2001 University of Michigan Football Team

School of Nursing Class of 2010

Top Row: Jocelyn Aden, Rachel Ades, Katrina Allen, Kayla Ashcraft, Kristie Baker, Amy Beaudoin, Heidi Beck, Beth Bentrum, Amber Blake, Lee Anna Braden, Dan Burd, Meaghan Burke, Mallory Calus, Irene Casillas, Veronica Cherney, Samantha Cholewa, Molly Conlen

Row 2: Wendy Corriveau, Meaghan Cotter, Kara DeGlopper, Colleen DeVoe, Hadley Dobbs, Kimberly Drury-Wallace, Hyesun Eitel, Sarah Elner, Douglas E. Elsey, Alyssa Fallot, Folake Famoye, Kristen Farr, Christine Fleck, Jennifer Fleming, Soncerae Gardner, Sarah Gilley, Joelle Gilmet

Row 3: Sara Goss, Amy Guffey, Taylor Griglak, Bridget Belvitch, Jaclyn Janks, Andrea Engles, Cassandra Smith, Lyndsy Brenner, Mallorie Patterson, Kristen Oltersdorf, Laura Kokx, Ross Zoet, Mary Osbach, Courtney Norman, Monica Habeck, Erica Hadley

Row 4: Amanda Hanert, Dayna Hasty, Nicole Heller, Ashley Howard, Robert Humburg, Andrew Humes, Grace Hwang, Amira Jackson, Kathryn Jipping, Shelly Johnson

Row 5: Lindsey Kappler, Jacqueline Klaiman, Sarah Knoedler, Jessica Kopicki, Kathryn Kovanda, Sarah Kovats, Emily Krogel, Kellie Kunkel, Kristin Lakatos, Chelsea Lazaroff, Bo Hwa Lee, Kelly Leja</p>

Row 6: Kelli Littlejohn, Emilee Losey, Patricia Luna, Wilma MacKenzie, Matt Malkowski, Rachel Mallas, Emily McCallister, Diane McDonald, Dorian Michelson, Mary Miller, Nicole Miller, Kristen Muehlhauser

Row 7: Renee Muller, Katherine Mulvaney, Eugene Ngala, Christine Novotny, Colleen O'Connor, Cassey Parrish, Kimberly Peters, Kathleen Potempa, Bonnie Hagerty, Heather Poucher, Charles Reisdorf, Eric Retzbach, Sarah Rhem, Shannon Rice, Amy Roberts, Christie Schonsheck

Row 8: Franciska Schuett, Rhonda Schultz, Kristina Seidl, Teresa Semaan, Shelley Sibbold, Stacy Slater, Mary Snell, Mallory Stanton, Dennis Stevens, Miranda Stoddard, Tatiana Tafla, Priscilla Tang, Bethany Thelen, Jessica Thibert, Rebecca Thurk, Lauren Tormoehlen, Chinasa Uwandu

Row 9: Margaret van Buitenen, Stacey Victor, Jennifer Waag, Kirstyn Wade, Ariel Warren, Elizabeth White, Natalie Wierenga, Jessica Wihowski, Wendy Witkowski, Aliza Wolfe, DaShaunn Woolard, Ting Wan Yip, Alexander Young, Kellie Zenz, Kristen Ziulkowski, Jessica Zmierski

Accounting for a meat packing business,

Reproduced from type-written copy.

Excelsior; or, Essays on politeness, education, and the means of attaining success in life ...

pt. I. For young gentlemen, by T. E. Howard.--pt. II. For young ladies, by a lady, (R. V. R.)

The American Nation: a history from original sources by associated scholars;

Vols. 1, 3-12, 14-16, 18-20, 22, 25-27 have no date on t. p.

The Techialoyan Codices and their Number

In this article, it is proposed a brief revision of the number of Techialoyan codices. To do this, the text starts with a definition of the corpus, that it is composed of manuscripts done between the half of the 17th century and beginning of the next. Around these documents there are many doubts and it remains much to do. In this case, we present a review around the lists and catalogues to see the changes of the group through time. Our conclusion is that is necessary recount the corpus, because there are fragmented documents and others are unknowns or copies. Finally, we show our count, that leaves the number less than fifty-six documents catalogued until today.

Boys In White: A Classic Of Qualitative Research Turns 50.

This article analyzes Boys in white: student culture in medical schoolby Howard S. Becker, Blanche Geer, Everett C. Hughes and Anselm Strauss, considered a model of qualitative research in sociology. The analysis investigates the trajectories of the authors, the book, qualitative analysis, and the medical students, emphasizing their importance in the origins of medical sociology and the sociology of medical education. In the trajectory of the authors, bibliographical information is given. The trajectory of qualitative research focuses on how this methodology influences the construction of the field. The investigation of the students' trajectory shows how they progress through their first years at medical school to build their own student culture.

National Institutes Of Health Consensus Development Project On Criteria For Clinical Trials In Chronic Graft-versus-host Disease: I. The 2014 Diagnosis And Staging Working Group Report.

The 2005 National Institutes of Health (NIH) Consensus Conference proposed new criteria for diagnosing and scoring the severity of chronic graft-versus-host disease (GVHD). The 2014 NIH consensus maintains the framework of the prior consensus with further refinement based on new evidence. Revisions have been made to address areas of controversy or confusion, such as the overlap chronic GVHD subcategory and the distinction between active disease and past tissue damage. Diagnostic criteria for involvement of mouth, eyes, genitalia, and lungs have been revised. Categories of chronic GVHD should be defined in ways that indicate prognosis, guide treatment, and define eligibility for clinical trials. Revisions have been made to focus attention on the causes of organ-specific abnormalities. Attribution of organ-specific abnormalities to chronic GVHD has been addressed. This paradigm shift provides greater specificity and more accurately measures the global burden of disease attributed to GVHD, and it will facilitate biomarker association studies.

A estimulação da inteligencia corporal cinestesica no contexto na educação fisica escolar

Universidade Estadual de Campinas. Faculdade de Educação Física

Possibilidades corporais como expressão da inteligencia humana no processo de ensino-aprendizagem

Universidade Estadual de Campinas . Faculdade de Educação Física

As emoções na educação fisica escolar

Universidade Estadual de Campinas . Faculdade de Educação Física

O Serviço de Recreação Operaria e a sociabilização do trabalhador sindicalizado no Brasil : 1943-1964

Universidade Estadual de Campinas . Faculdade de Educação Física

Genomic islands in the pathogenic filamentous fungus Aspergillus fumigatus

We present the genome sequences of a new clinical isolate of the important human pathogen, Aspergillus fumigatus, A1163, and two closely related but rarely pathogenic species, Neosartorya fischeri NRRL181 and Aspergillus clavatus NRRL1. Comparative genomic analysis of A1163 with the recently sequenced A. fumigatus isolate Af293 has identified core, variable and up to 2% unique genes in each genome. While the core genes are 99.8% identical at the nucleotide level, identity for variable genes can be as low 40%. The most divergent loci appear to contain heterokaryon incompatibility ( het) genes associated with fungal programmed cell death such as developmental regulator rosA. Cross-species comparison has revealed that 8.5%, 13.5% and 12.6%, respectively, of A. fumigatus, N. fischeri and A. clavatus genes are species-specific. These genes are significantly smaller in size than core genes, contain fewer exons and exhibit a subtelomeric bias. Most of them cluster together in 13 chromosomal islands, which are enriched for pseudogenes, transposons and other repetitive elements. At least 20% of A. fumigatus-specific genes appear to be functional and involved in carbohydrate and chitin catabolism, transport, detoxification, secondary metabolism and other functions that may facilitate the adaptation to heterogeneous environments such as soil or a mammalian host. Contrary to what was suggested previously, their origin cannot be attributed to horizontal gene transfer ( HGT), but instead is likely to involve duplication, diversification and differential gene loss (DDL). The role of duplication in the origin of lineage-specific genes is further underlined by the discovery of genomic islands that seem to function as designated ""gene dumps'' and, perhaps, simultaneously, as "" gene factories''.

Blood Meal-Derived Heme Decreases ROS Levels in the Midgut of Aedes aegypti and Allows Proliferation of Intestinal Microbiota

The presence of bacteria in the midgut of mosquitoes antagonizes infectious agents, such as Dengue and Plasmodium, acting as a negative factor in the vectorial competence of the mosquito. Therefore, knowledge of the molecular mechanisms involved in the control of midgut microbiota could help in the development of new tools to reduce transmission. We hypothesized that toxic reactive oxygen species (ROS) generated by epithelial cells control bacterial growth in the midgut of Aedes aegypti, the vector of Yellow fever and Dengue viruses. We show that ROS are continuously present in the midgut of sugar-fed (SF) mosquitoes and a blood-meal immediately decreased ROS through a mechanism involving heme-mediated activation of PKC. This event occurred in parallel with an expansion of gut bacteria. Treatment of sugar-fed mosquitoes with increased concentrations of heme led to a dose dependent decrease in ROS levels and a consequent increase in midgut endogenous bacteria. In addition, gene silencing of dual oxidase (Duox) reduced ROS levels and also increased gut flora. Using a model of bacterial oral infection in the gut, we show that the absence of ROS resulted in decreased mosquito resistance to infection, increased midgut epithelial damage, transcriptional modulation of immune-related genes and mortality. As heme is a pro-oxidant molecule released in large amounts upon hemoglobin degradation, oxidative killing of bacteria in the gut would represent a burden to the insect, thereby creating an extra oxidative challenge to the mosquito. We propose that a controlled decrease in ROS levels in the midgut of Aedes aegypti is an adaptation to compensate for the ingestion of heme.

HTLV-1 Tax Specific CD8+ T Cells Express Low Levels of Tim-3 in HTLV-1 Infection: Implications for Progression to Neurological Complications

The T cell immunoglobulin mucin 3 (Tim-3) receptor is highly expressed on HIV-1-specific T cells, rendering them partially ""exhausted'' and unable to contribute to the effective immune mediated control of viral replication. To elucidate novel mechanisms contributing to the HTLV-1 neurological complex and its classic neurological presentation called HAM/TSP (HTLV-1 associated myelopathy/tropical spastic paraparesis), we investigated the expression of the Tim-3 receptor on CD8(+) T cells from a cohort of HTLV-1 seropositive asymptomatic and symptomatic patients. Patients diagnosed with HAM/TSP down-regulated Tim-3 expression on both CD8(+) and CD4(+) T cells compared to asymptomatic patients and HTLV-1 seronegative controls. HTLV-1 Tax-specific, HLA-A*02 restricted CD8(+) T cells among HAM/TSP individuals expressed markedly lower levels of Tim-3. We observed Tax expressing cells in both Tim-3(+) and Tim-3(-) fractions. Taken together, these data indicate that there is a systematic downregulation of Tim-3 levels on T cells in HTLV-1 infection, sustaining a profoundly highly active population of potentially pathogenic T cells that may allow for the development of HTLV-1 complications.