Proceedings of the Linnean Society of New South Wales.

v.2=[no.5-8] (1877-1878)

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.3

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.7

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.1

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.4

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.2

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.5

Les champignons (fungi, hyménomycètes) qui croissent en France. Description et iconographie propriétés utiles ou vénéneuses.

Pls. v.6

Histoire des drogues d'origine végétale /

v. 1

Histoire des drogues d'origine végétale /

v. 2

Historia general y natural de las Indias, islas y tierra-firme del mar oceano,

3

Historia general y natural de las Indias, islas y tierra-firme del mar oceano,

1

Bribe-proof rules in the division problem

The division problem consists of allocating an amount of a perfectly divisible good among a group of n agents with single-peaked preferences. A rule maps preference profiles into n shares of the amount to be allocated. A rule is bribe-proof if no group of agents can compensate another agent to misrepresent his preference and, after an appropriate redistribution of their shares, each obtain a strictly preferred share. We characterize all bribe-proof rules as the class of efficient, strategy-proof, and weak replacement monotonic rules. In addition, we identify the functional form of all bribe-proof and tops-only rules.

Loss of Cutaneous TSLP-Dependent Immune Responses Skews the Balance of Inflammation from Tumor Protective to Tumor Promoting.

Inflammation can promote or inhibit cancer progression. In this study we have addressed the role of the proinflammatory cytokine thymic stromal lymphopoietin (TSLP) during skin carcinogenesis. Using conditional loss- and gain-of-function mouse models for Notch and Wnt signaling, respectively, we demonstrate that TSLP-mediated inflammation protects against cutaneous carcinogenesis by acting directly on CD4 and CD8 T cells. Genetic ablation of TSLP receptor (TSLPR) perturbs T-cell-mediated protection and results in the accumulation of CD11b(+)Gr1(+) myeloid cells. These promote tumor growth by secreting Wnt ligands and augmenting β-catenin signaling in the neighboring epithelium. Epithelial specific ablation of β-catenin prevents both carcinogenesis and the accumulation of CD11b(+)Gr1(+) myeloid cells, suggesting tumor cells initiate a feed-forward loop that induces protumorigenic inflammation.