993 resultados para Lang, Karl Heinrich, Ritter von, 1764-1835.
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v.9 (1866)
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v.14 (1871)
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v.4 (1861)
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v.12 (1869)
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v.3 (1860)
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v.7 (1864)
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This layer is a georeferenced raster image of the historic paper map entitled: Plan der befestigungen u. der hohenverhältnisse der häuser wer stadt Wien im Jahre 1566 : mit zugrundelegung der pläne von Bonifaz Wolmuet und Daniel Suttinger und mite benützung gleichzeitiger handschriftlicher quellen, verfast von Albert Cámesina Ritter von San Vittore ; Alb. R. v. Cámesina fec. 1878. It was published by Gemeinderathe der Reichshaupt u. Residenzstadt in 1880. Scale [ca 1:1,800]. Map in German. Covers Vienna, Austria in 1566. The image inside the map neatline is georeferenced to the surface of the earth and fit to the MGI 3-Degree Gauss Kruger coordinate system. All map collar and inset information is also available as part of the raster image, including any inset maps, profiles, statistical tables, directories, text, illustrations, index maps, legends, or other information associated with the principal map. This map shows features such as roads, drainage, built-up areas and selected buildings with street address and colored to show building uses, landowner names, and more. Includes index. This layer is part of a selection of digitally scanned and georeferenced historic maps from The Harvard Map Collection as part of the Imaging the Urban Environment project. Maps selected for this project represent major urban areas and cities of the world, at various time periods. These maps typically portray both natural and manmade features at a large scale. The selection represents a range of regions, originators, ground condition dates, scales, and purposes.
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Intraerythrocytic growth of the human malaria parasite Plasmodium falciparum depends on delivery of nutrients. Moreover, infection challenges cell volume constancy of the host erythrocyte requiring enhanced activity of cell volume regulatory mechanisms. Patch clamp recording demonstrated inwardly and outwardly rectifying anion channels in infected but not in control erythrocytes. The molecular identity of those channels remained elusive. We show here for one channel type that voltage dependence, cell volume sensitivity, and activation by oxidation are identical to ClC-2. Moreover, Western blots and FACS analysis showed protein and functional ClC-2 expression in human erythrocytes and erythrocytes from wild type (Clcn2(+/+)) but not from Clcn2(-/-) mice. Finally, patch clamp recording revealed activation of volume-sensitive inwardly rectifying channels in Plasmodium berghei-infected Clcn2(+/+) but not Clcn2(-/-) erythrocytes. Erythrocytes from infected mice of both genotypes differed in cell volume and inhibition of ClC-2 by ZnCl(2) (1 mm) induced an increase of cell volume only in parasitized Clcn2(+/+) erythrocytes. Lack of ClC-2 did not inhibit P. berghei development in vivo nor substantially affect the mortality of infected mice. In conclusion, activation of host ClC-2 channels participates in the altered permeability of Plasmodium-infected erythrocytes but is not required for intraerythrocytic parasite survival.
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Includes bibliographical references (p. 33-34).
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Mode of access: Internet.
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Mode of access: Internet.
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Bibliographical footnotes.
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Mode of access: Internet.
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"Quellen und abkürzungen": p. 70.