsis-Related Organ Failure Assessment (SOFA) Score (OR 1.1, CI 95% 1.1-1.2, P < 0.001). Diagnostic confidence increased with CDIS (OR 163, CI 95% 8.4-31.4, P < 0.001), definite pathogen isolation (OR 3.3, CI 95% 2.0-5.6) and clinical signs of consolidation (OR 2.1, CI 95% 1.3-3.3, P = 0.001). Although the CDIS, SOFA Score and the Simplified Acute Physiologic Score (SAPS II) were all associated with mortality, the SAPS II Score was the best predictor of mortality (P = 0.02). Diagnostic confidence for pneumonia is moderate but increases with more classical presentations. A small set of clinical parameters influence subjective assessment. Objective assessment using SAPS II Scoring is a better predictor of mortality.

Disease risk and mortality prediction in intensive care patients with pneumonia. Australian and New Zealand practice in intensive care (ANZPIC II)

This study of ventilated patients investigated pneumonia risk factors and outcome predictors in 476 episodes of pneumonia (48% community-acquired pneumonia, 24% hospital-acquired pneumonia, 28% ventilator-associated pneumonia) using a prospective survey in 14 intensive care units within Australia and New Zealand. For community acquired pneumonia, mortality increased with immunosuppression (OR 5.32, CI 95% 1.58-17.99, P < 0. 01), clinical signs of consolidation (OR 2.43, CI 95% 1.09-5.44, P = 0. 03) and Sepsis-Related Organ Failure Assessment (SOFA) scores (OR 1.19, CI 95% 1.08-1.30, P < 0. 001) but improved if appropriate antibiotic changes were made within three days of intensive care unit admission (OR 0.42, CI 95% 0.20-0.86, P = 0.02). For hospital-acquired pneumonia, immunosuppression (OR 6.98, CI 95% 1.16-42.2, P = 0.03) and non-metastatic cancer (OR 3.78, CI 95% 1.20-11.93, P = 0.02) were the principal mortality predictors. Alcoholism (OR 7.80, CI 95% 1.20-1750, P < 0.001), high SOFA scores (OR 1.44, CI 95% 1.20-1.75, P = 0.001) and the isolation of high risk organisms including Pseudomonas aeruginosa, Acinetobacter spp, Stenotrophomonas spp and methicillin resistant Staphylococcus aureus (OR 4.79, CI 95% 1.43-16.03, P = 0.01), were associated with increased mortality in ventilator-associated pneumonia. The use of non-invasive ventilation was independently protective against mortality for patients with community-acquired and hospital-acquired pneumonia (OR 0.35, CI 95% 0.18-0.68, P = 0.002). Mortality was similar for patients requiting both invasive and non-invasive ventilation and non-invasive ventilation alone (21% compared with 20% respectively, P = 0.56). Pneumonia risks and mortality predictors in Australian and New Zealand ICUs vary with pneumonia type. A history of alcoholism is a major risk factor for mortality in ventilator-associated pneumonia, greater in magnitude than the mortality effect of immunosuppression in hospital-acquired pneumonia or community-acquired pneumonia. Non-invasive ventilation is associated with reduced ICU mortality. Clinical signs of consolidation worsen, while rationalising antibiotic therapy within three days of ICU admission improves mortality for community-acquired pneumonia patients.