Biblioteca Digital

910 resultados para Benign prostate hyperplasia

The pharmacokinetic and tolerability profile of once-daily Zibotentan (ZD4054) in Japanese and Caucasian patients with hormone-resistant prostate cancer.

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Does dexamethasone work in androgen-independent prostate cancer (AIPC) by modulation of interleukin-8?

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A double blind, randomised, dose finding, phase II, multicentre study of radium-223 for the treatment of patients with metastatic castration-refractory prostate cancer (CRPC): EudraCT number: 2005-003680-22

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High activity rhenium-186 hydroxyethylidene diphosphonate (HEDP) with autologous peripheral blood stem cell (PBSC) transplant: A novel treatment strategy in hormone refractory prostate cancer metastatic to bone.

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Comparison of PSA measurements and scintigraphy in patients with skeletal metastases from carcinoma of the prostate treated with high activities of Re 186 HEDP.

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An Observational Study Assessing Bladder Volume Consistency during 3D Conformal Prostate Radiotherapy using an Ultrasonic Bladderscan Device

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The optimum method to determine the maximum tolerated activity for high activity Re186HEDP therapy with peripheral blood stem cell support for skeletal metastases from prostate cancer

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Prediction of response of skeletal metastases from cancer of the prostate to high activity Re-186 HEDP therapy.

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Autologous peripheral blood stem cell rescue permits administration of high activities of rhenium-186 HEDP in progressive hormone refractory prostate cancer metastatic to bone

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The relationship between skeletal metastatic load and therapeutic activity requirement in prostate cancer

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A randomised controlled trial to evaluate the efficacy of a 6 month dietary and physical activity intervention for prostate cancer patients receiving androgen deprivation therapy.

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Strategies for the Implementation of Chemotherapy and Radiotherapy for Metastatic Prostate Cancer

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Balancing the Harms and Benefits of Early Detection of Prostate Cancer

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Hyperacetylation in prostate cancer induces cell cycle aberrations, chromatin reorganisation and altered gene expression profiles.

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Changes in chromatin phenotype predict response to hormonal deprivation therapy in prostate cancer patients.

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