45 resultados para Locusts.


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Mode of access: Internet.

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"This edition is limited to one thousand sets".

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CONTENTS.--vol. I, no. 1. Notes on Indian insect pests. 1889. no. 2. Notes on Indian economic entomology. 1889. no. 3. Silkworms in India. 1890. no. 4. Notes on Indian economic entomology. 1890.--vol. II, no. 1. Economic entomology. 1891. no. 2. The wild silk insects of India. 1891. no. 3. On white insect wax in India. 1891. no. 4. The locusts of Bengal, Madras, Assam, and Bombay. 1891. no. 5. [Economic entomology, etc.] 1891. no. 6. A conspectus of the insects which affect crops in India. 1893.--vol. III, no. 1-3. [Notes on Indian insect pests, etc.] 1893-94. no. 4. An account of the insects and mites which attack the tea plant in India. 1895. no. 5-6. [Entomological notes] 1894-96.--vol. IV-vol. VI, no. 1. [Notes on Indian insect pests] 1896-1903.

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Publication 3335.

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"Autograph edition limited to 750 copies."

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Includes bibliographical references (p. 46).

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Literature cited: p. 28-29.

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Includes bibliographical references (p. 261-286) and index.

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Cover title.

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Advertisements on p. 4 at front and p. [2]-[12] at end.

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Papers reprinted from the Overland monthly and the Californian.

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Theories of sparse signal representation, wherein a signal is decomposed as the sum of a small number of constituent elements, play increasing roles in both mathematical signal processing and neuroscience. This happens despite the differences between signal models in the two domains. After reviewing preliminary material on sparse signal models, I use work on compressed sensing for the electron tomography of biological structures as a target for exploring the efficacy of sparse signal reconstruction in a challenging application domain. My research in this area addresses a topic of keen interest to the biological microscopy community, and has resulted in the development of tomographic reconstruction software which is competitive with the state of the art in its field. Moving from the linear signal domain into the nonlinear dynamics of neural encoding, I explain the sparse coding hypothesis in neuroscience and its relationship with olfaction in locusts. I implement a numerical ODE model of the activity of neural populations responsible for sparse odor coding in locusts as part of a project involving offset spiking in the Kenyon cells. I also explain the validation procedures we have devised to help assess the model's similarity to the biology. The thesis concludes with the development of a new, simplified model of locust olfactory network activity, which seeks with some success to explain statistical properties of the sparse coding processes carried out in the network.

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Temperature has profound effects on the neural function and behaviour of insects. When exposed to low temperature, migratory locusts (Locusta migratoria) enter chill coma (neuromuscular paralysis) and can resume normal body functions after returning to normal temperature. Our laboratory has studied phenomena underlying environmental stress-induced comas in locusts and found that they are associated with a sudden loss of K+ homeostasis and also a temporary electrical silence in the central nervous system (CNS). However, the mechanisms underlying chill coma entry and recovery are not well understood, particularly the role of the CNS has not been determined. Here, I investigated neural function during chill coma in the locust by measuring electrical activity in the CNS. As pre-exposure to moderately low temperatures, either chronically (cold acclimation) or acutely (rapid cold hardening; RCH), has been found to improve the insect’s cold tolerance, I also determined cold acclimation and RCH protocols that will improve the locust's cold tolerance and whether these protocols affect neural shutdown during chill coma in the locust. With an implanted thermocouple in the thorax, I determined the temperature associated with a loss of responsiveness (CTmin) in intact male adult locusts. In parallel experiments, I recorded field potential (FP) in the metathoracic ganglion (MTG) in semi-intact preparations to determine the temperature that would induce neural shutdown. I found that acclimation at 10 ˚C and RCH at 4 ˚C reduced chill coma recovery time (CCRT) in intact animal preparations and RCH at 4 ˚C for 4 hours reduced the temperature at neural shutdown in semi-intact preparations. These results suggest that pre-exposure to cold can improve the locust's resistance to chill coma and support the notion that the CNS has a role in determining entry into and exit from chill coma in locusts.