Alcohol and alcohol safety. A curriculum manual for elementary level. Volume II of II. A teacher's activities guide.

National Highway Traffic Safety Administration, Washington, D.C.

Lincoln Alcohol Safety Action Project. Roadside survey report. 1971-1972.

National Highway Traffic Safety Administration, Washington, D.C.

A household survey on alcohol and traffic safety issues. Analytical study no. 7.

National Highway Traffic Safety Administration, Washington, D.C.

On-the-road driving behavior and breath alcohol concentration. Final report.

National Highway Traffic Safety Administration, Washington, D.C.

Alcohol and alcohol safety. A curriculum manual for junior high level. Volume II of II. A teacher's activities guide.

National Highway Traffic Safety Administration, Washington, D.C.

Analysis of the spring 1976 telephone survey of community opinion and knowledge.

National Highway Traffic Safety Administration, Office of Driver and Pedestrian Programs, Washington, D.C.

Analysis of a random-assignment short term rehabilitation program. Analytic study 5 and 6.

National Highway Traffic Safety Administration, Office of Driver and Pedestrian Programs, Washington, D.C.

Short term rehabilitation (STR) study. Interim analysis of STR performance and effectiveness. 12-month analyses. Interim report.

National Highway Traffic Safety Administration, Office of Driver and Pedestrian Programs, Washington, D.C.

Alcohol and alcohol safety. A curriculum manual for senior high level. Volume I of II.

National Highway Traffic Safety Administration, Washington, D.C.

Alcohol and alcohol safety. A curriculum manual for elementary level. Volume I of II.

National Highway Traffic Safety Administration, Washington, D.C.

Alcohol and alcohol safety. A curriculum manual for junior high level. Volume I of II.

National Highway Traffic Safety Administration, Washington, D.C.

Roadside survey, Utah ASAP. Evaluation extention final report.

National Highway Traffic Safety Administration, Washington, D.C.

Public information and education. Analytic study no. 7. Final study.

National Highway Traffic Safety Administration, Office of Driver and Pedestrian Programs, Washington, D.C.

The relationship between acute alcohol consumption and consequent injury type

Aims: The aim of this study was to quantify the relationship between acute alcohol consumption and injury type (nature of injury, body region injured), while adjusting for the effect of known confounders (i.e. demographic and situational variables, usual drinking patterns, substance use and risk-taking behaviour). Methods: A cross-sectional study was conducted between October, 2000 and October, 2001 of patients aged >= 15 years presenting to a Queensland Emergency Department for treatment of an injury sustained in the preceding 24 h. There were three measures of acute alcohol consumption: drinking setting, quantity, and beverage type consumed in the 6 h prior to injury. Two variables were used to quantify injury type: nature of injury (fracture/dislocation, superficial, internal, and CNS injury) and body part injured (head/neck, facial, chest, abdominal, external, and extremities). Both were derived from patient medical records. Results: Five hundred and ninety three patients were interviewed. Logistic regression analyses indicated that, after controlling for relevant confounding variables, there was no significant association between any of the three measures of acute alcohol consumption and injury type. Conclusions: The effects of acute alcohol consumption are not specific to injury type. Interventions aimed at reducing the incidence of alcohol-related injury should not be targeted at specific injury types.

Acute alcohol consumption and mechanism of injury

Objective: The purpose of this study was to determine whether injury mechanism among injured patients is differentially distributed as a function of acute alcohol consumption (quantity, type, and drinking setting). Method: A cross-sectional study was conducted between October 2000 and October 2001 in the Gold Coast Hospital Emergency Department, Queensland, Australia. Data were collected quarterly over a 12-month period. Every injured patient who presented to the emergency department during the study period for treatment of an injury sustained less than 24 hours prior to presentation was approached for interview. The final sample comprised 593 injured patients (males = 377). Three measures of alcohol consumption in the 6 hours prior to injury were obtained from self-report: quantity, beverage type, and drinking setting. The main outcome measure was mechanism of injury which was categorized into six groups: road traffic crash (RTC), being hit by or against something, fall, cut/piercing, overdose/poisoning, and miscellaneous. Injury intent was also measured (intentional vs unintentional). Results: After controlling for relevant confounding variables, neither quantity nor type of alcohol was significantly associated with injury mechanism. However, drinking setting (i.e., licensed premise) was significantly associated with increased odds of sustaining an intentional versus unintentional injury (odds ratio [OR] = 2.79, 95% confidence interval [CI] = 1.4-5.6); injury through being hit by/against something versus other injury types (OR = 2.59, 95% CI = 1.4-4.9); and reduced odds of sustaining an injury through RTC versus non-RTC (OR = 0.02, 95% CI = 0.004-0.9), compared with not drinking alcohol prior to injury. Conclusions: No previous analytical studies have examined the relationship between injury mechanism and acute alcohol consumption (quantity, type, and setting) across all types of injury and all injury severities while controlling for potentially important confounders (demographic and situational confounders, risk-taking behavior, substance use, and usual drinking patterns). These data suggest that among injured patients, mechanism of injury is not differentially distributed as a function of quantity or type of acute alcohol consumption but may be differentially distributed as a function of drinking setting (i.e., RTC, intentional injury, being hit). Therefore, prevention strategies that focus primarily on the quantity and type of alcohol consumed should be directed generically across injury mechanisms and not limited to particular cause of injury campaigns.