Jesus-Christ, par sa tolerance, modele des Legislateurs.

Mode of access: Internet.

Tables of factors for one-sided tolerance limits for normal distribution,

Mode of access: Internet.

Mechanisms of tolerance and dependence /

Mode of access: Internet.

Tolerance of dowel bar placement.

Mode of access: Internet.

Through Gentile eyes; a plea for tolerance and good will,

Electronic text and image data

Changing headlines : building tolerance in the land of Lincoln : interim report for the Governor's Commission on Discrimination & Hate Crimes.

Cover title.

Rhymed plea for tolerance. In two dialogues. With a prefatory dialogue.

Mode of access: Internet.

On mutual tolerance, and on the ultimate test of truth: occasioned by a recent publication, entitled "A beacon to the Society of Friends" /

Bound with [2. Thompson, T.] A letter by Thomas Thompson. 1835. -[3] Tuke, S. Letter to John Wilkinson. 2d ed., 1836. -[4] Friends and Independents. 1836. -[5] Eaton, J. Barclay and Penn self-vindicated. 1836. -[6] Gurney, J.J. Strictures on certain parts of an anonymous pamphlet. 1836. -[7. Martin, H.] Philo answered. 1836. -[8. Treffry, J.] Strictures on a late publication. 1836. -[9. Martin, H.] A defence of the original principles. 1836. -[10. Society of Friends - London Yearly Meeting] The committe of the Yearly Meeting, appointed to visit and assist Lancashire Quarterly Meeting. 1835. -[11] Boulton, W. Three essays. 1836. [12. Braithwaite, J. B., comp.] Extracts from the writings of the early Friends. 1836. -[13] Early Friends and Dr. Ash. 1837. -[14] Lean, W. Brief observations. 1838. -[15. Howard, L.] An appeal to the Christian public. 1838. -[16] Bereus, pseud. A defence of the Friends' doctrine of baptism. 1838. -[17] Richardson, W. A scriptural examination. 1839,

Roots; a plea for tolerance.

Mode of access: Internet.

Tolerance of certain crops to several aquatic herbicides in irrigation water /

Caption title.

Etude sur quelques points de l'histoire de la tolerance au cana...

Thesis (doctoral)--

Beitrag zur Kenntnis der Arsenate des Zinks /

Thesis (doctoral)--Friedrich-Wilhelms-Universitat zu Berlin, 1895.

Induction and transmission of Bacillus thuringiensis tolerance in the flour moth Ephestia kuehniella

The use of Bacillus thuringiensis (Bt) endotoxins to control insect vectors of human diseases and agricultural pests is threatened by the possible evolution of resistance in major pest species. In addition to high levels of resistance produced by receptor insensitivity (5, 16, 17), several cases of tolerance to low to medium levels of toxin have been reported in laboratory colonies of lepidopteran species (3, 18). Because the molecular basis of some of these cases of tolerance to the toxin are not known, we explored alternative mechanisms. Here, we present evidence that tolerance to a Bt formulation in a laboratory colony of the flour moth Ephestia kuehniella can be induced by preexposure to a low concentration of the Bt formulation and that the tolerance correlates with an elevated immune response. The data also indicate that both immune induction and Bt tolerance can be transmitted to offspring by a maternal effect and that their magnitudes are determined by more than one gene.

Deletion of guanine nucleotide binding protein az subunit in mice induces a gene dose dependent tolerance to morphine

The Mechanism Underlying the development of tolerance to morphine, is still incompletely understood. Morphine binds to opioid receptors, Which in turn activates downstream second messenger cascades through heterotrimeric guanine nucleotide binding proteins (G proteins). In this paper, we show that G(z), a member of the inhibitory G protein family, plays an important role in mediating the analgesic and lethality effects of morphine after tolerance development. We blocked signaling through the G(z) second messenger cascade by genetic ablation of the alpha subunit of the G protein in mice. The Galpha(z) knockout Mouse develops significantly increased tolerance to morphine. which depends oil Galpha(z), gene dosage. Further experiments demonstrate that the enhanced morphine tolerance is not caused by pharmacokinetic and behavioural learning mechanisms. The results suggest that G(z) signaling pathways are involved ill transducing the analgesic and lethality effects of morphine following chronic morphine treatment. (C) 2004 Elsevier Ltd. All rights reserved.