Complex scalar dark matter in a B-L model

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)

Search for invisible decays of Higgs bosons in the vector boson fusion and associated ZH production modes

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Observation of the diphoton decay of the Higgs boson and measurement of its properties

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

O bóson de Higgs no modelo padrão das interações eletrofracas: aspectos fenomenológicos do setor escalar e resultados experimentais

Pós-graduação em Física - FEG

Soluções do tipo vórtice em um modelo de Maxwell-Chern-Simons-Higgs com campos de Gauge distintos

Pós-graduação em Física - FEG

New physics from warped compact extra dimensions: from model building to colliders signals

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Dirac gaugions, R symmetry and the 125 GeV Higgs

Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)

Search for vector-like T quarks decaying to top quarks and Higgs bosons in the all-hadronic channel using jet substructure

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Expression of cellular components in granulomatous inflammatory response in piaractus mesopotamicus model

Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)

Searches for heavy Higgs bosons in two-Higgs-doublet models and for t→ch decay using multilepton and diphoton final states in pp collisions at 8 TeV

Searches are presented for heavy scalar (H) and pseudoscalar (A) Higgs bosons posited in the two doublet model (2HDM) extensions of the standard model (SM). These searches are based on a data sample of pp collisions collected with the CMS experiment at the LHC at a center-of-mass energy of root s = 8 TeV and corresponding to an integrated luminosity of 19.5 fb(-1). The decays H -> hh and A -> Zh, where h denotes an SM-like Higgs boson, lead to events with three or more isolated charged leptons or with a photon pair accompanied by one or more isolated leptons. The search results are presented in terms of the H and A production cross sections times branching fractions and are further interpreted in terms of 2HDM parameters. We place 95% C.L. cross section upper limits of approximately 7 pb on sigma B for H -> hh and 2 pb for A -> Zh. Also presented are the results of a search for the rare decay of the top quark that results in a charm quark and an SM Higgs boson, t -> ch, the existence of which would indicate a nonzero flavor-changing Yukawa coupling of the top quark to the Higgs boson. We place a 95% C.L. upper limit of 0.56% on B(t -> ch).

Radiative Omega(-)->Xi(-)gamma decay and physics beyond the standard model

We investigate the impact of new physics beyond the standard model to the s → dγ process, which is responsible for the short-distance contribution to the radiative decay Ω-Ξ-γ. We study three representative extensions of the standard model: namely, a one-family technicolor model, a two-Higgs-doublet model, and a model containing scalar leptoquarks. When constraints arising from the observed b→sγ transition and the upper limit on D0-D̄0 mixing are taken into account, we find no significant contributions of new physics to the s→dy process.

Search for a nonstandard Higgs boson in diphoton events at p(p)over-bar collisions

We estimate the attainable limits on the coupling of a nonstandard Higgs boson to two photons taking into account the data collected by the Fermilab collaborations on diphoton events. We based our analysis on a general set of dimension-6 effective operators that give rise to anomalous couplings in the bosonic sector of the standard model. If the coefficients of all blind operators have the same magnitude, indirect bounds on the anomalous triple vector-boson couplings can also be inferred, provided there is no large cancellatton in the Higgs-gamma-gamma coupling.

Searching for an invisibly decaying Higgs boson in e+e-, eγ, and γγ collisions

Higgs bosons can have a substantial invisible branching ratio in many extensions of the Standard Model, such as models where the Higgs bosons decay predominantly into light or massless weakly interacting Goldstone bosons. In this work, we examine the production mechanisms and backgrounds for invisibly decaying Higgs bosons at the Next Linear e+e- Collider operating in the modes e+e-, eγ, and γγ. We demonstrate that such machine is much more efficient to survey for invisibly decaying Higgs bosons than the Large Hadron Collider at CERN.

Anomalous Higgs boson contribution to e(+)e(-)->b(b)over-bar gamma at CERN LEP 2

We study the effect of anomalous Hγγ and HZγ couplings, described by a general effective Lagrangian, on the process e+e-→bb̄γ at CERN LEP 2 energies. We include the relevant irreducible standard model background to this process, and from the photon energy spectrum, we determine the reach of LEP 2 to unravel the anomalous couplings by analyzing the significance of the signal for a Higgs boson with a mass up to 150 GeV.

Chronic VEGF Blockade Worsens Glomerular Injury in the Remnant Kidney Model

VEGF inhibition can promote renal vascular and parenchymal injury, causing proteinuria, hypertension and thrombotic microangiopathy. The mechanisms underlying these side effects are unclear. We investigated the renal effects of the administration, during 45 days, of sunitinib (Su), a VEGF receptor inhibitor, to rats with 5/6 renal ablation (Nx). Adult male Munich-Wistar rats were distributed among groups S+V, sham-operated rats receiving vehicle only; S+Su, S rats given Su, 4 mg/kg/day; Nx+V, Nx rats receiving V; and Nx+Su, Nx rats receiving Su. Su caused no change in Group S. Seven and 45 days after renal ablation, renal cortical interstitium was expanded, in association with rarefaction of peritubular capillaries. Su did not worsen hypertension, proteinuria or interstitial expansion, nor did it affect capillary rarefaction, suggesting little angiogenic activity in this model. Nx animals exhibited glomerulosclerosis (GS), which was aggravated by Su. This effect could not be explained by podocyte damage, nor could it be ascribed to tuft hypertrophy or hyperplasia. GS may have derived from organization of capillary microthrombi, frequently observed in Group Nx+Su. Treatment with Su did not reduce the fractional glomerular endothelial area, suggesting functional rather than structural cell injury. Chronic VEGF inhibition has little effect on normal rats, but can affect glomerular endothelium when renal damage is already present.