Arrest of Aaron Burr in Alabama, in 1807. [To which is prefixed, a prospectus of the author's "History of Alabama".

Mode of access: Internet.

Speech of Hon. J.A. McDougall, of California, on the arrest of Gen. Stone, and the rights of the soldier and citizen. Delivered in the Senate of the United States, April 15, 16, & 22, 1862.

Mode of access: Internet.

The seamen's bill. (Life boat equipment) Hearings held before the Committee on the merchant marine and fisheries, House of Representatives, on S. 136, an act to promote the welfare of American seamen in the merchant marine of the United States; to abolish arrest and imprisonment as a penalty for desertion and to secure the abrogation of treaty provisions in relation thereto: and to promote safety at sea. Hearings of December 13, 15, 16, 17, 18, and 19, 1913.

Joshua W. Alexander, Chairman.

Probable Cause to Arrest and Admissibility of Evidence

Mode of access: Internet.

Life stress and essential hypertension; a study of circulatory adjustments in man,

Bibliography: p.234-245.

Probable cause to arrest and admissibility of evidence /

Mode of access: Internet.

The Effect of Arrest on Intimate Partner Violence Recidivism

Thesis (Master's)--University of Washington, 2016-06

Brca1 inactivation induces p27Kip1-dependent cell cycle arrest and delayed development in the mouse mammary gland

One common characteristic of breast cancers arising in carriers of the predisposition gene BRCA1 is a loss of expression of the CDK inhibitor p27(Kip1) (p27), suggesting that p27 interacts epistatically with BRCA1. To investigate this relationship, we examined expression of p27 in mice expressing a dominant negative allele of Brca1 (MMTV-trBr) in the mammary gland. While these mice rarely develop tumors, they showed a 50% increase in p27 protein and a delay in mammary gland development associated with reduced proliferation. In contrast, on a p27 heterozygote background, MMTV-trBrca1 mice showed an increase in S phase cells, and normal mammary development. p27 was the only protein in the cyclin cyclin-dependent kinase network to show altered expression, suggesting that it may be a central mediator of cell cycle arrest in response to loss of function of BRCA1. Furthermore, in human mammary epithelial MCF7 cells expressing BRCA1-specific RNAi and in the BRCA1-deficient human tumor cell line HCC1937, p27 is elevated at the mRNA level compared to cells expressing wild-type BRCA1. We hypothesize that disruption of BRCA1 induces an increase in p27 that inhibits proliferation. Accordingly, reduction in p27 expression leads to enhancement of cellular proliferation in the absence of BRCA1.

Synthetic ceramides induce growth arrest or apoptosis by altering cellular redox status

Reactive oxygen species (ROS) and ceramide are each partly responsible for the signal transduction of a variety of extracellular agents. Furthermore, the application of synthetic, short-chain ceramides mimics the cellular responses to these extracellular agents. However, the significance of ROS involvement in ceramide signaling pathways is poorly understood. Here we describe that the (cellular responses to C2-/C6-ceramide of growth arrest in U937 monocytes and apoptosis in Jurkat T-cells are preceded by a rise in mitochondrial peroxide production. In Jurkat T-cells, this is associated with a large time- and dose-dependent loss of cellular glutathione. However, in U937 monocytes, glutathione loss is transient. Differences in the magnitude and kinetics of this alteration in cellular redox state associate with discrete outcomes, namely growth arrest or apoptosis. © 2002 Elsevier Science (USA). All rights reserved.