The modern band: a treatise on wind instruments, symphony band and military band,

Bk.I. pt.1. Wind instruments. - pt.2. Band instrumentation. -Bk.II. Score examples.

The Young and Field literary readers /

Bk. 1. A primer and first reader.

Mexico,

v. 1. bk. I. Boundaries [etc.] Population, Productions, Spanish colonial system. bk. II. [The wars of independence] bk. III. Government, Navy and Army, Religion, Revenue, Trade. Appendix of documents 1809-21. bk. IV. The mines of Mexico.--v. 2. bd. V-VI. Personal narrative. Particulars of a journey from Altamira to Catorce, by Robert Phillips. Account of the province of Texas, by General Wavel. Notes on the state of Sonora and Cinaloa, by Colonel Bourne. Annexed documents.

Hin forna lögbók Islendínga sem nefnist Grágás. Codex juris Islandorum antiqvissimus, qvi nominatur Grágás.

Added t.-p., engraved.

Kvæði Eggerts Ólafssonar : útgefin eptir þeim beztu handritum er feingizt gátu."

Edited and published by E. Jónsson, Th. Sæmundsson, and S. Thorarensen.

Kloppstokks Messías, einn hetju-diktr um endrlausnina,

Called 1-20 bók.

Human geography /

Cover-title.

Platons ausgewählte schriften : für den schulgebrauch /

v.1 Apology, Crito ed Cron, 1888;--v.2 Gorgias, ed. Deuschle 1886;--v.3 Laches, Euthyphro, ed. Cron, 1882;--v.4 Protagoras ed. Deuschle, 1884;--v.5 Symposium ed. Arnold Hug, 1884;--v.6 Phaedo, ed. Martinus Wohlrab, 1884;--v.7 Republic bk. 1 ed. Wohlrab, 1893.

Geschiedenis der Middennederlandsche dichtkunst.

CONTENTS: 1. deel. Voorbericht. Naamlijst van inteekenaren. Inleiding. 1. bk. Oudste volkspoëzie. 2. bk. Aanvang der ridderpoëzie.--2. deel. 3. bk. Bloei der ridderpoëzie. 4. bk. Verval der ridderpoëzie.--3. deel. 5. bk. Burgerlijke poëzie. 6. bk. Poëtische terugwerking. 7. bk. Lyrische en dramatische poëzie. Bijlagen. Uittreksels uit de ms. rekeningen der graven van Holland en van Bloys, berustende op hets Rijks archief te's Hage.

The Elson readers / by William H. Elson.

Primer. Revised edition of Elson-Runkel primer.--Bks.1-4. Revision of Elson primary school reader, books one to four.--Bks.5-6. Revision of Elson grammar school reader, books one to two.--Bk.7. Revision of junior high school literature, book one.

Muriel Sterling : a tale of the African veldt /

Wright, L.H. Amer. fiction, 1876-1900,

The works of Francis Rabelais, M.D.

Classified for chess, bk. 5, ch. 25-26.

A text book of physiology /

Includes indexes.

Population trends in Wisconsin urbanized areas and other cities of over 10,000, 1970-1975 /

WI docs no.: Adm.3/2:P 6/4

Epstein-Barr virus-mediated protection against etoposide-induced apoptosis in BJA-B B cell lymphoma cells: role of Bcl-2 and caspase proteins

Epstein-Barr virus (EBV)-infected B cell lymphomas are resistant to apoptosis during cancer development and treatment with therapies. The molecular controls that determine why EBV infection causes apoptosis resistance need further definition. EBV-positive and EBV-negative BJA-B B cell lymphoma cell lines were used to compare the expression of selected apoptosis-regulating Bcl-2 and caspase proteins in EBV-related apoptosis resistance, after 8 hr or 18-24 hr etoposide treatment (80 muM). Apoptosis was quantified using morphology and verified with Hoechst 33258 nuclear stain and electron microscopy. Fluorescence activated cell sorting (FACS) was used to analyse effects on cell cycle of the EBV infection as well as etoposide treatment. Anti-apoptotic Bcl-2 and Bcl-XL, pro-apoptotic Bax, caspase-3 and caspase-9 expression and activation were analysed using Western immunoblots and densitometry. EBV-positive cultures had significantly lower levels of apoptosis in untreated and etoposide-treated cultures in comparison with EBV-negative cultures (p < 0.05). FACS analysis indicated a strong G2/M block in both cell sublines after etoposide treatment. Endogenous Bcl-2 was minimal in the EBV-negative cells in comparison with strong expression in EBV-positive cells. These levels did not alter with etoposide treatment. Bcl-XL was expressed endogenously in both cell lines and had reduced expression in EBV-negative cells after etoposide treatment. Bax showed no etoposide-induced alterations in expression. Pro-caspase-9 and -3 were seen in both EBV-positive and -negative cells. Etoposide induced cleavage of caspase-9 in both cell lines, with the EBV-positive cells having proportionally less cleavage product, in agreement with their lower levels of apoptosis. Caspase-3 cleavage occurred in the EBV-negative etoposide-treated cells but not in the EBV-positive cells. The results indicate that apoptosis resistance in EBV-infected B cell lymphomas is promoted by an inactive caspase-3 pathway and elevated expression of Bcl-2 that is not altered by etoposide drug treatment.