Footnotes November/December 2002, Vol.26, no.11/12

Monthly newsletter for the State Library of Iowa.

Footnotes December 2001, Vol.25, no.12

Monthly newsletter of the State Library of Iowa

Footnotes December 2000, Vol.24,no.12

Monthly newsletter of the State Library of Iowa

ABD e-News, September 12, 2003

Weekly newsletter for the State of Iowa Alcoholic Beverages Division

[Costante] Girardengo [coureur cycliste italien disputant une course au vélodrome d'hiver le 7-12-19] : [photographie de presse] / [Agence Rol]

Référence bibliographique : Rol, 57116

12

This chapter discusses how the industrial ecological systems can help in dealing with environmental issues in developing countries, and it presents three case studies from India that highlight some of the unique environmental issues of developing world. Industrial ecology explores the assumption that the industrial system can be seen as a certain kind of ecosystem. The scope of industrial ecology goes well beyond waste exchange to the optimization of resources flowing through the economic system. Among the various specific aspects of developing countries, which have to be born in mind, is the fact that the pattern of resource flows in developing countries, and hence, the resultant environmental threat could be very different than what it is in the industrialized west. Typically, the flow of materials through the large, organized manufacturing facilities in the developing countries could be very small in relation to the overall material flow as the small, informal ?industry? plays a key role and forms a very significant portion of the economic activity. The case studies of the Tirupur textile industries, and the leather industry in India, illustrate how redefining the problem from a perspective of resource conservation, and on the basis of resource flow data could point to totally new directions for strategy planning. The case study of the Damodar Valley region amplifies the importance of looking beyond formal industry to solve an environmental problem. It shows that even for globally critical programs, such as climate change program in developing countries, it is just not enough to estimate the emissions from the formal industrial sectors.

Iowa Developments, January 2003, Vol.12, no.1

Report of development in Iowa

Iowa Developments, April 2003, Vol.12, no.2

Resource to help educate businesses or communities about state-sponsored programs.

Lottery Action, June 16-29, 2003, Vol. 10, no. 12

Newsletter for Iowa Lottery

[12] rue Dussourd à Asnières [crue de la Seine début janvier 1920] : [photographie de presse] / [Agence Rol]

Référence bibliographique : Rol, 57517

Missing Persons Summary, May 12, 2003

Missing persons summary

Genetically resistant mice lacking interleukin-12 are susceptible to infection with Leishmania major and mount a polarized Th2 cell response.

Mice with homologous disruption of the gene coding for either the p35 subunit or the p40 subunit of interleukin-12 (IL-12) and derived from a strain genetically resistant to infection with Leishmania major have been used to study further the role of this cytokine in resistance to infection and the differentiation of functional CD4+ T cell subsets in vivo. Wild-type 129/Sv/Ev mice are resistant to infection with L. major showing only small lesions which resolve spontaneously within a few weeks and develop a type 1 CD4+ T cell response. In contrast, mice lacking bioactive IL-12 (IL-12p35-/- and IL-12p40-/-) developed large, progressing lesions. Whereas resistant mice were able to mount a delayed-type hypersensitivity (DTH) response to Leishmania antigen, susceptible BALB/c mice as well as IL-12-deficient 129/Sv/Ev mice did not show any DTH reaction. To characterize the functional phenotype of CD4+ T cells triggered in infected wild-type mice and IL-12-deficient mice, the expression of mRNA for interferon-gamma (IFN-gamma) and interleukin-4 (IL-4) in purified CD4+ lymph node cells was analyzed. Wild-type 129/Sv/Ev mice showed high levels of mRNA for IFN-gamma and low levels of mRNA for IL-4 which is indicative of a Th1 response. In contrast, IL-12- deficient mice and susceptible BALB/c mice developed a strong Th2 response with high levels of IL-4 mRNA and low levels of IFN-gamma mRNA in CD4+ T cells. Similarly, lymph node cells from infected wild-type 129 mice produced predominantly IFN-gamma in response to stimulation with Leishmania antigen in vitro whereas lymph node cells from IL-12-deficient mice and susceptible BALB/c mice produced preferentially IL-4. Taken together, these results confirm in vivo the importance of IL-12 in induction of Th1 responses and protective immunity against L. major.

13-12-19, stade Jean-Bouin, hockey [sur gazon], équipe des Possibles : [photographie de presse] / [Agence Rol]

Référence bibliographique : Rol, 57224

22/12/19, roue de la loterie du tirage d'un million : [photographie de presse] / [Agence Rol]

Référence bibliographique : Rol, 57283