909 resultados para Cucumis melo var. reticulatus Naud.
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Entré à la Bibliothèque du roi en 1701 parmi les mss. d'Antoine Faure; cf. Delisle, Cab. des mss., I, 320; Omont, Anciens inventaires, IV, 400-410; — cardinal Jules Mazarin, cf. note "Catalogus msc. bibliothecae olim Naud. quae, illo mortuo, cessivere, simul cum libris editis, in manus cardinalis Mazarini" (A); — ancienne cote "51" (contreplat inf.)
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Contient : 1 « Catalogue des livres qui sont en l'estude de G. Naudé, à Paris » ; 2 Autre catalogue de livres, divisés par formats
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STOBBS, Lorne,W ABSTRACT Biochemical and Histological Investigations of viral localisation in the hypersensitive reaction of Phaseolus vulgaris L. var Pinto to tobacco mosaic virus infection. The infection of Phaseolus vulgaris L. var Pinto with tobacco mosaic virus (TMV) results in the production of distinct necrotic lesions confining the virus to restricted areas of the leaf surface. Biochemical and histological changes in the leaf tissue as a result of infection have been described. Trace accumulations of fluorescent metabolites, detected prior to lesion expression represent metabolites produced, by the cell in response to virus infection. These substances, are considered to undergo oxidation and in diffusing into adjacent cells, react with cellular constituents causing the death of these cells. Such cellular necrosis in advance of infection effectively limits virus spread. Chromatographic studies on extracts from TMV infected Pinto bean leaf tissue suggests that a number of extra-fluorescent metabolites produced on lesion'expression represent end products of phenolic oxidation r,eactionsoccurring earlier in these cells. Inhibition of phenolic oxidation by ascorbate infiltration or elevated temperature treatment resulted in the absence of extra-fluorescent metabolites and the continued movement of virus in the absence of necrosis. Further studies with i ascorbate infiltration indicated that irreversible necrotic events were determined as early as 12 tci 18 hrs after viral inoculation. Histochemical tests indicated that callose formation was initiated at this time, and occurred in response to necrotisation. Inhibition of necrosis by either ascorbate infiltration or elevated temp8rature treatment resulted in the absence of callose deposition. Scanning electron'micrographs of infected tissue revealed severe epidermal and palisade cell damage. Histochemical tests indicated extensive callose formation in cells bordering the lesion, and suggested the role of callose iTh the blockage of intercellular connections limiting virus movement. The significance of these cellular changes is discussed. ii
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Tesis (MaestrÃa en Ciencias en Producción AgrÃcola) UANL
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Tesis (MaestrÃa en Ciencas Especialidad en Producción AgrÃcola) UANL
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Tesis (MaestrÃa en Ciencias con Especialidad en InmunobiologÃa) UANL
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Tesis (MaestrÃa en Ciencias en Producción AgrÃcola) UANL
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Tesis (MaestrÃa en Ciencias en Producción AgrÃcola) UANL
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Tesis (MaestrÃa en Ciencias Forestales) UANL
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Tesis (Maestro en Ciencias con Especialidad en Microbiolo gÃa) U.A.N.L.
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Tesis (Maestro en Ciencias con especialidad en microbiolo gia) U.A.N.L.
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Tesis (MaestrÃa en Ciencias con Especialidad en EntomologÃa Médica) U.A.N.L.
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Tesis (MaestrÃa en Ciencias con Especialidad en MicrobiologÃa) U.A.N.L. Facultad de Ciencias Biológicas