Majmūʻat rasāʾil arbaʻah, al-ūlá Rafʻ al-ishtibāh ʻan ḥadīth man ṣallá fī masjidī arbaʻīn ṣalāh, al-thānīyah Tanbīh al-anām ʻan kayfīyat isqāṭ al-ṣalāh wa-al-ṣīyām, al-thālithah al-Kawākib al-lāmiʻāt fī ḥukm al-māʾiʻāt, al-rābiʻah Minnat al-Khāliq fī qawl al-rajul li-zawjatihi ghayr al-madkhūl bi-hā anti ṭāliq wa-ṭāliq wa-ṭāliq [microform] /

Microfilm.

Hādhihi taqrīrāt sharīfah wa-tadqīqāt munīfah ʻalá ḥāshiyat Abī al-Najā ʻalá sharḥ Khālid al-Azharī [microform] /

Microfilm.

Amālī ʻAlī ʻAbd al-Rāziq, min ʻulamāʼ al-Azhar, fī ʻilm al-bayān wa-tārīkhih [microform]

Microfilm.

al-ʻAdhārá al-māysāt fī al-azjāl wa-al-muwashshaḥāt [microform] ; naqalahā Fīlīb Qaʻdān al-Khāzin.

Microfilm.

Latviešu zinātne un literātūra /

Description based on: 1908/1909.

Rättsväsendet ... Justitieexpeditionens i Kejserliga senaten underdåniga berättelse ...

Mode of access: Internet.

Das Angeblich von rāmānuja verfasste vedāntatattvasāra mit einer Einleitung und Anmerkungen Herausgegeben und ins deutsche Übertragen /

Thesis (doctoral)--Universität Leipzig, 1906.

Hörsamkeit grosser räume,

Bibliographical foot-notes.

History of the Puritans; or, The rise, principles, and sufferings of the Protestant dissenters, to the glorious æra of the revolution;

Mode of access: Internet.

Aberrant reactive oxygen and nitrogen species generation in rheumatoid arthritis (RA):causes and consequences for immune function, cell survival, and therapeutic intervention

The infiltration and persistence of hematopoietic immune cells within the rheumatoid arthritis (RA) joint results in elevated levels of pro-inflammatory cytokines, increased reactive oxygen (ROS) and -nitrogen (RNS) species generation, that feeds a continuous self-perpetuating cycle of inflammation and destruction. Meanwhile, the controlled production of ROS is required for signaling within the normal physiological reaction to perceived "foreign matter" and for effective apoptosis. This review focuses on the signaling pathways responsible for the induction of the normal immune response and the contribution of ROS to this process. Evidence for defects in the ability of immune cells in RA to regulate the generation of ROS and the consequence for their immune function and for RA progression is considered. As the hypercellularity of the rheumatoid joint and the associated persistence of hematopoietic cells within the rheumatoid joint are symptomatic of unresponsiveness to apoptotic stimuli, the role of apoptotic signaling proteins (specifically Bcl-2 family members and the tumor suppressor p53) as regulators of ROS generation and apoptosis are considered, evaluating evidence for their aberrant expression and function in RA. We postulate that ROS generation is required for effective therapeutic intervention.