1000 resultados para I-ks
Resumo:
Soitinnus: mieskuoro
Resumo:
Soitinnus: lauluääni, piano.
Resumo:
Soitinnus: piano.
Resumo:
Soitinnus: lauluääni, piano.
Resumo:
Soitinnus: piano.
Resumo:
Soitinnus: puheääni, orkesteri
Resumo:
Soitinnus: lauluääni, viulu.
Resumo:
Soitinnus: lauluääni, piano.
Resumo:
Soitinnus: lauluääni, piano.
Resumo:
Soitinnus: lauluääni, piano.
Resumo:
Soitinnus: orkesteri.
Resumo:
Melartinin käsikirjoitusluettelon tunnus Mel 4:24 (ks. http://lib.siba.fi/fi/kokoelmat/kasikirjoitusarkisto/melartin_erkki/).
Resumo:
Ilmestyi alunperin 7 vihkona vuosina 1898-1899. Vihkojen 1-6 kansilehdillä: Sähkö : oppikirja sähkön alalla : käsikirja sähkötyömiehille : ruotsalaisen Karl Wallinin ja saksalaisen S. Frhr. Gaisbergin mukaan useain ammattimiesten suosiollisella avulla. Vihkojen 1-3 kansilehdillä lisäksi: toimittanut J. G. Toivonen. Vihkon 7 kansilehdellä: Sähkö. I, Sähkö ja sen käyttäminen valaistukseen ja työvoiman siirtämiseen / Karl Wallinin mukaan. - Gaisbergin osuus ilmestynyt nähtävästi erikseen, ks.: Käsikirja sähkövalaistuslaitosten asettajille.
Resumo:
The Kaposi-associated Herpesvirus (KSHV) also known as Human Herpesvirus 8 (HHV-8) is associated with the development of Kaposi’s sarcoma (KS) and others limphoprolipheratives diseases such as Primary Effusion Lymphoma (PEL) and Multicentric Castleman Disease (MCD). Even though the virus is considered lymphotropic, it is able to infect others cell types such as macrophages, dendritic cells, endothelial cells, monocytes and fibroblasts. After infection, KSHV be latent expressing essential viral genes to its maintenance in a infected cell. However, in some circumstances may occur the reactivation of lytic cycle producing new viral particles. K1 protein of KSHV interferes in the cellular signaling inducing proliferation and supporting cellular transformation. K1 is encoded by viral ORF-K1, which shows high variability between different genotypes of KSHV. So far, it is not clear whether different isoforms of K1 have specific immunobiological features. The KSHV latency is maintained under strict control by the immune system supported by an adequate antigen presentation involving Human Leucocyte Antigen (HLA) class I and II. Polymorphisms of HLA class I and II genes confer an enormous variability in molecules that recognize a large amount of antigens, but also can increase the susceptibility to autoimmune diseases. Therefore, the present study aims to genotype HLA class I (A and B) and class II (DR and DQ) from volunteers to identify haplotypes that can provide better response to K1 epitopes of different KSHV genotypes. First of all, 20 volunteers were selected to genotype HLA genes. In our results we observed prevalence of certain HLA class I haplotypes as HLAA1, HLA-A2, HLA-A24, HLA-A26, HLA-B8, HLA-B18 e HLA-B44. After the in silico analysis using BIMAS and SYFPEITHI databases, we observed high scores for epitopes from the B genotype of KSHV, indicating...(Complete abstract click electronic access below)
Resumo:
This article gives details of our proposal to replace ordinary chiral SU(3)L×SU(3)R perturbation theory χPT3 by three-flavor chiral-scale perturbation theory χPTσ. In χPTσ, amplitudes are expanded at low energies and small u,d,s quark masses about an infrared fixed point αIR of three-flavor QCD. At αIR, the quark condensate ⟨q¯q⟩vac≠0 induces nine Nambu-Goldstone bosons: π,K,η, and a 0++ QCD dilaton σ. Physically, σ appears as the f0(500) resonance, a pole at a complex mass with real part ≲ mK. The ΔI=1/2 rule for nonleptonic K decays is then a consequence of χPTσ, with a KSσ coupling fixed by data for γγ→ππ and KS→γγ. We estimate RIR≈5 for the nonperturbative Drell-Yan ratio R=σ(e+e−→hadrons)/σ(e+e−→μ+μ−) at αIR and show that, in the many-color limit, σ/f0 becomes a narrow qq¯ state with planar-gluon corrections. Rules for the order of terms in χPTσ loop expansions are derived in Appendix A and extended in Appendix B to include inverse-power Li-Pagels singularities due to external operators. This relates to an observation that, for γγ channels, partial conservation of the dilatation current is not equivalent to σ-pole dominance.